About: Johannes Grillari

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	researcher (en) গবেষক (bn) ഗവേഷകൻ (ml) дослідник (uk) հետազոտող (hy) hulumtues (sq) onderzoeker (nl) österreichischer Traumatologe (de)
rdfs:label	Johannes Grillari (de) Johannes Grillari (en) Johannes Grillari (es) Johannes Grillari (fr) Johannes Grillari (nl) Johannes Grillari (sl) Johannes Grillari (ast) Johannes Grillari (bar)
skos:prefLabel	Johannes Grillari (de) Johannes Grillari (en) Johannes Grillari (es) Johannes Grillari (fr) Johannes Grillari (nl) Johannes Grillari (sl) Johannes Grillari (ast) Johannes Grillari (bar)
name	Johannes Grillari (de) Johannes Grillari (en) Johannes Grillari (es) Johannes Grillari (fr) Johannes Grillari (nl) Johannes Grillari (sl) Johannes Grillari (ast) Johannes Grillari (bar)
Scopus author ID	wds:Q42775711-7A1BB55B-43ED-497B-BEC2-76CD29732740
Scopus author ID	6507010731
given name	wds:Q42775711-D93F2FCE-623E-4C8B-8C75-7F9027D84772
given name	Johannes
instance of	wds:Q42775711-E194769B-2FB2-46A9-9EEB-10D7E61F0F3C
instance of	Homo sapiens
Loop ID	wds:Q42775711-019C2B29-7525-449B-AB76-253A1077BEBD
Loop ID	41480
occupation	wds:Q42775711-E5F5ACA9-F0E7-4A67-9CF1-B52D6011F72F
languages spoken, written or signed	wds:Q42775711-4E26CA5F-60DE-436B-97A0-02A1531B688C
sex or gender	wds:Q42775711-EBE9CA55-54D9-4839-98FB-CDD73D1D47F3
country of citizenship	wds:Q42775711-A62550E8-519F-4D39-ACD4-483237D1AD5D
date of birth	wds:Q42775711-8457A5B7-D510-43E3-9B56-7FB43F259AD8
educated at	wds:Q42775711-4295093c-4fd5-6969-9c86-a229de29fa0e
occupation	traumatologist
languages spoken, written or signed	German
sex or gender	male
country of citizenship	Austria
date of birth	1969-02-09 00:00:00Z (xsd:dateTime)
educated at	University of Natural Resources and Life Sciences, Vienna
ORCID iD	wds:Q42775711-82A3970A-5B71-4382-9685-D6A4BDBCC4AA
ORCID iD	https://orcid.org/0000-0001-5474-6332
ORCID iD	0000-0001-5474-6332
ResearcherID	wds:Q42775711-62A48A26-55F3-439A-8D80-EBD9BD31BDEC
Publons author ID	wds:Q42775711-edbe89f7-b368-4379-8265-add465f4b6d7
ResearcherID	B-2967-2011
Publons author ID	2811293
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q42775711 https://de.wikipedia.org/wiki/Johannes_Grillari
is author of	Early embryonic lethality of mice lacking the essential protein SNEV CHO microRNA engineering is growing up: recent successes and future challenges miR-17, miR-19b, miR-20a, and miR-106a are down-regulated in human aging Upregulation of miR-24 is associated with a decreased DNA damage response upon etoposide treatment in highly differentiated CD8(+) T cells sensitizing them to apoptotic cell death Differentially circulating miRNAs after recent osteoporotic fractures can influence osteogenic differentiation. Next-generation sequencing of the Chinese hamster ovary microRNA transcriptome: Identification, annotation and profiling of microRNAs as targets for cellular engineering miR-17-92 cluster: ups and downs in cancer and aging Vesicular Galectin-3 levels decrease with donor age and contribute to the reduced osteo-inductive potential of human plasma derived extracellular vesicles Secretion of microvesicular miRNAs in cellular and organismal aging. Erratum: Vesicular Galectin-3 levels decrease with donor age and contribute to the reduced osteo-inductive potential of human plasma derived extracellular vesicles Contributions of DNA interstrand cross-links to aging of cells and organisms Generation of induced pluripotent stem cells from urine Generation of human induced pluripotent stem cells from urine samples. SNEV is an evolutionarily conserved splicing factor whose oligomerization is necessary for spliceosome assembly Identification of evolutionarily conserved genetic regulators of cellular aging MicroRNA-26 family is required for human adipogenesis and drives characteristics of brown adipocytes Alterations in fatty acid utilization and an impaired antioxidant defense mechanism are early events in podocyte injury: a proteomic analysis Interaction of U-box E3 ligase SNEV with PSMB4, the beta7 subunit of the 20 S proteasome Ubiquitous overexpression of the DNA repair factor dPrp19 reduces DNA damage and extends Drosophila life span SNEV(Prp19/PSO4) deficiency increases PUVA-induced senescence in mouse skin SNEVhPrp19/hPso4 Regulates Adipogenesis of Human Adipose Stromal Cells ATM-dependent phosphorylation of SNEVhPrp19/hPso4 is involved in extending cellular life span and suppression of apoptosis Haploinsufficiency of senescence evasion factor causes defects of hematopoietic stem cells functions SNEV overexpression extends the life span of human endothelial cells Chinese hamster genome sequenced from sorted chromosomes. Construction of a stability landscape of the CH3 domain of human IgG1 by combining directed evolution with high throughput sequencing Prediction of transcribed PIWI-interacting RNAs from CHO RNAseq data Analysis of microRNA transcription and post-transcriptional processing by Dicer in the context of CHO cell proliferation Stable overexpression of miR-17 enhances recombinant protein production of CHO cells Dynamic mRNA and miRNA profiling of CHO-K1 suspension cell cultures. Unraveling the Chinese hamster ovary cell line transcriptome by next-generation sequencing Computational identification of microRNA gene loci and precursor microRNA sequences in CHO cell lines Utilization and evaluation of CHO-specific sequence databases for mass spectrometry based proteomics. UVB-Induced Senescence of Human Dermal Fibroblasts Involves Impairment of Proteasome and Enhanced Autophagic Activity. Oncostatin M is a novel inhibitor of TGF-β1-induced matricellular protein expression. A screening method to assess biological effects of microRNA overexpression in Chinese hamster ovary cells Endogenous microRNA clusters outperform chimeric sequence clusters in Chinese hamster ovary cells miRNAs--pathway engineering of CHO cell factories that avoids translational burdening.

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