About: Zhenlin Li

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

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rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) 研究者 (ja) araştırmacı (tr) wetenschapper (nl) дослідник (uk) वैज्ञानिक (hi) باحت (ary) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy)
rdfs:label	Zhenlin Li (en) Zhenlin Li (es) Zhenlin Li (nl) Zhenlin Li (ast)
skos:prefLabel	Zhenlin Li (en) Zhenlin Li (es) Zhenlin Li (nl) Zhenlin Li (ast)
name	Zhenlin Li (en) Zhenlin Li (es) Zhenlin Li (nl) Zhenlin Li (ast)
Scopus author ID	wds:Q57065645-D10E5C8E-74C2-49BB-97E8-57D94647A8F4
Scopus author ID	7409078284
instance of	wds:Q57065645-A3E5E77C-5590-4B0B-B6E8-72915384F5AD
instance of	Homo sapiens
doctoral student	wds:Q57065645-E46B0046-AC55-4528-9222-E090A37FC1CC
doctoral student	Nicolas Diguet
occupation	wds:Q57065645-0E5287B1-E724-41AA-BD1D-82A7787A8753
employer	wds:Q57065645-D68231D8-E412-4E3E-8876-B0D962F5BCDD
sex or gender	wds:Q57065645-834c18e1-2088-4dde-ab0d-21ec966a7ad6
VIAF ID	wds:Q57065645-BE8E1169-2CA7-48FC-9386-101C7888BA61
IdRef ID	wds:Q57065645-75D43335-8C66-49A8-9DC9-4DF52929D05B
educated at	wds:Q57065645-9781B3C7-13CA-44F2-917B-5097EFE03143
VIAF ID	http://viaf.org/viaf/292575621
IdRef ID	http://www.idref.fr/128687959/id
occupation	researcher
employer	French National Institute of Health and Medical Research
sex or gender	male
VIAF ID	292575621
IdRef ID	128687959
educated at	Paris Descartes University
ORCID iD	wds:Q57065645-1119856A-B048-4559-9FBD-3EB4DDE2A4C7
ORCID iD	https://orcid.org/0000-0002-3706-4505
ORCID iD	0000-0002-3706-4505
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q57065645
is author of	Mosaic inactivation of the serum response factor gene in the myocardium induces focal lesions and heart failure. Serum response factor is required for sprouting angiogenesis and vascular integrity A crucial role for Pax3 in the development of the hypaxial musculature and the long-range migration of muscle precursors. Inactivation of serum response factor contributes to decrease vascular muscular tone and arterial stiffness in mice. On noxious desmin: functional effects of a novel heterozygous desmin insertion mutation on the extrasarcomeric desmin cytoskeleton and mitochondria. Null mutation in the desmin gene gives rise to a cardiomyopathy. Study of regulation of mitochondrial respiration in vivo Desmin is essential for the tensile strength and integrity of myofibrils but not for myogenic commitment, differentiation, and fusion of skeletal muscle Synemin acts as a regulator of signalling molecules during skeletal muscle hypertrophy. A missense mutation in the alphaB-crystallin chaperone gene causes a desmin-related myopathy Conditional immortalization of normal and dysgenic mouse muscle cells by the SV40 large T antigen under the vimentin promoter control Interactions of intermediate filament protein synemin with dystrophin and utrophin Transgenic Mice Overexpressing the 5-Hydroxytryptamine Transporter Gene in Smooth Muscle Develop Pulmonary Hypertension SRF selectively controls tip cell invasive behavior in angiogenesis. Identification of a repeated domain within mammalian alpha-synemin that interacts directly with talin Absence of dystrophin in mice reduces NO-dependent vascular function and vascular density: total recovery after a treatment with the aminoglycoside gentamicin. An SRF/miR-1 axis regulates NCX1 and annexin A5 protein levels in the normal and failing heart New role for serum response factor in postnatal skeletal muscle growth and regeneration via the interleukin 4 and insulin-like growth factor 1 pathways Spontaneous muscular dystrophy caused by a retrotransposal insertion in the mouse laminin α2 chain gene Analysis of skeletal and cardiac muscle from desmin knock-out and normal mice by high resolution separation of myosin heavy-chain isoforms Proteome modulation in H9c2 cardiac cells by microRNAs miR-378 and miR-378. Aerobic Exercise and Pharmacological Treatments Counteract Cachexia by Modulating Autophagy in Colon Cancer Targeted inactivation of serum response factor in the developing heart results in myocardial defects and embryonic lethality Temporally controlled onset of dilated cardiomyopathy through disruption of the SRF gene in adult heart Selective involvement of serum response factor in pressure-induced myogenic tone in resistance arteries Posttranslational modifications of desmin and their implication in biological processes and pathologies Laminin receptor involvement in the anti-angiogenic activity of pigment epithelium-derived factor Excessive microvascular adaptation to changes in blood flow in mice lacking gene encoding for desmin Role of alpha1beta1-integrin in arterial stiffness and angiotensin-induced arterial wall hypertrophy in mice Human synemin gene generates splice variants encoding two distinct intermediate filament proteins Mechanical properties and structure of carotid arteries in mice lacking desmin Different expression of synemin isoforms in glia and neurons during nervous system development Lower active force generation and improved fatigue resistance in skeletal muscle from desmin deficient mice. Mechanical alterations in smooth muscle from mice lacking desmin. Comparative study of immune responses induced after immunization with plasmids encoding the HIV-1 Nef protein under the control of the CMV-IE or the muscle-specific desmin promoter. Human desmin gene: cDNA sequence, regional localization and exclusion of the locus in a familial desmin-related myopathy. Muscle creatine kinase deficiency triggers both actin depolymerization and desmin disorganization by advanced glycation end products in dilated cardiomyopathy Passive stiffness is increased in soleus muscle of desmin knockout mouse Synemin isoforms during mouse development: multiplicity of partners in vascular and neuronal systems. Synemin expression in developing normal and pathological human retina and lens Synemin interacts with the LIM domain protein zyxin and is essential for cell adhesion and migration Endothelial mineralocorticoid receptor activation enhances endothelial protein C receptor and decreases vascular thrombosis in mice MyoD, myogenin, and desmin-nls-lacZ transgene emphasize the distinct patterns of satellite cell activation in growth and regeneration Inducible mouse model of chronic intestinal pseudo-obstruction by smooth muscle-specific inactivation of the SRF gene Selective Tuberous Sclerosis Complex 1 Gene Deletion in Smooth Muscle Activates Mammalian Target of Rapamycin Signaling and Induces Pulmonary Hypertension. Migration of myogenic cells from the somites to the fore-limb buds of developing mouse embryos Efficacy of epicardially delivered adipose stroma cell sheets in dilated cardiomyopathy. Regulation of Connective Tissue Growth Factor and Cardiac Fibrosis by an SRF/MicroRNA-133a Axis Contribution of Rare and Common Genetic Variants to Plasma Lipid Levels and Carotid Stiffness and Geometry: A Substudy of the Paris Prospective Study 3. Vimentin knockout results in increased expression of sub-endothelial basement membrane components and carotid stiffness in mice

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