About: David A. Walsh

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About: David A. Walsh Goto Sponge NotDistinct Permalink

An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

British rheumatologist

Attributes	Values
rdf:type	Item
description	wetenschapper (nl) дослідник (uk) հետազոտող (hy) British rheumatologist (en)
rdfs:label	David A Walsh (nl) David A Walsh (ast) ডেভিড এ ওয়ালশ (bn) David A Walsh (sl) David A. Walsh (en) David A. Walsh (es)
skos:prefLabel	David A Walsh (nl) David A Walsh (ast) ডেভিড এ ওয়ালশ (bn) David A Walsh (sl) David A. Walsh (en) David A. Walsh (es)
name	David A Walsh (nl) David A Walsh (ast) ডেভিড এ ওয়ালশ (bn) David A Walsh (sl) David A. Walsh (en) David A. Walsh (es)
Scopus author ID	wds:Q42395833-4D4D91CF-FD85-41CC-8258-F83BE5AA73AC
Scopus author ID	55979750800
given name	wds:Q42395833-45238E5E-26D0-4ABC-B7A0-4F7CCAEFB94F
given name	David
instance of	wds:Q42395833-911A49B5-D001-46FE-A9BA-BCDF6C378CC4
instance of	Homo sapiens
skos:altLabel	David Walsh (en)
occupation	wds:Q42395833-9858B7E4-696B-4FB4-BFE6-8D96FE9C71C7
employer	wds:Q42395833-8A4C48CF-9E05-4267-A704-FB3B0BC7A893 wds:Q42395833-C92D114F-5C66-4FAF-A6BC-C4D265A2E894
University of Barcelona authority ID	wds:Q42395833-E87AA459-A847-4709-B9E6-BED7A6BCA8AB
NUKAT ID	wds:Q42395833-2C544DD3-6178-4659-A4BD-17E1A1D3BC76
University of Barc...D (former scheme)	wds:Q42395833-7B1DDC1D-780E-488A-A4BB-659A6A5BDFB8
sex or gender	wds:Q42395833-2DABB1C6-5C24-4C7F-BA6B-41ECD1DC0A55
ISNI	wds:Q42395833-5E55740B-C254-416A-9C15-77D91808156C
VIAF ID	wds:Q42395833-49DFEB71-EB0F-457A-9A58-6148C355410E
Library of Congress authority ID	wds:Q42395833-6F16962F-B6DA-44B6-B96C-91DA605D433B
IdRef ID	wds:Q42395833-B49232E4-1CCC-4F54-B192-4D5FE6499520
family name	wds:Q42395833-3FDCF8BB-3768-42CC-9B6C-A3179FF840DE
WorldCat Identities ID (superseded)	wds:Q42395833-03C49C7E-2B39-4020-81FA-21C33ABE38D9
CANTIC ID	wds:Q42395833-5E1F3521-8EF2-4A82-87BE-8DC513CF3105
VIAF ID	http://viaf.org/viaf/14597207
Library of Congress authority ID	http://id.loc.gov/authorities/names/no2008157638
IdRef ID	http://www.idref.fr/129722472/id
occupation	rheumatologist
employer	Sherwood Forest Hospitals NHS Foundation Trust University of Nottingham
University of Barcelona authority ID	981058529846006706
NUKAT ID	n2009147617
University of Barc...D (former scheme)	a1261523
sex or gender	male
ISNI	0000000117916208
VIAF ID	14597207
Library of Congress authority ID	no2008157638
IdRef ID	129722472
family name	Walsh
WorldCat Identities ID (superseded)	lccn-no2008157638
CANTIC ID	981058529846006706
ORCID iD	wds:Q42395833-2BCA6008-FC5B-4554-8884-5AA466955BD3
ORCID iD	https://orcid.org/0000-0002-6316-5692
ORCID iD	0000-0002-6316-5692
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q42395833
is author of	Substance P in intervertebral discs. Binding sites on vascular endothelium of the human annulus fibrosus Osteoprotegerin reduces the development of pain behaviour and joint pathology in a model of osteoarthritis Pain phenotype in patients with knee osteoarthritis: classification and measurement properties of painDETECT and self-report Leeds assessment of neuropathic symptoms and signs scale in a cross-sectional study. Combined effect of bradykinin B2 and neurokinin-1 receptor activation on endothelial cell proliferation in acute synovitis British Society for Rheumatology and british health professionals in Rheumatology guideline for the management of rheumatoid arthritis (the first two years). Antiinflammatory agents. 3. Synthesis and pharmacological evaluation of 2-amino-3-benzoylphenylacetic acid and analogs Performance problems of patients with chronic low-back pain and the measurement of patient-centered outcome. AT1 receptor characteristics of angiotensin analogue binding in human synovium. Angiogenesis and nerve growth factor at the osteochondral junction in rheumatoid arthritis and osteoarthritis. Neurovascular invasion at the osteochondral junction and in osteophytes in osteoarthritis Increased vascular penetration and nerve growth in the meniscus: a potential source of pain in osteoarthritis Contributions of angiogenesis to inflammation, joint damage, and pain in a rat model of osteoarthritis Mechanisms and targets of angiogenesis and nerve growth in osteoarthritis. Inflammation and angiogenesis in osteoarthritis Monoarthritis in the rat knee induces bilateral and time-dependent changes in substance P and calcitonin gene-related peptide immunoreactivity in the spinal cord Angiogenesis in the synovium and at the osteochondral junction in osteoarthritis Interstitial vascularity in fibrosing alveolitis Hand and foot surgery rates in rheumatoid arthritis have declined from 1986 to 2011, but large-joint replacement rates remain unchanged: results from two UK inception cohorts Research bureaucracy in the United Kingdom: ethics committees have important roles in research Autoradiographic localization and analysis of endothelin-1 binding sites in human synovial tissue Angiogenesis in the pathogenesis of inflammatory joint and lung diseases Pain beliefs and perceived physical disability of patients with chronic low back pain Osteochondral angiogenesis and increased protease inhibitor expression in OA. Sequential development of angiotensin receptors and angiotensin I converting enzyme during angiogenesis in the rat subcutaneous sponge granuloma. Remission in early rheumatoid arthritis: predicting treatment response Mechanisms, impact and management of pain in rheumatoid arthritis Baseline factors predicting change from the initial DMARD treatment during the first 2 years of rheumatoid arthritis: experience in the ERAN inception cohort Augmented pain behavioural responses to intra-articular injection of nerve growth factor in two animal models of osteoarthritis Design, study quality and evidence of analgesic efficacy in studies of drugs in models of OA pain: a systematic review and a meta-analysis Increased function of pronociceptive TRPV1 at the level of the joint in a rat model of osteoarthritis pain Blocking the tropomyosin receptor kinase A (TrkA) receptor inhibits pain behaviour in two rat models of osteoarthritis Cannabinoid CB2 receptors regulate central sensitization and pain responses associated with osteoarthritis of the knee joint Pathophysiological mechanisms of angiogenesis. Angiogenesis in osteoarthritis and spondylosis: successful repair with undesirable outcomes. Angiogenesis in osteoarthritis.

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