About: Christopher S. Kovacs

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

Canadian endocrinologist

Attributes	Values
rdf:type	Item
description	연구원 (ko) গবেষক (bn) 研究者 (ja) дослідник (uk) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) hulumtues (sq) onderzoeker (nl) Canadian endocrinologist (en)
rdfs:label	Christopher S Kovacs (nl) Christopher S Kovacs (sl) Christopher S. Kovacs (en) Christopher S. Kovacs (es) Christopher S. Kovacs (it)
skos:prefLabel	Christopher S Kovacs (nl) Christopher S Kovacs (sl) Christopher S. Kovacs (en) Christopher S. Kovacs (es) Christopher S. Kovacs (it)
name	Christopher S Kovacs (nl) Christopher S Kovacs (sl) Christopher S. Kovacs (en) Christopher S. Kovacs (es) Christopher S. Kovacs (it)
Scopus author ID	wds:Q37840198-8B58C808-F5E9-41BA-92BB-8C4936DD3F8C
Scopus author ID	20034059700
given name	wds:Q37840198-A8E2AB1A-70F2-43D2-84EB-F7D199393AB2
given name	Christopher
instance of	wds:Q37840198-B1729925-48E6-4B41-98F0-DF9A66D3B296
instance of	Homo sapiens
skos:altLabel	Chris Kovacs (en) Christopher Kovacs (en)
occupation	wds:Q37840198-35f31936-4119-2454-e7bd-a634004812dd
employer	wds:Q37840198-1F204F6B-30BB-4C87-9678-8A27B50BF118
sex or gender	wds:Q37840198-7E2E5C64-B9AA-4590-A938-CC6F77478B35
VIAF ID	wds:Q37840198-5935cd1e-4ee9-4473-b73d-c9214ce5fa9a
GND ID	wds:Q37840198-ecbdde2c-6362-4c57-b777-faff69e9346c
family name	wds:Q37840198-56DF91C0-D01C-4F5D-9F82-C56F0D3C9B2C
VIAF ID	http://viaf.org/viaf/101477691
GND ID	https://d-nb.info/gnd/1202474624
occupation	endocrinologist
employer	Memorial University of Newfoundland
sex or gender	male
VIAF ID	101477691
GND ID	1202474624
family name	Kovacs
Springer Nature person ID	wds:Q37840198-0CA8C376-B126-446B-85C2-DAF8A07F140E
ORCID iD	wds:Q37840198-0F9BE317-FF6A-4E72-A5CF-BA4F2F0D6A17
Dimensions author ID	wds:Q37840198-A86DB306-8D29-47D8-AA19-C6FAD900348B
ORCID iD	https://orcid.org/0000-0002-5219-9993
Springer Nature person ID	01365135074.40
ORCID iD	0000-0002-5219-9993
Dimensions author ID	01365135074.40
ResearcherID	wds:Q37840198-9AB3C020-2234-4B3F-86D6-CA2F3D62F9CD
Publons author ID	wds:Q37840198-278E9D29-4305-4FA4-8349-913DF105F0ED
ResearcherID	AFS-2635-2022
Publons author ID	1179316
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q37840198
is author of	The 2011 report on dietary reference intakes for calcium and vitamin D from the Institute of Medicine: what clinicians need to know Calcium and bone disorders during pregnancy and lactation The nonskeletal effects of vitamin D: an Endocrine Society scientific statement. IOM committee members respond to Endocrine Society vitamin D guideline PTH regulates fetal blood calcium and skeletal mineralization independently of PTHrP Parathyroid hormone regulates fetal-placental mineral homeostasis Novel hexad repeats conserved in a putative transporter with restricted expression in cell types associated with growth, calcium exchange and homeostasis. Maternal-fetal calcium and bone metabolism during pregnancy, puerperium, and lactation Bone metabolism in the fetus and neonate Pregnancy up-regulates intestinal calcium absorption and skeletal mineralization independently of the vitamin D receptor Arrested pulmonary alveolar cytodifferentiation and defective surfactant synthesis in mice missing the gene for parathyroid hormone-related protein The vitamin D receptor is not required for fetal mineral homeostasis or for the regulation of placental calcium transfer in mice Hemophilia, low bone mass, and osteopenia/osteoporosis. Vitamin D insufficiency common in newborns, children and pregnant women living in Newfoundland and Labrador, Canada Calcium and Bone Metabolism Disorders During Pregnancy and Lactation Safety and tolerability of atypical antipsychotics in patients with bipolar disorder: prevalence, monitoring and management. Fetal parathyroids are not required to maintain placental calcium transport Calcitropic gene expression suggests a role for the intraplacental yolk sac in maternal-fetal calcium exchange Empagliflozin improves glycaemic and weight control as add-on therapy to pioglitazone or pioglitazone plus metformin in patients with type 2 diabetes: a 24-week, randomized, placebo-controlled trial The 2011 Dietary Reference Intakes for Calcium and Vitamin D: what dietetics practitioners need to know Calcium and bone metabolism during pregnancy and lactation. FGF-21 and skeletal remodeling during and after lactation in C57BL/6J mice. Maternal Mineral and Bone Metabolism During Pregnancy, Lactation, and Post-Weaning Recovery Hypercalcemia of malignancy in the palliative care patient: a treatment strategy Maternal vitamin D deficiency: Fetal and neonatal implications Meta-analysis of genome-wide studies identifies WNT16 and ESR1 SNPs associated with bone mineral density in premenopausal women Calcium channel TRPV6 is involved in murine maternal-fetal calcium transport. The role of vitamin D in pregnancy and lactation: insights from animal models and clinical studies Osteoporosis presenting in pregnancy, puerperium, and lactation Presentation and management of osteoporosis presenting in association with pregnancy or lactation Bone development and mineral homeostasis in the fetus and neonate: roles of the calciotropic and phosphotropic hormones Role of parathyroid hormone (PTH) and PTH-related protein (PTHrP) in regulating mineral homeostasis during fetal development Associations of Protein Intake and Protein Source with Bone Mineral Density and Fracture Risk: A Population-Based Cohort Study Empagliflozin as Add-on Therapy to Pioglitazone With or Without Metformin in Patients With Type 2 Diabetes Mellitus Ablation of calcitonin/calcitonin gene-related peptide-alpha impairs fetal magnesium but not calcium homeostasis Upregulation of calcitriol during pregnancy and skeletal recovery after lactation do not require parathyroid hormone. Calcitonin Plays a Critical Role in Regulating Skeletal Mineral Metabolism during Lactation Calcium, phosphorus, and bone metabolism in the fetus and newborn Primary hyperparathyroidism masked by antituberculous therapy-induced vitamin D deficiency. Skeletal recovery after weaning does not require PTHrP.

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