About: Peter Jordan

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molecular oncobiologist

Attributes	Values
rdf:type	Item
description	biochemicus (nl) molecular oncobiologist (en)
rdfs:label	Peter Jordan (en) Peter Jordan (es) Peter Jordan (nl) Peter Jordan (sq) Peter Jordan (ast) পিটার জর্ডান (bn)
skos:prefLabel	Peter Jordan (en) Peter Jordan (es) Peter Jordan (nl) Peter Jordan (sq) Peter Jordan (ast) পিটার জর্ডান (bn)
name	Peter Jordan (en) Peter Jordan (es) Peter Jordan (nl) Peter Jordan (sq) Peter Jordan (ast) পিটার জর্ডান (bn)
Scopus author ID	wds:Q39965078-38E7FB2C-23C5-4432-BD69-6D91BE18A233
Scopus author ID	28267819400
given name	wds:Q39965078-BA57C215-E63B-4E2D-AEA9-8F65BD67E040
given name	Peter
instance of	wds:Q39965078-1ACFD3FC-118B-49C3-B961-E22FF9507B1E
instance of	Homo sapiens
CIÊNCIAVITAE ID	wds:Q39965078-1646BEA7-53F9-4D21-9B8A-DDA71C9A2D9E
CIÊNCIAVITAE ID	A01A-1F55-88DB
occupation	wds:Q39965078-582C5CF1-F115-4BE4-9455-A7E06CA4D1D5
employer	wds:Q39965078-15139EC2-3B52-42EE-8318-73CD4D0F047D wds:Q39965078-AE50C250-FF6C-47AA-B439-9131B0DD4B28 wds:Q39965078-E2B254F0-6409-4F7C-BD49-ED3F7EC22751
place of birth	wds:Q39965078-81d07740-41bd-31ea-7f9d-a64515684d74
sex or gender	wds:Q39965078-CBE536E1-6F9E-4B4B-A682-C95AD451DA8F
VIAF ID	wds:Q39965078-72E0838A-1F09-4688-AD0B-DC8A3D2418E8
IdRef ID	wds:Q39965078-3B88BAC8-0C4D-4E68-A32A-50182D471454
date of birth	wds:Q39965078-34a87f6c-4c31-e20a-ea2a-25043e14e93b
educated at	wds:Q39965078-19bab720-4760-cb92-caa7-05d4abcd0a2c
family name	wds:Q39965078-017A5A60-62BF-4A00-844F-0F896542CBBC
VIAF ID	http://viaf.org/viaf/47155707014522410185
IdRef ID	http://www.idref.fr/236095188/id
occupation	biochemist
employer	German Cancer Research Center National Institute of Health Dr. Ricardo Jorge University of Göttingen
place of birth	West Germany
sex or gender	male
VIAF ID	47155707014522410185
IdRef ID	236095188
date of birth	1962-01-01 00:00:00Z (xsd:dateTime)
educated at	Heidelberg University
family name	Jordan
ORCID iD	wds:Q39965078-DB48EBF6-C730-4E2D-9871-00E91725B48D
ORCID iD	https://orcid.org/0000-0002-1425-9211
ORCID iD	0000-0002-1425-9211
ResearcherID	wds:Q39965078-E0C39946-3DD7-4627-877D-E90C5D5B6B02
Publons author ID	wds:Q39965078-85e636d2-e066-4108-99e0-4dc29b43cae7
ResearcherID	G-9335-2012
Publons author ID	1672380
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q39965078
is author of	Mixed lineage kinase 3 gene mutations in mismatch repair deficient gastrointestinal tumours Targeting of adenovirus E1A and E4-ORF3 proteins to nuclear matrix-associated PML bodies. Protein kinase WNK3 increases cell survival in a caspase-3-dependent pathway Emerging roles for WNK kinases in cancer. Posttranscriptional Regulation of Splicing Factor SRSF1 and Its Role in Cancer Cell Biology Posttranscriptional Regulation and RNA Binding Proteins in Cancer Biology Tumor-related alternatively spliced Rac1b is not regulated by Rho-GDP dissociation inhibitors and exhibits selective downstream signaling Protein kinase WNK2 inhibits cell proliferation by negatively modulating the activation of MEK1/ERK1/2 In vivo evidence that TATA-binding protein/SL1 colocalizes with UBF and RNA polymerase I when rRNA synthesis is either active or inactive Regulatory Crosstalk by Protein Kinases on CFTR Trafficking and Activity Silencing of the tumor suppressor gene WNK2 is associated with upregulation of MMP2 and JNK in gliomas Rac1 signaling modulates BCL-6-mediated repression of gene transcription. Loss of WNK2 expression by promoter gene methylation occurs in adult gliomas and triggers Rac1-mediated tumour cell invasiveness WNK2 modulates MEK1 activity through the Rho GTPase pathway The topography of chromosomes and genes in the nucleus. Antagonistic SR proteins regulate alternative splicing of tumor-related Rac1b downstream of the PI3-kinase and Wnt pathways. Increased Rac1b expression sustains colorectal tumor cell survival Cisplatin inhibits synthesis of ribosomal RNA in vivo Antagonistic regulation of cystic fibrosis transmembrane conductance regulator cell surface expression by protein kinases WNK4 and spleen tyrosine kinase B-Raf(V600E) cooperates with alternative spliced Rac1b to sustain colorectal cancer cell survival Three-way translocation (X;20;16)(p11;q13;q23) in essential thrombocythemia implicates NFATC2 in dysregulation of CSF2 expression and megakaryocyte proliferation. Expression of Rac1b stimulates NF-kappaB-mediated cell survival and G1/S progression Molecular mechanisms involved in cisplatin cytotoxicity. PCR amplification introduces errors into mononucleotide and dinucleotide repeat sequences Expression of tumor-related Rac1b antagonizes B-Raf-induced senescence in colorectal cells. Contribution of casein kinase 2 and spleen tyrosine kinase to CFTR trafficking and protein kinase A-induced activity. Unmasking the role of KRAS and BRAF pathways in MSI colorectal tumors. Functional analysis of splicing mutations in the IDS gene and the use of antisense oligonucleotides to exploit an alternative therapy for MPS II. Morphological and ultrastructural effects of microcystin-LR from Microcystis aeruginosa extract on a kidney cell line Rac1, but not Rac1B, stimulates RelB-mediated gene transcription in colorectal cancer cells Phosphorylation of SRSF1 by SRPK1 regulates alternative splicing of tumor-related Rac1b in colorectal cells Therapeutic strategies based on modified U1 snRNAs and chaperones for Sanfilippo C splicing mutations A missense mutation in the APC tumor suppressor gene disrupts an ASF/SF2 splicing enhancer motif and causes pathogenic skipping of exon 14. Beyond COX-inhibition: 'side-effects' of ibuprofen on neoplastic development and progression. Protein kinase WNK1 promotes cell surface expression of glucose transporter GLUT1 by regulating a Tre-2/USP6-BUB2-Cdc16 domain family member 4 (TBC1D4)-Rab8A complex. Microcystin-LR activates the ERK1/2 kinases and stimulates the proliferation of the monkey kidney-derived cell line Vero-E6. Gene cloning at the computer screen: possibilities and problems The Rac1 splice form Rac1b favors mouse colonic mucosa regeneration and contributes to intestinal cancer progression WNK2 Inhibits Autophagic Flux in Human Glioblastoma Cell Line

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