About: Andreas Schedl

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) researcher (en) 연구원 (ko) গবেষক (bn) 研究者 (ja) ഗവേഷകൻ (ml) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq)
rdfs:label	Andreas Schedl (en) Andreas Schedl (es) Andreas Schedl (nl) Andreas Schedl (sl) Andreas Schedl (ast)
skos:prefLabel	Andreas Schedl (en) Andreas Schedl (es) Andreas Schedl (nl) Andreas Schedl (sl) Andreas Schedl (ast)
name	Andreas Schedl (en) Andreas Schedl (es) Andreas Schedl (nl) Andreas Schedl (sl) Andreas Schedl (ast)
given name	wds:Q46899614-4B538356-61B0-401F-92F3-0B2565ADAEFA
given name	Andreas
instance of	wds:Q46899614-290B41B5-F01D-46EB-A8C6-780219DB7A2B
instance of	Homo sapiens
doctoral student	wds:Q46899614-305EDFA9-26B5-4597-89DD-873BD3D7EF13 wds:Q46899614-4E1F8780-19B9-4414-B8FF-7D7D497E770C
doctoral student	Glenda Comai Yasmine Neirijnck
occupation	wds:Q46899614-02C94DAC-6B79-4762-B17C-EFDCF21C6554
sex or gender	wds:Q46899614-9273341F-C14B-4BDA-937E-311BD8CF342C
VIAF ID	wds:Q46899614-C0472B23-7E24-48C1-9CD6-60471098D413
IdRef ID	wds:Q46899614-0DF5F2CA-4B40-48DC-91B1-4A63F2940A96
VIAF ID	http://viaf.org/viaf/173597285
IdRef ID	http://www.idref.fr/149085753/id
occupation	researcher
sex or gender	male
VIAF ID	173597285
IdRef ID	149085753
ORCID iD	wds:Q46899614-2A0F3A40-9384-4CCC-AE49-16F3B15D8136
ORCID iD	https://orcid.org/0000-0001-9380-7396
ORCID iD	0000-0001-9380-7396
ResearcherID	wds:Q46899614-1F861133-ECB5-44FA-B095-E29324516B4D
Publons author ID	wds:Q46899614-4bb614c0-667d-4bad-ba8d-83c4de98527d
ResearcherID	F-8823-2015
Publons author ID	2404314
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q46899614
is author of	Pax6 controls progenitor cell identity and neuronal fate in response to graded Shh signaling Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel disease Visceral and subcutaneous fat have different origins and evidence supports a mesothelial source. Deficiency of an enzyme of tyrosine metabolism underlies altered gene expression in newborn liver of lethal albino mice. The Wilms' tumor suppressor WT1: approaches to gene function. Intermediate filament protein nestin is expressed in developing kidney and heart and might be regulated by the Wilms' tumor suppressor Wt1. Sox9 in testis determination The Angiocrine Factor Rspondin3 Is a Key Determinant of Liver Zonation The Sox9 transcription factor determines glial fate choice in the developing spinal cord Sox9 is essential for outer root sheath differentiation and the formation of the hair stem cell compartment WT1 is a key regulator of podocyte function: reduced expression levels cause crescentic glomerulonephritis and mesangial sclerosis The Wilms' tumor gene Wt1 is required for normal development of the retina A splice variant of the Wilms' tumour suppressor Wt1 is required for normal development of the olfactory system The major podocyte protein nephrin is transcriptionally activated by the Wilms' tumor suppressor WT1. YAC transgenic analysis reveals Wilms' tumour 1 gene activity in the proliferating coelomic epithelium, developing diaphragm and limb. Wt1 is not essential for hematopoiesis in the mouse Essential role of Sox9 in the pathway that controls formation of cardiac valves and septa Transgenic mice generated by pronuclear injection of a yeast artificial chromosome. Influence of PAX6 gene dosage on development: overexpression causes severe eye abnormalities. R-spondin1 is essential in sex determination, skin differentiation and malignancy. WT1 controls antagonistic FGF and BMP-pSMAD pathways in early renal progenitors. Expression patterns of the Wtx/Amer gene family during mouse embryonic development Conditional Sox9 ablation reduces chondroitin sulfate proteoglycan levels and improves motor function following spinal cord injury Two splice variants of the Wilms' tumor 1 gene have distinct functions during sex determination and nephron formation The transcription factor Sox9 has essential roles in successive steps of the chondrocyte differentiation pathway and is required for expression of Sox5 and Sox6 Sox9 induces testis development in XX transgenic mice Functional analysis of Sox8 and Sox9 during sex determination in the mouse Aniridia-associated translocations, DNase hypersensitivity, sequence comparison and transgenic analysis redefine the functional domain of PAX6. The transcriptional control of trunk neural crest induction, survival, and delamination Cbx2, a polycomb group gene, is required for Sry gene expression in mice. The reticulocalbin gene maps to the WAGR region in human and to the Small eye Harwell deletion in mouse Chromosome jumping from flanking markers defines the minimal region for alf/hsdr-1 within the albino-deletion complex SOX9 controls epithelial branching by activating RET effector genes during kidney development Renal abnormalities and their developmental origin The cerebellin 4 precursor gene is a direct target of SRY and SOX9 in mice Activation of beta-catenin signaling by Rspo1 controls differentiation of the mammalian ovary. Deletion of long-range regulatory elements upstream of SOX9 causes campomelic dysplasia Coronary vessel development requires activation of the TrkB neurotrophin receptor by the Wilms' tumor transcription factor Wt1. Oncogenicity of the developmental transcription factor Sox9. An Inducible Mouse Model for PAX2-Dependent Glomerular Disease: Insights into a Complex Pathogenesis WT1 maintains adrenal-gonadal primordium identity and marks a population of AGP-like progenitors within the adrenal gland Germ line transmission of yeast artificial chromosomes in transgenic mice. Bone Marrow Transplantation Can Attenuate the Progression of Mesangial Sclerosis Gene interactions in gonadal development.

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