About: Gian Paolo Fadini

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher ORCID ID = 0000-0002-6510-2097

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) 연구원 (ko) araştırmacı (tr) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) onderzoeker (nl) researcher ORCID ID = 0000-0002-6510-2097 (en)
rdfs:label	Gian Paolo Fadini (en) Gian Paolo Fadini (es) Gian Paolo Fadini (it) Gian Paolo Fadini (nl) Gian Paolo Fadini (ast)
skos:prefLabel	Gian Paolo Fadini (en) Gian Paolo Fadini (es) Gian Paolo Fadini (it) Gian Paolo Fadini (nl) Gian Paolo Fadini (ast)
name	Gian Paolo Fadini (en) Gian Paolo Fadini (es) Gian Paolo Fadini (it) Gian Paolo Fadini (nl) Gian Paolo Fadini (ast)
Scopus author ID	wds:Q57544235-856B819A-79AA-47A7-9F4C-D7B2664F352D
Scopus author ID	6506582748
given name	wds:Q57544235-0BC743C0-1DBF-4154-834C-BD05A334FB2A
given name	Gian
instance of	wds:Q57544235-E2816CBA-4127-4645-8DE1-068F7AE2939C
instance of	Homo sapiens
occupation	wds:Q57544235-8E9F39A3-0668-419F-9AE0-A928612FA636
employer	wds:Q57544235-AD8F8B56-07BE-4677-B1C6-F37069BE776E
sex or gender	wds:Q57544235-3FAE774E-2725-4EC9-9B12-FCC06EB60902
SBN author ID	wds:Q57544235-E993CAC7-E270-4DBF-B8FA-432562F0567C
on focus list of Wikimedia project	wds:Q57544235-8FFCAC3A-2F40-45BB-9C70-EC91D1E21810
occupation	researcher
employer	University of Padua
sex or gender	male
SBN author ID	PISV015665
on focus list of Wikimedia project	WikiProject COVID-19
ORCID iD	wds:Q57544235-C14769ED-1016-48AF-AC4C-DAF3AC40576B
ORCID iD	https://orcid.org/0000-0002-6510-2097
ORCID iD	0000-0002-6510-2097
Padua Research Archive author ID	wds:Q57544235-F62091E1-8613-489F-A669-4888932F36CA
Padua Research Archive author ID	26483
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q57544235
is author of	Emerging role of circulating calcifying cells in the bone-vascular axis NETosis is induced by high glucose and associated with type 2 diabetes. Carotid plaque calcification predicts future cardiovascular events in type 2 diabetes Endothelial progenitor cells in the natural history of atherosclerosis. Widespread increase in myeloid calcifying cells contributes to ectopic vascular calcification in type 2 diabetes Reduced endothelial progenitor cells and brachial artery flow-mediated dilation as evidence of endothelial dysfunction in ocular hypertension and primary open-angle glaucoma Cardiovascular actions of GLP-1 and incretin-based pharmacotherapy. Downregulation of the longevity-associated protein sirtuin 1 in insulin resistance and metabolic syndrome: potential biochemical mechanisms The oral dipeptidyl peptidase-4 inhibitor sitagliptin increases circulating endothelial progenitor cells in patients with type 2 diabetes: possible role of stromal-derived factor-1alpha. Cardiovascular effects of DPP-4 inhibition: beyond GLP-1. The effects of dipeptidyl peptidase-4 inhibition on microvascular diabetes complications. Myeloid calcifying cells promote atherosclerotic calcification via paracrine activity and allograft inflammatory factor-1 overexpression. Simvastatin Rapidly and Reversibly Inhibits Insulin Secretion in Intact Single-Islet Cultures Erratum to: Simvastatin Rapidly and Reversibly Inhibits Insulin Secretion in Intact Single-Islet Cultures Sarcoidosis is a Th1/Th17 multisystem disorder Circulating endothelial progenitor cells are reduced in peripheral vascular complications of type 2 diabetes mellitus. Diabetes impairs progenitor cell mobilisation after hindlimb ischaemia–reperfusion injury in rats Significance of endothelial progenitor cells in subjects with diabetes. Number and Function of Endothelial Progenitor Cells as a Marker of Severity for Diabetic Vasculopathy Endothelial dysfunction: causes and consequences in patients with diabetes mellitus. Comparative effectiveness of liraglutide in the treatment of type 2 diabetes Peripheral blood CD34+KDR+ endothelial progenitor cells are determinants of subclinical atherosclerosis in a middle-aged general population. Low CD34+ cell count and metabolic syndrome synergistically increase the risk of adverse outcomes. Circulating CD34+ cells, metabolic syndrome, and cardiovascular risk Independent glucose and weight-reducing effects of Liraglutide in a real-world population of type 2 diabetic outpatients. Diabetes Limits Stem Cell Mobilization Following G-CSF but Not Plerixafor Endothelial progenitor cells in diabetes mellitus. Dipeptidyl peptidase-4 inhibition and vascular repair by mobilization of endogenous stem cells in diabetes and beyond Diabetes causes bone marrow autonomic neuropathy and impairs stem cell mobilization via dysregulated p66Shc and Sirt1. Sensory neuropathy hampers nociception-mediated bone marrow stem cell release in mice and patients with diabetes. The p66(Shc) redox adaptor protein is induced by saturated fatty acids and mediates lipotoxicity-induced apoptosis in pancreatic beta cells. Diabetes impairs mobilization of stem cells for the treatment of cardiovascular disease: a meta-regression analysis. Stem cell compartmentalization in diabetes and high cardiovascular risk reveals the role of DPP-4 in diabetic stem cell mobilopathy Circulating progenitor cells are reduced in patients with severe lung disease Circulating myeloid calcifying cells have antiangiogenic activity via thrombospondin-1 overexpression The endothelium abridges insulin resistance to premature aging Endothelial dysfunction in type 2 diabetes mellitus. Amelioration of glucose control mobilizes circulating pericyte progenitor cells in type 2 diabetic patients with microangiopathy. Circulating levels of endothelial progenitor cell mobilizing factors in the metabolic syndrome. Vascular smooth muscle cells and monocyte-macrophages accomplice in the accelerated atherosclerosis of insulin resistance states A perspective on NETosis in diabetes and cardiometabolic disorders Technical notes on endothelial progenitor cells: ways to escape from the knowledge plateau. Elevated white blood cell count is associated with prevalence and development of the metabolic syndrome and its components in the general population Monocyte-macrophage polarization balance in pre-diabetic individuals. High abundance proteins depletion vs low abundance proteins enrichment: comparison of methods to reduce the plasma proteome complexity. p66Shc deletion or deficiency protects from obesity but not metabolic dysfunction in mice and humans Age-associated cardiovascular risk and metabolomics of mitochondrial dysfunction. Circulating progenitor cell count for cardiovascular risk stratification: a pooled analysis The dipeptidyl peptidase-4 inhibitor saxagliptin improves function of circulating pro-angiogenic cells from type 2 diabetic patients A reappraisal of the role of circulating (progenitor) cells in the pathobiology of diabetic complications. Optimized glycaemic control achieved with add-on basal insulin therapy improves indexes of endothelial damage and regeneration in type 2 diabetic patients with macroangiopathy: a randomized crossover trial comparing detemir versus glargine. Endothelial dysfunction in diabetes: the role of reparatory mechanisms Insulin-induced glucose control improves HDL cholesterol levels but not reverse cholesterol transport in type 2 diabetic patients

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