About: James C. Eisenach

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

American anesthesiologist

Attributes	Values
rdf:type	Item
owl:sameAs	James C. Eisenach
description	ikertzailea (eu) 연구원 (ko) 研究者 (ja) investigador (es) araştırmacı (tr) эрдэмтэн (mn) वैज्ञानिक (hi) изилдөөчү (ky) 研究者 (zh) onderzoeker (nl) American anesthesiologist (en)
rdfs:label	James C. Eisenach (de) James C. Eisenach (en) James C. Eisenach (es) James C. Eisenach (fr) James C. Eisenach (it) James C. Eisenach (nl) James C. Eisenach (pt) James C. Eisenach (pt-br) James C. Eisenach (ast)
skos:prefLabel	James C. Eisenach (de) James C. Eisenach (en) James C. Eisenach (es) James C. Eisenach (fr) James C. Eisenach (it) James C. Eisenach (nl) James C. Eisenach (pt) James C. Eisenach (pt-br) James C. Eisenach (ast)
name	James C. Eisenach (de) James C. Eisenach (en) James C. Eisenach (es) James C. Eisenach (fr) James C. Eisenach (it) James C. Eisenach (nl) James C. Eisenach (pt) James C. Eisenach (pt-br) James C. Eisenach (ast)
given name	wds:Q29791450-6F109BD2-9809-455F-8300-36D437DE46FA
given name	James
instance of	wds:Q29791450-1DA88163-60A3-48F4-A0EC-CC7CDBD6313D
instance of	Homo sapiens
Google Knowledge Graph ID	wds:Q29791450-945FC711-0DEA-4005-894E-CE1259479E76
Google Knowledge Graph ID	http://g.co/kg/g/11c741tvjs
Google Knowledge Graph ID	/g/11c741tvjs
skos:altLabel	J C Eisenach (en) J Eisenach (en) J. C. Eisenach (en) J. Eisenach (en) James C Eisenach (en) James C Eisenach (nl) James Eisenach (en)
occupation	wds:Q29791450-57557504-0B77-446C-91EA-2E80C7F0EE1E
employer	wds:Q29791450-70a65efd-c873-4840-86cb-5e9d157d5eef
languages spoken, written or signed	wds:q29791450-11a5b0f3-43b3-791b-4628-6305b4e441db
sex or gender	wds:Q29791450-2907E8B8-F441-46C4-A45C-1EF6F14EA91A
country of citizenship	wds:Q29791450-13517072-047E-4C0C-818E-1E1AED81D67A
writing language	wds:q29791450-b3d8db79-4ce8-96b5-e031-d6900a90455f
educated at	wds:Q29791450-10471270-A20B-4508-9CD1-04125946DA94 wds:Q29791450-10FCC6A1-B84A-452D-917E-F10288C8F9A9 wds:Q29791450-D8B9BEB3-4AC7-4C4B-81B9-0B9E1125BA7E
family name	wds:q29791450-87e0bb8a-48f9-2fc4-ca60-67fe21029352
occupation	researcher
employer	Wake Forest University
languages spoken, written or signed	English
sex or gender	male
country of citizenship	United States of America
writing language	English
educated at	University of California, San Francisco University of Nebraska system California Institute of Technology
family name	Eisenach
ORCID iD	wds:Q29791450-07c629fe-4a45-8f77-878a-a1f27426ac74
ORCID iD	https://orcid.org/0000-0002-9841-0176
ORCID iD	0000-0002-9841-0176
is owl:sameAs of	James C. Eisenach
is about of	James C. Eisenach https://www.wikidata.org/wiki/Special:EntityData/Q29791450 James C. Eisenach
is author of	Gabapentin inhibits γ-amino butyric acid release in the locus coeruleus but not in the spinal dorsal horn after peripheral nerve injury in rats Peripheral nerve injury and gabapentin, but not their combination, impair attentional behavior via direct effects on noradrenergic signaling in the brain Perineural clonidine reduces mechanical hypersensitivity and cytokine production in established nerve injury. Epidural Neostigmine versus Fentanyl to Decrease Bupivacaine Use in Patient-controlled Epidural Analgesia during Labor: A Randomized, Double-blind, Controlled Study Cephalad movement of morphine and fentanyl in humans after intrathecal injection Knock down of the alpha 5 nicotinic acetylcholine receptor in spinal nerve-ligated rats alleviates mechanical allodynia Analgesia from a peripherally active kappa-opioid receptor agonist in patients with chronic pancreatitis Perineural alpha(2A)-adrenoceptor activation inhibits spinal cord neuroplasticity and tactile allodynia after nerve injury Severity of acute pain after childbirth, but not type of delivery, predicts persistent pain and postpartum depression Age-dependent responses to nerve injury-induced mechanical allodynia. Oral donepezil reduces hypersensitivity after nerve injury by a spinal muscarinic receptor mechanism Intrathecal clonidine reduces hypersensitivity after nerve injury by a mechanism involving spinal m4 muscarinic receptors Toxicology profile of N-methyl-D-aspartate antagonists delivered by intrathecal infusion in the canine model Prior laparotomy or corticosterone potentiates lipopolysaccharide-induced fever and sickness behaviors Pain intensity and duration can be enhanced by prior challenge: initial evidence suggestive of a role of microglial priming Letter: Very fast zinc-catalyzed hydrolysis of an anhydride. A model for the rate and mechanism of carboxypeptidase A catalysis The antinociceptive effects of spinal cyclooxygenase inhibitors on uterine cervical distension Alpha2-adrenoceptor stimulation transforms immune responses in neuritis and blocks neuritis-induced pain. Hyperexcitability of axotomized and neighboring unaxotomized sensory neurons is reduced days after perineural clonidine at the site of injury

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