About: Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis

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About: Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis Goto Sponge NotDistinct Permalink

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scientific article published on 01 April 2009

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) article scientifique publié en 2009 (fr) наукова стаття, опублікована у квітні 2009 (uk) im April 2009 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'abril de 2009 (ast) scientific article published on 01 April 2009 (en)
publication date	wds:Q57130124-646FB43E-0030-4C69-A207-C46AA88304CB
publication date	2009-04-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q57130124-4F678D80-535E-45C1-AFF7-624B5ABD4912
language of work or name	English
cites work	wds:Q57130124-53F0FE3C-FA02-425A-927E-509C0ACB8D8A wds:Q57130124-8B8EBF5D-4FE4-47B8-8DF3-F70F65A0FE9A wds:Q57130124-8FB3CC33-524A-4BC1-9361-EA030554AE3F
cites work	Constitutive p40 promoter activation and IL-23 production in the terminal ileum mediated by dendritic cells Evaluation of diagnostic criteria for ankylosing spondylitis. A proposal for modification of the New York criteria STAT3 and NF-kappaB signal pathway is required for IL-23-mediated IL-17 production in spontaneous arthritis animal model IL-1 receptor antagonist-deficient mice
author name string	wds:Q57130124-05CA4BAB-38BD-4EFF-AB82-2AA394F7F36E wds:Q57130124-55CA4CC4-139C-4000-AF43-94E5D7E6DDC0 wds:Q57130124-A25F45BA-2D49-4EBE-BE71-152935E5EAED wds:Q57130124-B00DE96C-205A-4854-B731-0226291FD8C3 wds:Q57130124-B5AB6CEE-2CE7-40BA-AF0C-6286FE353975 wds:Q57130124-F00B1298-B8CE-4990-AC3B-A084DD0E551F wds:Q57130124-F4D862FE-9FD0-4A6A-9A8C-DB3A80F098FE
author name string	Michele Bombardieri Vito Franco Rossana Porcasi Sergio Peralta Alfonso Principato Annarita Giardina Ennio Giardina
rdfs:label	Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis (en) Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis (nl)
skos:prefLabel	Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis (en) Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis (nl)
name	Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis (en) Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis (nl)
author	wds:Q57130124-0F7F4466-1D7A-413D-A974-F748CB2AA59B wds:Q57130124-7F240996-E950-4287-981D-FABF61082993 wds:Q57130124-B212AC53-2B2C-4C5B-A6F1-691B15FC2917 wds:Q57130124-B661689D-3D2D-4316-97A1-2414897DD765 wds:Q57130124-D626A354-AF8A-4868-8C9F-9111D04142E7
author	Claudio Tripodo Costantino Pitzalis Francesco Ciccia Antonio Craxì Giovani Triolo
title	wds:Q57130124-677652FC-2DD7-4BEF-B1FC-FA2697392856
title	Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis (en)
page(s)	wds:Q57130124-DD787152-F8D7-43BD-B813-F4458F5ADF1A
page(s)	955-965
instance of	wds:Q57130124-8D113483-B373-408E-9283-400932BD2A65
instance of	scholarly article
main subject	wds:Q57130124-1B58D681-3EAD-4A72-8860-5C4A91D22D8D wds:Q57130124-3714B96C-BCE6-466E-911A-59BCA8EF4EEC wds:Q57130124-F7A7833A-7A98-43EF-B079-834AD622EC89 wds:Q57130124-e05b07bc-741b-41e3-ad44-af82609b7bc3
main subject	inflammation interleukins ankylosing spondylitis overexpression
PubMed ID	wds:Q57130124-55D065A9-E5B8-4607-BE44-9CEA5C656917
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/19333939
PubMed ID	19333939
published in	wds:Q57130124-CC27C853-254E-49FA-BE07-F70BEA03221E
published in	Arthritis and Rheumatism
issue	wds:Q57130124-946D3FF5-6734-4EEF-90D7-DA81285696B5
volume	wds:Q57130124-30D85CA2-D844-4AB3-BB2F-93CD0E1BD16E
issue	4
volume	60
DOI	wds:Q57130124-BB058794-A8BC-44D0-B8A3-054E3E5CC27C
DOI	http://dx.doi.org/10.1002/ART.24389
DOI	10.1002/ART.24389
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q57130124
is cites work of	Th17 cytokines and arthritis Spondyloarthritis IL-23 induces spondyloarthropathy by acting on ROR-γt+ CD3+CD4-CD8- entheseal resident T cells HLA-B27 misfolding and ankylosing spondylitis Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis Evidence that autophagy, but not the unfolded protein response, regulates the expression of IL-23 in the gut of patients with ankylosing spondylitis and subclinical gut inflammation In situ analysis of interleukin-23- and interleukin-12-positive cells in the spine of patients with ankylosing spondylitis Interleukin-23: a key cytokine in inflammatory diseases From HLA-B27 to spondyloarthritis: a journey through the ER. The role of IL-17-secreting mast cells in inflammatory joint disease The IL-23-IL-17 axis in inflammatory arthritis New perspectives on the ligands and function of the killer cell immunoglobulin-like receptor KIR3DL2 in health and disease Macrophage phenotype in the subclinical gut inflammation of patients with ankylosing spondylitis The role of T helper type 17 cells in inflammatory arthritis Targeting the interleukin-23/17 axis in axial spondyloarthritis. Delineation of the innate and adaptive T-cell immune outcome in the human host in response to Campylobacter jejuni infection. Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis. Interactions of the innate and adaptive arms of the immune system in the pathogenesis of spondyloarthritis T-cell receptor signaling and the pathogenesis of autoimmune arthritis: insights from mouse and man. Interleukin-23-Dependent γ/δ T Cells Produce Interleukin-17 and Accumulate in the Enthesis, Aortic Valve, and Ciliary Body in Mice. Review: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyond Microbiome in Inflammatory Arthritis and Human Rheumatic Diseases. Autoimmunity's collateral damage: Gut microbiota strikes 'back'. Gut inflammation and microbiome in spondyloarthritis Type 3 innate lymphoid cells producing IL-17 and IL-22 are expanded in the gut, in the peripheral blood, synovial fluid and bone marrow of patients with ankylosing spondylitis. Interleukin-22 and interleukin-22-producing NKp44+ natural killer cells in subclinical gut inflammation in ankylosing spondylitis.

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