About: Charles A Stanley

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) ricercatore (it) 研究者 (ja) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy)
rdfs:label	Charles A Stanley (en) Charles A Stanley (es) Charles A Stanley (nl) Charles A Stanley (ast) চার্লস এ স্ট্যানলি (bn)
skos:prefLabel	Charles A Stanley (en) Charles A Stanley (es) Charles A Stanley (nl) Charles A Stanley (ast) চার্লস এ স্ট্যানলি (bn)
name	Charles A Stanley (en) Charles A Stanley (es) Charles A Stanley (nl) Charles A Stanley (ast) চার্লস এ স্ট্যানলি (bn)
Scopus author ID	wds:Q56939652-D7927C2F-0D4B-432F-90E7-1A374173081A
Scopus author ID	7202995121
given name	wds:Q56939652-F53453DC-F1C6-46C8-A8F6-98F53F2264B2
given name	Charles
instance of	wds:Q56939652-8FC968C0-7F16-43D3-B348-353C5A099958
instance of	Homo sapiens
skos:altLabel	Charles Stanley (en)
occupation	wds:Q56939652-6C7A906D-8C5F-4E66-9659-23C6254B5FE1
sex or gender	wds:Q56939652-c74f0aa7-40df-04cd-a2bf-13767dda9959
VIAF ID	wds:Q56939652-A44A759B-93B5-4971-AB59-0C4F98F3A29A
IdRef ID	wds:Q56939652-B3B81A9F-A5E9-4825-95D7-31911129A541
PLWABN ID	wds:Q56939652-52DBAD33-01D6-42B4-9CC5-87B8C1282B8E
family name	wds:Q56939652-6FFD1D0B-A341-4E15-ABDF-025C003F3C79
WorldCat Identities ID (superseded)	wds:Q56939652-2C4960D2-BCB1-480C-B513-A3367ECEC64E
National Library of Israel J9U ID	wds:Q56939652-F04E4129-C381-4E34-AE23-FE02F6D3477D
VIAF ID	http://viaf.org/viaf/313517603
IdRef ID	http://www.idref.fr/160164788/id
occupation	researcher
sex or gender	male
VIAF ID	313517603
IdRef ID	160164788
PLWABN ID	9810620289705606
family name	Stanley
WorldCat Identities ID (superseded)	lccn-n2011185582
National Library of Israel J9U ID	987007404213605171
ORCID iD	wds:Q56939652-D4753501-B440-4B03-8364-361ECA3CD897
ORCID iD	https://orcid.org/0000-0003-4881-9392
ORCID iD	0000-0003-4881-9392
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q56939652
is author of	Structural studies on ADP activation of mammalian glutamate dehydrogenase and the evolution of regulation Familial and sporadic hyperinsulinism: histopathologic findings and segregation analysis support a single autosomal recessive disorder A novel KCNJ11 mutation associated with congenital hyperinsulinism reduces the intrinsic open probability of beta-cell ATP-sensitive potassium channels Elimination of KATP channels in mouse islets results in elevated [U-13C]glucose metabolism, glutaminolysis, and pyruvate cycling but a decreased gamma-aminobutyric acid shunt A signaling role of glutamine in insulin secretion Regulation of leucine-stimulated insulin secretion and glutamine metabolism in isolated rat islets Green tea polyphenols modulate insulin secretion by inhibiting glutamate dehydrogenase. Effects of a GTP-insensitive Mutation of Glutamate Dehydrogenase on Insulin Secretion in Transgenic Mice Protein-sensitive hypoglycemia without leucine sensitivity in hyperinsulinism caused by KATP channel mutations Familial hyperinsulinism caused by an activating glucokinase mutation Hyperinsulinism and hyperammonemia in infants with regulatory mutations of the glutamate dehydrogenase gene Glutamate dehydrogenase: structure, allosteric regulation, and role in insulin homeostasis The structure and allosteric regulation of mammalian glutamate dehydrogenase Multiple phenotypes in phosphoglucomutase 1 deficiency Clinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations Follow-up analysis of genome-wide association data identifies novel loci for type 1 diabetes A genome-wide association study identifies KIAA0350 as a type 1 diabetes gene Mutations in the sulonylurea receptor gene are associated with familial hyperinsulinism in Ashkenazi Jews. Genetic heterogeneity in familial hyperinsulinism. Clinical and molecular characterization of a dominant form of congenital hyperinsulinism caused by a mutation in the high-affinity sulfonylurea receptor Familial leucine-sensitive hypoglycemia of infancy due to a dominant mutation of the beta-cell sulfonylurea receptor Genotype-phenotype correlations in children with congenital hyperinsulinism due to recessive mutations of the adenosine triphosphate-sensitive potassium channel genes A Mutation in the TMD0-L0 Region of Sulfonylurea Receptor-1 (L225P) Causes Permanent Neonatal Diabetes Mellitus (PNDM) Advances in Diagnosis and Treatment of Hyperinsulinism in Infants and Children Comparative genetic analysis of inflammatory bowel disease and type 1 diabetes implicates multiple loci with opposite effects Insights into the structure and regulation of glucokinase from a novel mutation (V62M), which causes maturity-onset diabetes of the young A novel susceptibility locus for type 1 diabetes on Chr12q13 identified by a genome-wide association study Hyperinsulinism in infancy and childhood: when an insulin level is not always enough Clinical features and insulin regulation in infants with a syndrome of prolonged neonatal hyperinsulinism. Insulin-like growth factor binding protein-1 levels in the diagnosis of hypoglycemia caused by hyperinsulinism. Glycemic response to glucagon during fasting hypoglycemia: an aid in the diagnosis of hyperinsulinism. Acute insulin responses to leucine in children with the hyperinsulinism/hyperammonemia syndrome Green Tea Polyphenols Control Dysregulated Glutamate Dehydrogenase in Transgenic Mice by Hijacking the ADP Activation Site Protein-sensitive and fasting hypoglycemia in children with the hyperinsulinism/hyperammonemia syndrome. Mechanism of hyperinsulinism in short-chain 3-hydroxyacyl-CoA dehydrogenase deficiency involves activation of glutamate dehydrogenase Short-chain 3-hydroxyacyl-coenzyme A dehydrogenase associates with a protein super-complex integrating multiple metabolic pathways GLP-1 receptor antagonist exendin-(9-39) elevates fasting blood glucose levels in congenital hyperinsulinism owing to inactivating mutations in the ATP-sensitive K+ channel Mechanisms of Disease: advances in diagnosis and treatment of hyperinsulinism in neonates Exendin-(9-39) corrects fasting hypoglycemia in SUR-1-/- mice by lowering cAMP in pancreatic beta-cells and inhibiting insulin secretion Medium-chain and long-chain acyl CoA dehydrogenase deficiency: clinical, pathologic and ultrastructural differentiation from Reye's syndrome. Pancreatic surgery in infants with Beckwith-Wiedemann syndrome and hyperinsulinism Structures of bovine glutamate dehydrogenase complexes elucidate the mechanism of purine regulation

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