About: Exogenous Pancreatic Kallikrein Improves Diabetic Cardiomyopathy in Streptozotocin-Induced Diabetes

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About: Exogenous Pancreatic Kallikrein Improves Diabetic Cardiomyopathy in Streptozotocin-Induced Diabetes Goto Sponge NotDistinct Permalink

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scholarly article by Meng Wu et al published 2018 in Frontiers in Pharmacology

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rdf:type	Item
description	wetenschappelijk artikel (nl) im Januar 2018 veröffentlichter wissenschaftlicher Artikel (de) наукова стаття, опублікована у 2018 (uk) article scientifique publié en 2018 (fr) scholarly article by Meng Wu et al published 2018 in Frontiers in Pharmacology (en)
publication date	wds:Q58797121-08A35F9A-4C7C-4155-A1D1-34F6278D4844
publication date	2018-01-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q58797121-61E986F0-C84B-459E-BAD8-CC417ADE82CB
language of work or name	English
cites work	wds:Q58797121-02A55A6D-060B-40D3-A847-78407C30FCD0 wds:Q58797121-1905E82B-4AAC-4A1E-961F-EFDD74EC1F0B wds:Q58797121-1D1B2D13-1972-4BA7-AC6B-0CAF52B03070 wds:Q58797121-1D339E04-B7D2-4839-9CB4-7D4912691A58 wds:Q58797121-2140EFD4-F3C7-4CBB-B3F4-1DB4FC222A96 wds:Q58797121-2BDA6B88-6914-48CC-8300-E5BAF0685AFF wds:Q58797121-3B39AF2B-548A-4BC4-9EF2-1142E4F899C7 wds:Q58797121-3DEDF347-C76C-4A5C-AB15-0181479457C2 wds:Q58797121-3E130F64-FD26-4A8B-8369-AF980FEFDB0D wds:Q58797121-4196A14C-51DD-4C23-AEE4-C32CC827BD5B wds:Q58797121-436F33C1-A75A-4EAA-8A24-81D6746AE114 wds:Q58797121-45A4D7F5-8836-47CC-A275-E6A8E890E950 wds:Q58797121-4CDF1183-FDCE-421C-B2B3-1C4D54980247 wds:Q58797121-501E4ED8-651C-4F54-B248-F601DBBABCBD wds:Q58797121-57322656-9707-4ABE-88CA-1282110A0DAB wds:Q58797121-5BB5D6FC-4549-4E89-8CF7-E991CA9C91F3 wds:Q58797121-5DE021D3-7F5B-4684-A527-BC7D3FE43463 wds:Q58797121-6437A627-A4DA-4008-AA4C-0A82E50D53E8 wds:Q58797121-646AE9A0-BA86-4783-8E8C-6BE3E7098CDD wds:Q58797121-6485BADB-45A2-481F-B827-1686251BB046 wds:Q58797121-65175F8F-FE7D-4CA5-8331-1D32CA66003D wds:Q58797121-70937208-5DC5-4F63-8E61-5A4105583372 wds:Q58797121-712A45DA-B55D-4586-95F0-4E13BBED71C2 wds:Q58797121-74B0F189-D4AF-47E0-9449-DAE47E9878C9 wds:Q58797121-76E46414-0307-46E0-A001-1ACC8675F17D wds:Q58797121-7EBD1028-676D-492F-B075-05783A9B4C57 wds:Q58797121-82B47BED-5F02-4BA4-AFC3-A2FECA97159E wds:Q58797121-8500A10C-7513-4C29-BA64-FD60E662EC84 wds:Q58797121-8A7DEEA0-21C7-4F22-B0A9-882DD0B03561 wds:Q58797121-8BCEDBC4-DD82-4B14-97FF-8948F9B402D1 wds:Q58797121-8D292954-CFAB-40D6-ACB2-6021C74C332A wds:Q58797121-8D4BB41A-ECFA-4497-AFAA-2BBB45851D45 wds:Q58797121-8F864EA3-82DF-49BB-9B7D-CBBF36CB2DBE wds:Q58797121-90798D5F-DB9E-49B0-A5D4-F8285362878B wds:Q58797121-9164B200-CBC9-4E56-B2A8-DB7CB66FF3A5 wds:Q58797121-96CE9EE4-A3ED-4A48-A38A-E84C32882ED7 wds:Q58797121-9D672014-529A-45B4-9AE1-161733BCB73F wds:Q58797121-AB48C8C2-9859-446E-9FAA-1DFFD25AB8CD wds:Q58797121-AF75479A-B90D-4FE8-B564-1ADFEECA55A8 wds:Q58797121-AFD7E05B-7257-458A-8BC5-5152A6E74082 wds:Q58797121-B0167DBD-6B39-49AA-8F6C-1428114F9C8E wds:Q58797121-BA04C390-9B4E-44C4-BD79-878979D364D3 wds:Q58797121-BD5AE2DB-0A09-4C70-8A6A-5524C7F4A4F8 wds:Q58797121-C62103DB-80DC-4763-AAF2-054F2B4AABCD wds:Q58797121-C6FF3CB2-2B49-43AA-BDD4-97B672D42ED9 wds:Q58797121-C7EFB722-992D-4DB2-82BF-ADE38FB9FA1D wds:Q58797121-CAFF40CC-74DD-438E-832C-BDFFD036497D wds:Q58797121-CF93307B-F599-478C-ACE4-A3DA1CBA29B7 wds:Q58797121-D1C3BB4D-AE25-4AA5-99EC-2F8351AD151B wds:Q58797121-D3BDFE53-2204-4F01-AF27-E0103497A686 wds:Q58797121-D7B38562-AE6B-486C-9EB0-319D87E882B3 wds:Q58797121-DACF1729-EC9A-4926-B7DA-D71E5C2DB292 wds:Q58797121-E02CAA81-5B1E-48FF-8CE8-029B873F4BFC wds:Q58797121-E0D76350-40D7-4D12-A0D6-9D290A0C7F50 wds:Q58797121-E51B63B5-E62B-478D-80E9-BCE410405D99 wds:Q58797121-E5DCB52D-1373-4934-A88A-451D8A3621D5 wds:Q58797121-E83CCB73-D591-4FB3-B392-DF720A85BBCC wds:Q58797121-EA207E08-63AE-4A12-9B72-C5627517CD5F wds:Q58797121-F9BD5768-CF9A-47AC-A837-A1874175C5EA wds:Q58797121-FD268E8E-68B3-408E-8C5D-2ACAC3F521BC wds:Q58797121-FFA32E15-74CA-4704-93A3-13024804A232
cites work	Phospholamban interactome in cardiac contractility and survival: A new vision of an old friend Prevention of cardiac fibrosis and left ventricular dysfunction in diabetic cardiomyopathy in rats by transgenic expression of the human tissue kallikrein gene Insulin resistance and hyperinsulinaemia in diabetic cardiomyopathy Reciprocal phosphorylation and regulation of endothelial nitric-oxide synthase in response to bradykinin stimulation SUMO1-dependent modulation of SERCA2a in heart failure Diabetic cardiomyopathy, causes and effects Inhibition of miR-25 improves cardiac contractility in the failing heart. New type of cardiomyopathy associated with diabetic glomerulosclerosis Nrf2 is critical in defense against high glucose-induced oxidative damage in cardiomyocytes Role of diabetes in congestive heart failure: the Framingham study Natriuretic peptides (BNP and NT-proBNP): measurement and relevance in heart failure Biological tyrosine nitration: a pathophysiological function of nitric oxide and reactive oxygen species. Plasma brain natriuretic peptide as an indicator of left ventricular systolic function and long-term survival after acute myocardial infarction. Comparison with plasma atrial natriuretic peptide and N-terminal proatrial natriuretic peptide Fluvastatin-induced reduction of oxidative stress ameliorates diabetic cardiomyopathy in association with improving coronary microvasculature. Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity Human apolipoprotein A-I gene transfer reduces the development of experimental diabetic cardiomyopathy The role of TGF-beta signaling in myocardial infarction and cardiac remodeling Removal of Abnormal Myofilament O-GlcNAcylation Restores Ca2+ Sensitivity in Diabetic Cardiac Muscle. Spironolactone attenuates experimental uremic cardiomyopathy by antagonizing marinobufagenin Cardioprotective effects of rutin via alteration in TNF-α, CRP, and BNP levels coupled with antioxidant effect in STZ-induced diabetic rats. Luteolin ameliorates cardiac failure in type I diabetic cardiomyopathy. Conundrum of pathogenesis of diabetic cardiomyopathy: role of vascular endothelial dysfunction, reactive oxygen species, and mitochondria Prevention by sulforaphane of diabetic cardiomyopathy is associated with up-regulation of Nrf2 expression and transcription activation Transgenic activation of the kallikrein-kinin system inhibits intramyocardial inflammation, endothelial dysfunction and oxidative stress in experimental diabetic cardiomyopathy Emerging role of oxidative stress in metabolic syndrome and cardiovascular diseases: important role of Rac/NADPH oxidase Impact of diabetes on cardiac structure and function: the strong heart study Renal Denervation Findings on Cardiac and Renal Fibrosis in Rats with Isoproterenol Induced Cardiomyopathy High glucose inhibits glucose-6-phosphate dehydrogenase, leading to increased oxidative stress and beta-cell apoptosis Myocardial expression of rat bradykinin receptors and two tissue kallikrein genes in experimental diabetes.

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