About: Keith W. Kelley

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American immunophysiologist, researcher and academic

Attributes	Values
rdf:type	Item
owl:sameAs	Keith W. Kelley
description	forskare (sv) ikertzailea (eu) 연구원 (ko) 研究者 (ja) araştırmacı (tr) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) onderzoeker (nl) American immunophysiologist, researcher and academic (en)
rdfs:label	Keith W. Kelley (en) Keith W. Kelley (es) Keith W. Kelley (nl) Keith W. Kelley (sl) Keith W. Kelley (ast)
skos:prefLabel	Keith W. Kelley (en) Keith W. Kelley (es) Keith W. Kelley (nl) Keith W. Kelley (sl) Keith W. Kelley (ast)
name	Keith W. Kelley (en) Keith W. Kelley (es) Keith W. Kelley (nl) Keith W. Kelley (sl) Keith W. Kelley (ast)
Scopus author ID	wds:Q47809179-36CFF3C6-11E1-4766-B99B-632A05E69F99 wds:Q47809179-654D3376-793B-47E6-B017-AB6A2973BEEE
Scopus author ID	56649831000 57193329129
given name	wds:Q47809179-4688B5B9-2748-4C82-8D79-8AE09EFC635F
given name	Keith
image	wds:Q47809179-5E388616-1A0A-491C-8C95-9E9FBEAABFB5
instance of	wds:Q47809179-6265657E-977C-4746-8CF9-4DB3F1270B19
image
instance of	Homo sapiens
Google Knowledge Graph ID	wds:Q47809179-20DA046F-BFB8-4D4B-81E3-EEE1CA6CE91F
Google Knowledge Graph ID	http://g.co/kg/g/11g9r2zv66
Google Knowledge Graph ID	/g/11g9r2zv66
skos:altLabel	Keith Kelley (en)
occupation	wds:Q47809179-41DF2EA3-D3F0-40F2-9C16-443BC3FF45BE
employer	wds:Q47809179-049B850B-2C74-48A0-95CB-BEE069D7F59B wds:Q47809179-6B8C17A8-6219-46E3-BC38-ADD14D2A3524 wds:Q47809179-C141AABA-4C09-4802-8184-73EB95B9DD55 wds:Q47809179-FC022063-C32D-4818-8AD5-D9AC35FB6026
sex or gender	wds:Q47809179-8EE6D58C-04EB-4597-A464-04E59DDE5865
date of birth	wds:Q47809179-74AB3B37-F996-44D2-8BA9-E30B237094DA
educated at	wds:Q47809179-f0df7276-26d9-4874-9d40-874edca2e49a
family name	wds:Q47809179-B4A33577-BA1B-42F9-AA2B-AE9C6426EB7E
occupation	researcher
employer	Shenzhen University National Institute for Agricultural Research University of Illinois at Urbana–Champaign Washington State University
sex or gender	male
date of birth	1947-11-05 00:00:00Z (xsd:dateTime)
educated at	University of Illinois at Urbana–Champaign
family name	Kelley
ORCID iD	wds:Q47809179-9694037F-D462-411A-88D4-63EDCFC8245B
ORCID iD	https://orcid.org/0000-0002-6837-8793
ORCID iD	0000-0002-6837-8793
is about of	Keith W. Kelley https://www.wikidata.org/wiki/Special:EntityData/Q47809179 Keith W. Kelley
is author of	RETRACTED: A new mechanism of neurodegeneration: a proinflammatory cytokine inhibits receptor signaling by a survival peptide Central and peripheral prostaglandins are involved in sickness behavior in birds. Mechanisms of the behavioural effects of cytokines. Uncoupling of interleukin-6 from its signalling pathway by dietary n-3-polyunsaturated fatty acid deprivation alters sickness behaviour in mice. Synergy between tumor necrosis factor alpha and interleukin-1 in the induction of sickness behavior in mice. In vivo and in vitro evidence for the involvement of tumor necrosis factor-alpha in the induction of leptin by lipopolysaccharide Interferon-gamma-induced priming for secretion of superoxide anion and tumor necrosis factor-alpha declines in macrophages from aged rats From inflammation to sickness and depression: when the immune system subjugates the brain Enhancement of coproporphyrinogen III transport into isolated transformed leukocyte mitochondria by ATP. Exaggerated neuroinflammation and sickness behavior in aged mice following activation of the peripheral innate immune system. A novel role of growth hormone and insulin-like growth factor-I. Priming neutrophils for superoxide anion secretion NMDA receptor blockade by ketamine abrogates lipopolysaccharide-induced depressive-like behavior in C57BL/6J mice Inoculation of Bacillus Calmette-Guerin to mice induces an acute episode of sickness behavior followed by chronic depressive-like behavior. Spatio-temporal differences in the profile of murine brain expression of proinflammatory cytokines and indoleamine 2,3-dioxygenase in response to peripheral lipopolysaccharide administration. Bacille Calmette-Guérin inoculation induces chronic activation of peripheral and brain indoleamine 2,3-dioxygenase in mice. Stress and immunity: an integrated view of relationships between the brain and the immune system Proinflammatory cytokine impairment of insulin-like growth factor I-induced protein synthesis in skeletal muscle myoblasts requires ceramide. Tumor necrosis factor alpha inhibits cyclin A expression and retinoblastoma hyperphosphorylation triggered by insulin-like growth factor-I induction of new E2F-1 synthesis Interleukin-1 signaling in mouse astrocytes involves Akt: a study with interleukin-4 and IL-10 Mechanisms of sickness-induced decreases in food-motivated behavior Twenty years of research on cytokine-induced sickness behavior Subdiaphragmatic vagotomy blocks induction of IL-1 beta mRNA in mice brain in response to peripheral LPS Low nociceptor GRK2 prolongs prostaglandin E2 hyperalgesia via biased cAMP signaling to Epac/Rap1, protein kinase Cepsilon, and MEK/ERK. Alcoholism and inflammation: neuroimmunology of behavioral and mood disorders Molecular basis of sickness behavior. Voluntary wheel running reverses age-induced changes in hippocampal gene expression In vitro and in vivo evidence for a role of the P2X7 receptor in the release of IL-1 beta in the murine brain A new concept in neurodegeneration: TNFalpha is a silencer of survival signals Tumor necrosis factor-alpha induces neuronal death by silencing survival signals generated by the type I insulin-like growth factor receptor. Elevated cyclin E levels, inactive retinoblastoma protein, and suppression of the p27(KIP1) inhibitor characterize early development of promyeloid cells into macrophages CSF concentrations of brain tryptophan and kynurenines during immune stimulation with IFN-alpha: relationship to CNS immune responses and depression Cytokines and sickness behavior. Temporal and spatial relationships between lipopolysaccharide-induced expression of Fos, interleukin-1beta and inducible nitric oxide synthase in rat brain. The type 1 TNF receptor and its associated adapter protein, FAN, are required for TNFalpha-induced sickness behavior Different receptor mechanisms mediate the pyrogenic and behavioral effects of interleukin 1

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