scholarly article by Richard J Roman & Julian H Lombard published July 2007 in Hypertension
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| - wetenschappelijk artikel (nl)
- наукова стаття, опублікована в липні 2007 (uk)
- article scientifique publié en 2007 (fr)
- im Juli 2007 veröffentlichter wissenschaftlicher Artikel (de)
- scholarly article by Richard J Roman & Julian H Lombard published July 2007 in Hypertension (en)
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| - Richard J Roman
- Julian H Lombard
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| - Does 20-hydroxyeicosatetraenoic acid contribute to sex differences in cardiovascular risk by increasing oxidative stress? (en)
- Does 20-hydroxyeicosatetraenoic acid contribute to sex differences in cardiovascular risk by increasing oxidative stress? (nl)
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| - Does 20-hydroxyeicosatetraenoic acid contribute to sex differences in cardiovascular risk by increasing oxidative stress? (en)
- Does 20-hydroxyeicosatetraenoic acid contribute to sex differences in cardiovascular risk by increasing oxidative stress? (nl)
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| - Does 20-hydroxyeicosatetraenoic acid contribute to sex differences in cardiovascular risk by increasing oxidative stress? (en)
- Does 20-hydroxyeicosatetraenoic acid contribute to sex differences in cardiovascular risk by increasing oxidative stress? (nl)
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| - Does 20-hydroxyeicosatetraenoic acid contribute to sex differences in cardiovascular risk by increasing oxidative stress? (en)
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| - 10.1161/HYPERTENSIONAHA.107.090803
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is cites work
of | - A 20-hydroxyeicosatetraenoic acid agonist, N-[20-hydroxyeicosa-5(Z),14(Z)-dienoyl]glycine, opposes the fall in blood pressure and vascular reactivity in endotoxin-treated rats.
- A synthetic analogue of 20-HETE, 5,14-HEDGE, reverses endotoxin-induced hypotension via increased 20-HETE levels associated with decreased iNOS protein expression and vasodilator prostanoid production in rats
- 20-HETE in acute kidney injury
- Contribution of iNOS/sGC/PKG pathway, COX-2, CYP4A1, and gp91(phox) to the protective effect of 5,14-HEDGE, a 20-HETE mimetic, against vasodilation, hypotension, tachycardia, and inflammation in a rat model of septic shock
- Role of 20-HETE in the hypoxia-induced activation of Ca2+-activated K+ channel currents in rat cerebral arterial muscle cells
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