About: Androgen-Independent Prostate Cancer Is a Heterogeneous Group of Diseases

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rdf:type	Item
description	article (en) wetenschappelijk artikel (nl) article scientifique publié en 2004 (fr) наукова стаття, опублікована в грудні 2004 (uk) im Dezember 2004 veröffentlichter wissenschaftlicher Artikel (de)
publication date	wds:Q57693579-47C01950-E2E4-4FD4-938F-61BA80EE3B15
publication date	2004-12-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q57693579-2F2B67C3-A595-4FF9-A3D1-A0DA67F921F1
language of work or name	English
author name string	wds:Q57693579-1A361444-A6B3-49A5-A067-82378201FAAE wds:Q57693579-2F022490-EF91-4DC5-AB25-4562A1785991 wds:Q57693579-3E6B4A3D-1DB0-4275-8BC2-270E0DF1282C wds:Q57693579-87B7B882-2B1B-4654-8772-02EEE9104DD5 wds:Q57693579-99DEE45F-E538-430C-B71B-079370E457CB wds:Q57693579-B1B01DFF-2384-4A27-8B83-7AE25930D0E5 wds:Q57693579-ECC0CFA1-923F-448E-BA89-0D21EA7AB601
author name string	Ming Zhou Debashis Ghosh Rohit Mehra Robert Kim Rajal B Shah Tarek A Bismar Gary R Macvicar
rdfs:label	Androgen-Independent Prostate Cancer Is a Heterogeneous Group of Diseases (en) Androgen-Independent Prostate Cancer Is a Heterogeneous Group of Diseases (nl)
skos:prefLabel	Androgen-Independent Prostate Cancer Is a Heterogeneous Group of Diseases (en) Androgen-Independent Prostate Cancer Is a Heterogeneous Group of Diseases (nl)
name	Androgen-Independent Prostate Cancer Is a Heterogeneous Group of Diseases (en) Androgen-Independent Prostate Cancer Is a Heterogeneous Group of Diseases (nl)
author	wds:Q57693579-1387B52D-2E4D-41BF-A91A-D76B9B55A566 wds:Q57693579-B3AC9AB5-8EF3-4C09-BFA7-0AA2304BDF2D wds:Q57693579-B64786BB-3462-4D11-8C18-03EB6BB09C5D wds:Q57693579-B94FC827-9696-420C-9799-6EF2E373EF09 wds:Q57693579-D1D81983-1D98-4673-BF15-93D7B14EFF55 wds:Q57693579-EED60570-D3C2-4FB3-BCE2-DBCF42A6B5EE
author	Kenneth J. Pienta Arul Chinnaiyan Sooryanarayana Varambally Mark Rubin Ronglai Shen Jason Harwood
title	wds:Q57693579-F375A647-3593-4297-9494-DB0BBC3CB176
title	Androgen-independent prostate cancer is a heterogeneous group of diseases: lessons from a rapid autopsy program (en)
page(s)	wds:Q57693579-0035CA83-C9A8-4FC2-BA45-5049711A04C1
page(s)	9209-9216
instance of	wds:Q57693579-1EFC4F11-3686-4AB4-8D5F-4421F38EAA08
instance of	scholarly article
main subject	wds:Q57693579-68CA0E3F-7FCB-45CD-B55C-BAD0C37F124D
main subject	prostate cancer
PubMed ID	wds:Q57693579-55D405CB-B6B8-4B74-A10B-78E8AAC8D64D
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/15604294
PubMed ID	15604294
published in	wds:Q57693579-D9753139-5BD8-44E7-9543-9BADF9716CC6
published in	Cancer Research
issue	wds:Q57693579-9442596E-F2F8-485D-BCD3-A7138EEDF958
volume	wds:Q57693579-060790B8-36D4-4657-BB6A-52725F86C798
issue	24
volume	64
DOI	wds:Q57693579-78DF3C5A-AF7B-477E-84BF-DCB088933A4A
DOI	http://dx.doi.org/10.1158/0008-5472.CAN-04-2442
DOI	10.1158/0008-5472.CAN-04-2442
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q57693579
is cites work of	Siah2-dependent concerted activity of HIF and FoxA2 regulates formation of neuroendocrine phenotype and neuroendocrine prostate tumors Monoclonal antibody targeting of N-cadherin inhibits prostate cancer growth, metastasis and castration resistance Cadherin-11 promotes the metastasis of prostate cancer cells to bone Adaptation or selection--mechanisms of castration-resistant prostate cancer IL-6/IL-6R as a potential key signaling pathway in prostate cancer development Selective inhibitory effect of HPMA copolymer-cyclopamine conjugate on prostate cancer stem cells Frequent loss of cystatin E/M expression implicated in the progression of prostate cancer Maintenance of intratumoral androgens in metastatic prostate cancer: a mechanism for castration-resistant tumor growth Ethics guidelines for research with the recently dead Prostate epithelial Pten/TP53 loss leads to transformation of multipotential progenitors and epithelial to mesenchymal transition Targeting TGF-beta in prostate cancer: therapeutic possibilities during tumor progression Phospho-MED1-enhanced UBE2C locus looping drives castration-resistant prostate cancer growth Targeted high throughput sequencing in clinical cancer settings: formaldehyde fixed-paraffin embedded (FFPE) tumor tissues, input amount and tumor heterogeneity E-cadherin as an indicator of mesenchymal to epithelial reverting transitions during the metastatic seeding of disseminated carcinomas. Increased expression of anterior gradient-2 is significantly associated with poor survival of prostate cancer patients Regulation of prostate cancer progression by galectin-3 Distinct genetic alterations in small cell carcinoma from different anatomic sites The androgen receptor can signal through Wnt/beta-Catenin in prostate cancer cells as an adaptation mechanism to castration levels of androgens The PSA(-/lo) prostate cancer cell population harbors self-renewing long-term tumor-propagating cells that resist castration Whole-Exome Sequencing of Metastatic Cancer and Biomarkers of Treatment Response. Nuclear versus cytoplasmic localization of filamin A in prostate cancer: immunohistochemical correlation with metastases Copy number analysis indicates monoclonal origin of lethal metastatic prostate cancer AR-regulated TWEAK-FN14 pathway promotes prostate cancer bone metastasis Minimum information specification for in situ hybridization and immunohistochemistry experiments (MISFISHIE) Androgen receptor-negative human prostate cancer cells induce osteogenesis in mice through FGF9-mediated mechanisms Amplification and overexpression of prosaposin in prostate cancer Active sonic hedgehog signaling between androgen independent human prostate cancer cells and normal/benign but not cancer-associated prostate stromal cells ERG cooperates with androgen receptor in regulating trefoil factor 3 in prostate cancer disease progression Computational modeling of pancreatic cancer reveals kinetics of metastasis suggesting optimum treatment strategies Modeling somatic evolution in tumorigenesis Biomarkers in the management and treatment of men with metastatic castration-resistant prostate cancer Characterization of bone metastases from rapid autopsies of prostate cancer patients Telomeres and telomerase in prostatic intraepithelial neoplasia and prostate cancer biology PSA reduces prostate cancer cell motility by stimulating TRPM8 activity and plasma membrane expression Co-culturing human prostate carcinoma cells with hepatocytes leads to increased expression of E-cadherin PRSS3/mesotrypsin is a therapeutic target for metastatic prostate cancer Cross-species regulatory network analysis identifies a synergistic interaction between FOXM1 and CENPF that drives prostate cancer malignancy

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