About: Tao Jin

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher (ORCID 0000-0001-9039-0628)

Attributes	Values
rdf:type	Item
description	ikertzailea (eu) 연구원 (ko) গবেষক (bn) 研究者 (ja) эрдэмтэн (mn) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) onderzoeker (nl) researcher (ORCID 0000-0001-9039-0628) (en)
rdfs:label	Tao Jin (en) Tao Jin (es) Tao Jin (nl) Tao Jin (ast) Tao Jin (sl)
skos:prefLabel	Tao Jin (en) Tao Jin (es) Tao Jin (nl) Tao Jin (ast) Tao Jin (sl)
name	Tao Jin (en) Tao Jin (es) Tao Jin (nl) Tao Jin (ast) Tao Jin (sl)
Scopus author ID	wds:Q41127272-2C7D7B49-89DE-4173-9B72-271FEAA9EDEE
Scopus author ID	35792221600
instance of	wds:Q41127272-4247EDC5-72DA-4D26-8B2C-FA7C22588285
instance of	Homo sapiens
Loop ID	wds:Q41127272-EF1B2CAA-27B1-4ABC-9334-D9FD0B599BE0
Loop ID	290268
occupation	wds:Q41127272-681E226E-E189-496C-B0B0-18981E8A7795
employer	wds:Q41127272-6D806206-00F8-444C-98D6-E1F59722582D wds:Q41127272-84AEAB3A-E1DD-4EF0-9E01-182A79279D4D wds:Q41127272-B94B0B29-A31E-4BDE-8E9B-285A3157E66E wds:Q41127272-F3C22CF7-6D0B-4893-A584-A265AD5DAA7C
ISNI	wds:Q41127272-DB2B2285-55C7-4E3D-A687-484007DB45C0
VIAF ID	wds:Q41127272-3A56F33B-7552-4D08-A92F-BDCFA8AB6FD6
Libris-URI	wds:Q41127272-a422a4c8-295c-4561-82c7-04979881c11f
date of birth	wds:Q41127272-32B43029-3F10-42A4-91A9-524D1F221D90
WorldCat Identities ID (superseded)	wds:Q41127272-179AE924-8162-4A69-81A1-E3CEE0022C07
SELIBR ID	wds:Q41127272-1D1331C5-A07B-4D13-B932-DEBA96902563
VIAF ID	http://viaf.org/viaf/2447557
Libris-URI	https://libris.kb.se/zw9cgvth2lqqs6d
SELIBR ID	http://libris.kb.se/resource/auth/253215
occupation	researcher
employer	Sahlgrenska University Hospital
ISNI	0000000054730923
VIAF ID	2447557
Libris-URI	zw9cgvth2lqqs6d
date of birth	1973-01-01 00:00:00Z (xsd:dateTime)
WorldCat Identities ID (superseded)	viaf-2447557
SELIBR ID	253215
ORCID iD	wds:Q41127272-4B3C652C-1BAF-4646-B1F7-4279878C79C8
ORCID iD	https://orcid.org/0000-0001-9039-0628
ORCID iD	0000-0001-9039-0628
ResearcherID	wds:Q41127272-FDD402BE-1EB5-41E8-B47C-871D06F1F7B4
Publons author ID	wds:Q41127272-8c295468-e0bc-42f3-b19f-2baed57a3eeb
ResearcherID	K-3323-2015
Publons author ID	1279100
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q41127272
is author of	Staphylococcus aureus resists human defensins by production of staphylokinase, a novel bacterial evasion mechanism. Activation of plasminogen by staphylokinase reduces the severity of Staphylococcus aureus systemic infection Surface proteins of Staphylococcus aureus play an important role in experimental skin infection TNF Production in Macrophages Is Genetically Determined and Regulates Inflammatory Disease in Rats Fatal outcome of bacteraemic patients caused by infection with staphylokinase-deficient Staphylococcus aureus strains Plasminogen activation by staphylokinase enhances local spreading of S. aureus in skin infections RAGE Deficiency Impairs Bacterial Clearance in Murine Staphylococcal Sepsis, but Has No Significant Impact on Staphylococcal Septic Arthritis Identification of a novel chemokine-dependent molecular mechanism underlying rheumatoid arthritis-associated autoantibody-mediated bone loss The combination of a tumor necrosis factor inhibitor and antibiotic alleviates staphylococcal arthritis and sepsis in mice CTLA4 Immunoglobulin but Not Anti-Tumor Necrosis Factor Therapy Promotes Staphylococcal Septic Arthritis in Mice. Antibiotic-killed Staphylococcus aureus induces destructive arthritis in mice Staphylokinase promotes the establishment of Staphylococcus aureus skin infections while decreasing disease severity Sulfatide attenuates experimental Staphylococcus aureus sepsis through a CD1d-dependent pathway. Relationship between elevated cerebrospinal fluid levels of plasminogen activator inhibitor 1 and neuronal destruction in patients with neuropsychiatric systemic lupus erythematosus IL-1 Receptor Antagonist Treatment Aggravates Staphylococcal Septic Arthritis and Sepsis in Mice. Biofilm formation by Staphylococcus aureus isolates from skin and soft tissue infections Staphylokinase Control of Staphylococcus aureus Biofilm Formation and Detachment Through Host Plasminogen Activation Tranexamic acid, an inhibitor of plasminogen activation, aggravates staphylococcal septic arthritis and sepsis Soluble E-cadherin in systemic lupus erythematosus The role of urokinase in innate immunity against Staphylococcus aureus Decreased serum levels of TGF-β1 are associated with renal damage in female patients with systemic lupus erythematosus. Galectin-3 Is a Target for Proteases Involved in the Virulence of Staphylococcus aureus Single-stranded polyinosinic acid oligonucleotides trigger leukocyte production of proteins belonging to fibrinolytic and coagulation cascades Human skin commensals augment Staphylococcus aureus pathogenesis Tissue Plasminogen Activator Coating on Implant Surfaces Reduces Staphylococcus aureus Biofilm Formation. Deficiency of the Complement Component 3 but Not Factor B Aggravates Staphylococcus aureus Septic Arthritis in Mice. Urokinase-type plasminogen activator, an endogenous antibiotic Impact of cell wall peptidoglycan O-acetylation on the pathogenesis of Staphylococcus aureus in septic arthritis. Radiological features of experimental staphylococcal septic arthritis by micro computed tomography scan Pre-treatment with IL2 gene therapy alleviates Staphylococcus aureus arthritis in mice Staphylokinase reduces plasmin formation by endogenous plasminogen activators Linkage between endosomal escape of LNP-mRNA and loading into EVs for transport to other cells The Expression of von Willebrand Factor-Binding Protein Determines Joint-Invading Capacity of Staphylococcus aureus, a Core Mechanism of Septic Arthritis Both anti-TNF and CTLA4 Ig treatments attenuate the disease severity of staphylococcal dermatitis in mice. A novel mouse model for septic arthritis induced by Pseudomonas aeruginosa The YIN and YANG of lipoproteins in developing and preventing infectious arthritis by Staphylococcus aureus Reply to Bouchiat et al

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