About: Kingston H Mills

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) researcher (en) 연구원 (ko) ricercatore (it) গবেষক (bn) 研究者 (ja) ഗവേഷകൻ (ml) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq)
rdfs:label	Kingston H Mills (en) Kingston H Mills (es) Kingston H Mills (nl) Kingston H Mills (sl) Kingston H Mills (ast)
skos:prefLabel	Kingston H Mills (en) Kingston H Mills (es) Kingston H Mills (nl) Kingston H Mills (sl) Kingston H Mills (ast)
name	Kingston H Mills (en) Kingston H Mills (es) Kingston H Mills (nl) Kingston H Mills (sl) Kingston H Mills (ast)
Scopus author ID	wds:Q45449153-916A94FF-7FD9-4F80-AB7A-EA616FA64ABF
Scopus author ID	7202875495
given name	wds:Q45449153-4733d66d-40b8-ca6e-7955-da06f470c99c
given name	Kingston
instance of	wds:Q45449153-A2B9AA11-C5AA-4EA0-8ECD-7B5EAB74EB86
instance of	Homo sapiens
Loop ID	wds:Q45449153-5A094764-703B-4F6C-B542-A1776DCE864B
Loop ID	24494
skos:altLabel	Kingston Mills (en)
occupation	wds:Q45449153-F473C843-14EA-4BA2-83B4-5A2E28645F5D
employer	wds:Q45449153-06029BC7-2CAF-4933-89AD-B311DA554484 wds:Q45449153-46314EF0-365C-4A67-AB31-49E491920629 wds:Q45449153-4E937AD3-BC61-4F30-B326-89F42E3A02B0 wds:Q45449153-5A88615F-62BB-4435-8801-EFF1E7859AE7 wds:Q45449153-F950E00C-3F79-4746-A08B-B95F3DEF527F
sex or gender	wds:Q45449153-f6112bed-4457-04f4-8e90-7dfde4c14c81
VIAF ID	wds:Q45449153-332BD58D-07A8-4F08-BC90-66C136EFDF54
Library of Congress authority ID	wds:Q45449153-9FA2ADB7-EE98-4FD7-BA82-B4C37A0979E5
educated at	wds:Q45449153-0CEE9A38-82FC-4960-8D85-2D2241F5F0E9 wds:Q45449153-11E5D731-5BDC-451A-908D-DDB2358AA4EF
family name	wds:Q45449153-94D27CE6-13A6-4360-8DF1-C803E0AA884A
WorldCat Identities ID (superseded)	wds:Q45449153-3AC66DB1-299D-4177-879C-E8CE16AFD43D
VIAF ID	http://viaf.org/viaf/5160149235101876690007
Library of Congress authority ID	http://id.loc.gov/authorities/names/no2017046381
occupation	researcher
employer	Maynooth University Francis Crick Institute University College London National Institute for Biological Standards and Control University College Dublin
sex or gender	male
VIAF ID	5160149235101876690007
Library of Congress authority ID	no2017046381
educated at	University College Dublin
family name	Mills
WorldCat Identities ID (superseded)	lccn-no2017046381
ORCID iD	wds:Q45449153-700C7A53-B5A1-40AE-AED5-55426926809F
ORCID iD	https://orcid.org/0000-0003-3646-8222
ORCID iD	0000-0003-3646-8222
ResearcherID	wds:Q45449153-C85B114D-9B2D-45B2-9368-ABF44389737F
Publons author ID	wds:Q45449153-2eb8b468-1c35-4843-8cce-de5473c779b1
ResearcherID	F-5684-2013
Publons author ID	2641338
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q45449153
is author of	Trained immunity: A program of innate immune memory in health and disease. Network analysis of adipose tissue gene expression highlights altered metabolic and regulatory transcriptomic activity in high-fat-diet-fed IL-1RI knockout mice Therapeutic potential of targeting IL-17 Influence of gastrointestinal commensal bacteria on the immune responses that mediate allergy and asthma. Collapse of a fish population after exposure to a synthetic estrogen. Staphylococcus aureus infection of mice expands a population of memory γδ T cells that are protective against subsequent infection. Protection against SIV infection in macaques by immunization with inactivated virus from the BK28 molecular clone, but not with BK28-derived recombinant env and gag proteins A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases. Pathogen-specific T regulatory 1 cells induced in the respiratory tract by a bacterial molecule that stimulates interleukin 10 production by dendritic cells: a novel strategy for evasion of protective T helper type 1 responses by Bordetella pertussi Innate immune memory: a paradigm shift in understanding host defense. Cholera Toxin Promotes the Induction of Regulatory T Cells Specific for Bystander Antigens by Modulating Dendritic Cell Activation Roads to the development of improved pertussis vaccines paved by immunology Whole-cell but not acellular pertussis vaccines induce convulsive activity in mice: evidence of a role for toxin-induced interleukin-1beta in a new murine model for analysis of neuronal side effects of vaccination Relative contribution of Th1 and Th17 cells in adaptive immunity to Bordetella pertussis: towards the rational design of an improved acellular pertussis vaccine. Toll-like receptor 4-mediated innate IL-10 activates antigen-specific regulatory T cells and confers resistance to Bordetella pertussis by inhibiting inflammatory pathology. A critical role for the TLR signaling adapter Mal in alveolar macrophage-mediated protection against Bordetella pertussis. A novel TLR2 agonist from Bordetella pertussis is a potent adjuvant that promotes protective immunity with an acellular pertussis vaccine. Adenylate cycalse toxin of Bordetella pertussis inhibits TLR-induced IRF-1 and IRF-8 activation and IL-12 production and enhances IL-10 through MAPK activation in dendritic cells Pertussis toxin potentiates Th1 and Th2 responses to co-injected antigen: adjuvant action is associated with enhanced regulatory cytokine production and expression of the co-stimulatory molecules B7-1, B7-2 and CD28. Depletion of NK cells results in disseminating lethal infection with Bordetella pertussis associated with a reduction of antigen-specific Th1 and enhancement of Th2, but not Tr1 cells. TLR4 mediates vaccine-induced protective cellular immunity to Bordetella pertussis: role of IL-17-producing T cells. CD11c+CD8 + Dendritic Cells Promote Protective Immunity to Respiratory Infection with Bordetella pertussis T-cell strategies in AIDS vaccines: MHC-restricted T-cell responses to HIV proteins. Interleukin-1 and IL-23 induce innate IL-17 production from gammadelta T cells, amplifying Th17 responses and autoimmunity.

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