About: Marc Remke

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researcher

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rdf:type	Item
description	forskare (sv) researcher (en) 연구원 (ko) ricercatore (it) গবেষক (bn) 研究者 (ja) ഗവേഷകൻ (ml) wetenschapper (nl) дослідник (uk) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq)
rdfs:label	Marc Remke (en) Marc Remke (es) Marc Remke (it) Marc Remke (nl) Marc Remke (sl) Marc Remke (sv) Marc Remke (ast)
skos:prefLabel	Marc Remke (en) Marc Remke (es) Marc Remke (it) Marc Remke (nl) Marc Remke (sl) Marc Remke (sv) Marc Remke (ast)
name	Marc Remke (en) Marc Remke (es) Marc Remke (it) Marc Remke (nl) Marc Remke (sl) Marc Remke (sv) Marc Remke (ast)
given name	wds:Q43153350-157D54A6-E715-4D88-80DF-0006DCDB389A
given name	Marc
instance of	wds:Q43153350-934ABED4-115D-40FD-8F66-A9A5580C124A
instance of	Homo sapiens
occupation	wds:Q43153350-D495FD1E-5435-4FB3-8594-B7D917F08E0D
sex or gender	wds:Q43153350-A092716C-0F6E-4339-8693-EF114ECB1326
VIAF ID	wds:Q43153350-B2802FBC-1751-4E56-9C93-BBB20B803B41
GND ID	wds:Q43153350-307e126b-ca39-403d-b232-15cffc6e70de
WorldCat Identities ID (superseded)	wds:Q43153350-9A704456-FB44-4570-8CA3-216FD9B56ED2
VIAF ID	http://viaf.org/viaf/103891607
GND ID	https://d-nb.info/gnd/140368787
occupation	researcher
sex or gender	male
VIAF ID	103891607
GND ID	140368787
WorldCat Identities ID (superseded)	viaf-103891607
ORCID iD	wds:Q43153350-C0EC8D7D-E30D-4106-9B69-CF18B63D6E9F
Dimensions author ID	wds:Q43153350-0AA2A8CF-B779-49CA-AE95-15E7189E6AF9
ORCID iD	https://orcid.org/0000-0002-9404-9993
ORCID iD	0000-0002-9404-9993
Dimensions author ID	01001656432.42
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q43153350
is author of	Molecular staging of intracranial ependymoma in children and adults Combined hereditary and somatic mutations of replication error repair genes result in rapid onset of ultra-hypermutated cancers Inhibition of BRD4 attenuates tumor cell self-renewal and suppresses stem cell signaling in MYC driven medulloblastoma. Rapid, reliable, and reproducible molecular sub-grouping of clinical medulloblastoma samples Adult and pediatric medulloblastomas are genetically distinct and require different algorithms for molecular risk stratification. Telomerase inhibition abolishes the tumorigenicity of pediatric ependymoma tumor-initiating cells TERT promoter mutations are highly recurrent in SHH subgroup medulloblastoma Genome sequencing of pediatric medulloblastoma links catastrophic DNA rearrangements with TP53 mutations The eEF2 kinase confers resistance to nutrient deprivation by blocking translation elongation Novel oncogene amplifications in tumors from a family with Li-Fraumeni syndrome. Biological and clinical heterogeneity of MYCN-amplified medulloblastoma. Targeting sonic hedgehog-associated medulloblastoma through inhibition of Aurora and Polo-like kinases. The G protein α subunit Gαs is a tumor suppressor in Sonic hedgehog-driven medulloblastoma New Brain Tumor Entities Emerge from Molecular Classification of CNS-PNETs Role of LIM and SH3 protein 1 (LASP1) in the metastatic dissemination of medulloblastoma Divergent clonal selection dominates medulloblastoma at recurrence Aberrant patterns of H3K4 and H3K27 histone lysine methylation occur across subgroups in medulloblastoma. An activated mutant BRAF kinase domain is sufficient to induce pilocytic astrocytoma in mice BRAF gene duplication constitutes a mechanism of MAPK pathway activation in low-grade astrocytomas Functional characterization of a BRAF insertion mutant associated with pilocytic astrocytoma Combined molecular analysis of BRAF and IDH1 distinguishes pilocytic astrocytoma from diffuse astrocytoma. Embryonal tumor with abundant neuropil and true rosettes (ETANTR), ependymoblastoma, and medulloepithelioma share molecular similarity and comprise a single clinicopathological entity Epigenomic alterations define lethal CIMP-positive ependymomas of infancy Pediatric and adult sonic hedgehog medulloblastomas are clinically and molecularly distinct Oncogenic FAM131B-BRAF fusion resulting from 7q34 deletion comprises an alternative mechanism of MAPK pathway activation in pilocytic astrocytoma Fusion of TTYH1 with the C19MC microRNA cluster drives expression of a brain-specific DNMT3B isoform in the embryonal brain tumor ETMR. Molecular subgroups of medulloblastoma: the current consensus Dissecting the genomic complexity underlying medulloblastoma Outcome prediction in pediatric medulloblastoma based on DNA copy-number aberrations of chromosomes 6q and 17q and the MYC and MYCN loci Subgroup-specific structural variation across 1,000 medulloblastoma genomes Molecular subgroups of medulloblastoma: an international meta-analysis of transcriptome, genetic aberrations, and clinical data of WNT, SHH, Group 3, and Group 4 medulloblastomas Subgroup-specific prognostic implications of TP53 mutation in medulloblastoma Cytogenetic prognostication within medulloblastoma subgroups Recurrence patterns across medulloblastoma subgroups: an integrated clinical and molecular analysis Robust molecular subgrouping and copy-number profiling of medulloblastoma from small amounts of archival tumour material using high-density DNA methylation arrays TP53 mutation is frequently associated with CTNNB1 mutation or MYCN amplification and is compatible with long-term survival in medulloblastoma. Genome sequencing of SHH medulloblastoma predicts genotype-related response to smoothened inhibition Risk stratification of childhood medulloblastoma in the molecular era: the current consensus Characterization of novel biomarkers in selecting for subtype specific medulloblastoma phenotypes. Medulloblastoma molecular dissection: the way toward targeted therapy ATM regulates 3-methylpurine-DNA glycosylase and promotes therapeutic resistance to alkylating agents G-protein coupled receptor expression patterns delineate medulloblastoma subgroups

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