About: Matthijs Verhage

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) researcher (en) 연구원 (ko) 研究者 (ja) araştırmacı (tr) дослідник (uk) эрдэмтэн (mn) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) onderzoeker (nl)
rdfs:label	Matthijs Verhage (en) Matthijs Verhage (es) Matthijs Verhage (nl) Matthijs Verhage (ast)
skos:prefLabel	Matthijs Verhage (en) Matthijs Verhage (es) Matthijs Verhage (nl) Matthijs Verhage (ast)
name	Matthijs Verhage (en) Matthijs Verhage (es) Matthijs Verhage (nl) Matthijs Verhage (ast)
Scopus author ID	wds:Q56905118-C3B8AA59-277B-4F8D-B89E-96E5A969DE74
Scopus author ID	7004678763
given name	wds:Q56905118-38043F7B-B0C5-4ECE-841A-8B01BD070AEB
given name	Matthijs
instance of	wds:Q56905118-9E5D52DB-CF4F-4FFD-B89B-EF128AFC1579
instance of	Homo sapiens
skos:altLabel	M Verhage (en)
Nationale Thesaurus voor Auteursnamen ID	wds:Q56905118-f9af0d56-3cd7-434a-9e42-55e87c43420a
occupation	wds:Q56905118-E6EBBC84-9354-4A59-99C1-466FFB02F120
ISNI	wds:Q56905118-832F624F-BF84-4A76-BAEB-84A5B160CB7E
VIAF ID	wds:Q56905118-E3A2D26D-8358-41FD-AAD4-D228F8F93319
GND ID	wds:Q56905118-d600808a-18dc-4bfc-bbeb-2f7d736ca93d
IdRef ID	wds:Q56905118-7ebd8567-19b3-4b62-94ef-c91f855725aa
WorldCat Identities ID (superseded)	wds:Q56905118-6729178B-3594-4A2B-AF47-45C4B2C1F7AB
Nationale Thesaurus voor Auteursnamen ID	http://data.bibliotheken.nl/id/thes/p080444113
VIAF ID	http://viaf.org/viaf/286057586
GND ID	https://d-nb.info/gnd/1162559373
IdRef ID	http://www.idref.fr/185534430/id
Nationale Thesaurus voor Auteursnamen ID	080444113
occupation	researcher
ISNI	0000000392138965
VIAF ID	286057586
GND ID	1162559373
IdRef ID	185534430
WorldCat Identities ID (superseded)	viaf-286057586
ORCID iD	wds:Q56905118-3C3AEC3A-7C6C-48E0-BBC3-2FE8012C09AD wds:Q56905118-E95C673C-616F-4F3E-91C0-795DBA9E0C0A
ORCID iD	https://orcid.org/0000-0002-2514-0216 https://orcid.org/0000-0002-6085-7503
ORCID iD	0000-0002-2514-0216 0000-0002-6085-7503
is owl:sameAs of	Matthijs Verhage
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q56905118
is author of	Conformational switch of syntaxin-1 controls synaptic vesicle fusion Synaptic assembly of the brain in the absence of neurotransmitter secretion Synaptotagmin interaction with SNAP-25 governs vesicle docking, priming, and fusion triggering. Doc2b is a high-affinity Ca2+ sensor for spontaneous neurotransmitter release Synaptotagmin-1 Docks Secretory Vesicles to Syntaxin-1/SNAP-25 Acceptor Complexes Matrix-dependent local retention of secretory vesicle cargo in cortical neurons. Evaluation of the Ca2+ concentration in purified nerve terminals: relationship between Ca2+ homeostasis and synaptosomal preparation Vesicle trafficking: pleasure and pain from SM genes. Vesicle docking in regulated exocytosis. Dissecting docking and tethering of secretory vesicles at the target membrane. Tetanus toxin and botulinum toxins type A and B inhibit glutamate, gamma-aminobutyric acid, aspartate, and met-enkephalin release from synaptosomes. Clues to the locus of action Bidirectional modulation of classical fear conditioning in mice by 5-HT(1A) receptor ligands with contrasting intrinsic activities. Genome-wide association analysis identifies 13 new risk loci for schizophrenia Differential involvement of the dorsal hippocampus in passive avoidance in C57bl/6J and DBA/2J mice Sheltering behavior and locomotor activity in 11 genetically diverse common inbred mouse strains using home-cage monitoring Deletion of Munc18-1 in 5-HT neurons results in rapid degeneration of the 5-HT system and early postnatal lethality. Rab3a is involved in transport of synaptic vesicles to the active zone in mouse brain nerve terminals. Ca(2+)-induced recruitment of the secretory vesicle protein DOC2B to the target membrane. Multiple Ca2+ sensors in secretion: teammates, competitors or autocrats? DOC2A and DOC2B are sensors for neuronal activity with unique calcium-dependent and kinetic properties. Presynaptic plasticity: the regulation of Ca(2+)-dependent transmitter release. Munc18-1 stabilizes syntaxin 1, but is not essential for syntaxin 1 targeting and SNARE complex formation Munc18-1 expression levels control synapse recovery by regulating readily releasable pool size Fast vesicle fusion in living cells requires at least three SNARE complexes. Functional gene group analysis reveals a role of synaptic heterotrimeric G proteins in cognitive ability Meta-analysis of genome-wide association data of bipolar disorder and major depressive disorder. Phenotypic complexity, measurement bias, and poor phenotypic resolution contribute to the missing heritability problem in genetic association studies Finding the right motivation: genotype-dependent differences in effective reinforcements for spatial learning. Munc18-1 phosphorylation by protein kinase C potentiates vesicle pool replenishment in bovine chromaffin cells. Munc18-1 regulates first-phase insulin release by promoting granule docking to multiple syntaxin isoforms Doc2b is a key effector of insulin secretion and skeletal muscle insulin sensitivity Characterization of the release of cholecystokinin-8 from isolated nerve terminals and comparison with exocytosis of classical transmitters. Munc18-1 mutations that strongly impair SNARE-complex binding support normal synaptic transmission. Functional gene group analysis identifies synaptic gene groups as risk factor for schizophrenia. Laminar and columnar development of barrel cortex relies on thalamocortical neurotransmission Automated analysis of neuronal morphology, synapse number and synaptic recruitment Munc18 and Munc13 regulate early neurite outgrowth Ba2+ replaces Ca2+/calmodulin in the activation of protein phosphatases and in exocytosis of all major transmitters Somatodendritic secretion in oxytocin neurons is upregulated during the female reproductive cycle. A solution to dependency: using multilevel analysis to accommodate nested data Docking of secretory vesicles is syntaxin dependent Munc18-1 in secretion: lonely Munc joins SNARE team and takes control.

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