About: Jordi Alberch

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) ricercatore (it) গবেষক (bn) 研究者 (ja) ഗവേഷകൻ (ml) araştırmacı (tr) wetenschapper (nl) дослідник (uk) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq)
rdfs:label	Jordi Alberch (en) Jordi Alberch (es) Jordi Alberch (nl) Jordi Alberch (sl) Jordi Alberch (ast)
skos:prefLabel	Jordi Alberch (en) Jordi Alberch (es) Jordi Alberch (nl) Jordi Alberch (sl) Jordi Alberch (ast)
name	Jordi Alberch (en) Jordi Alberch (es) Jordi Alberch (nl) Jordi Alberch (sl) Jordi Alberch (ast)
Scopus author ID	wds:Q39933641-88719BA6-86BA-4A77-8F9A-89E6B5F4E154
Scopus author ID	7003644890
given name	wds:Q39933641-53D99563-6145-4FA9-8595-F97EB08DDCAE
given name	Jordi
instance of	wds:Q39933641-F4DBB7A9-F635-46FA-B1D9-0DD4C4DCF7A6
instance of	Homo sapiens
occupation	wds:Q39933641-F150E75C-5D12-460D-B924-D297D50CB448
employer	wds:Q39933641-00455161-B81E-4FA6-B6BD-6DA14BC97A6F
University of Barcelona authority ID	wds:Q39933641-602995AC-68B2-4CA4-9735-6156B3F6470D
University of Barc...D (former scheme)	wds:Q39933641-808ACDBB-8188-471F-A428-DE61C9897C72
sex or gender	wds:Q39933641-218FC65B-59AF-492F-A692-99B57FD6C45E
ISNI	wds:Q39933641-883C951A-3362-4B88-98AF-CCEE307C28B7
VIAF ID	wds:Q39933641-0121C483-92A7-47E0-9B55-8A36C950C618
date of birth	wds:Q39933641-5FE54D8D-67E8-411F-8D42-D65C557D99DF
educated at	wds:Q39933641-5C5571D1-8E8E-4794-833E-9E418E2C0726
WorldCat Identities ID (superseded)	wds:Q39933641-7335EBA4-617B-49EA-892B-94C3A0861863
National Library of Spain ID	wds:Q39933641-A2F46C54-42B6-43EC-9A93-6BF3C0192251
CANTIC ID	wds:Q39933641-0289734F-C53A-429C-A744-54C4546D85CE
VIAF ID	http://viaf.org/viaf/87614932
National Library of Spain ID	http://datos.bne.es/resource/XX824003
occupation	researcher
employer	University of Barcelona
University of Barcelona authority ID	981058507476706706
University of Barc...D (former scheme)	a1034391
sex or gender	male
ISNI	0000000060895468
VIAF ID	87614932
date of birth	1959-01-01 00:00:00Z (xsd:dateTime)
educated at	University of Barcelona
WorldCat Identities ID (superseded)	viaf-87614932
National Library of Spain ID	XX824003
CANTIC ID	981058507476706706
ORCID iD	wds:Q39933641-6D5AA20E-C9B1-45C2-A3F7-F1416C7144F2
ORCID iD	https://orcid.org/0000-0002-8684-2721
ORCID iD	0000-0002-8684-2721
ResearcherID	wds:Q39933641-F51200F1-2D9B-4FDB-B27D-61B849E18BD1
Publons author ID	wds:Q39933641-bdb68148-e6a2-4055-ac2f-523e6b128e3d
ResearcherID	K-5702-2014
Publons author ID	2493191
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q39933641
is author of	Nolz1 promotes striatal neurogenesis through the regulation of retinoic acid signaling Effect of opioids on acetylcholine release evoked by K+ or glutamic acid from rat neostriatal slices. Brain-derived neurotrophic factor modulates the severity of cognitive alterations induced by mutant huntingtin: involvement of phospholipaseCgamma activity and glutamate receptor expression Altered cholesterol homeostasis contributes to enhanced excitotoxicity in Huntington's disease. Cystamine and cysteamine increase brain levels of BDNF in Huntington disease via HSJ1b and transglutaminase Neuroprotection by GDNF-secreting stem cells in a Huntington's disease model: optical neuroimage tracking of brain-grafted cells. Brain-derived neurotrophic factor, neurotrophin-3 and neurotrophin-4/5 differentially regulate the phenotype and prevent degenerative changes in striatal projection neurons after excitotoxicity in vivo. Regulation of hippocampal cGMP levels as a candidate to treat cognitive deficits in Huntington's disease Long-term memory deficits in Huntington's disease are associated with reduced CBP histone acetylase activity. Intrastriatal grafting of a GDNF-producing cell line protects striatonigral neurons from quinolinic acid excitotoxicity in vivo. Loss of striatal type 1 cannabinoid receptors is a key pathogenic factor in Huntington's disease. Effect of glatiramer acetate (Copaxone) on the immunophenotypic and cytokine profile and BDNF production in multiple sclerosis: a longitudinal study. BMP-2 and cAMP elevation confer locus coeruleus neurons responsiveness to multiple neurotrophic factors Mutant huntingtin impairs the post-Golgi trafficking of brain-derived neurotrophic factor but not its Val66Met polymorphism. Brain-derived neurotrophic factor regulates the onset and severity of motor dysfunction associated with enkephalinergic neuronal degeneration in Huntington's disease. Brain-derived neurotrophic factor modulates dopaminergic deficits in a transgenic mouse model of Huntington's disease Striatal-enriched protein tyrosine phosphatase expression and activity in Huntington's disease: a STEP in the resistance to excitotoxicity Dopaminergic and glutamatergic signaling crosstalk in Huntington's disease neurodegeneration: the role of p25/cyclin-dependent kinase 5. Calcineurin is involved in the early activation of NMDA-mediated cell death in mutant huntingtin knock-in striatal cells Helios transcription factor expression depends on Gsx2 and Dlx1&2 function in developing striatal matrix neurons. Altered P2X7-receptor level and function in mouse models of Huntington's disease and therapeutic efficacy of antagonist administration. Disease-specific phenotypes in dopamine neurons from human iPS-based models of genetic and sporadic Parkinson's disease. Full motor recovery despite striatal neuron loss and formation of irreversible amyloid-like inclusions in a conditional mouse model of Huntington's disease. Differential effects of glial cell line-derived neurotrophic factor and neurturin on developing and adult substantia nigra dopaminergic neurons Age-dependent decline of motor neocortex but not hippocampal performance in heterozygous BDNF mice correlates with a decrease of cortical PSD-95 but an increase of hippocampal TrkB levels. Bax deficiency promotes an up-regulation of Bim(EL) and Bak during striatal and cortical postnatal development, and after excitotoxic injury. BH3-only proteins Bid and Bim(EL) are differentially involved in neuronal dysfunction in mouse models of Huntington's disease Tachykinins protect cholinergic neurons from quinolinic acid excitotoxicity in striatal cultures.

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