About: CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development

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rdf:type	Item
description	article scientifique publié en 2004 (fr) artículu científicu espublizáu en 2004 (ast) vědecký článek publikovaný v roce 2004 (cs) 2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած (hyw) наукова стаття, опублікована в липні 2004 (uk) 2004 թվականի հուլիսին հրատարակված գիտական հոդված (hy) vedecký článok (publikovaný 2004/07/15) (sk) مقالة علمية (نشرت في 15-7-2004) (ar) wetenschappelijk artikel (gepubliceerd op 2004/07/15) (nl) scientific journal article (en) im Juli 2004 veröffentlichter wissenschaftlicher Artikel (de)
publication date	wds:Q28594824-313D8D12-05B1-4BC4-8753-87CD9CC12482
publication date	2004-07-15 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28594824-C14F4E46-38A1-4A70-A1F3-FDD925D04A2C
language of work or name	English
author name string	wds:Q28594824-0DFBB8ED-B1B5-492C-AC5F-F28075D4D896 wds:Q28594824-E384E240-AC38-4436-B015-67806E7E6AE7
OpenCitations bibliographic resource ID	wds:Q28594824-CF3E26A2-BA73-46F7-999D-28DBA9580982
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/925820
author name string	Edward J. Pearce Amy S. McKee
OpenCitations bibliographic resource ID	925820
rdfs:label	CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (en) CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (nl) CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (ast)
skos:prefLabel	CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (en) CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (nl) CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (ast)
name	CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (en) CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (nl) CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (ast)
title	wds:Q28594824-1ADC4677-227F-4C42-BB5D-38829EA46A71
title	CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development (en)
page(s)	wds:Q28594824-9FA16DEC-DCE6-4610-B28A-A9B976D4F0E2
page(s)	1224–1231
instance of	wds:Q28594824-A5F13AB3-15C0-4D36-A992-4A8F693003C6
instance of	scholarly article
main subject	wds:Q28594824-0DFBF643-40E6-484E-A75F-619EBC192DCB wds:Q28594824-348C6FDA-6534-44EC-B271-C0D45DCB21E7 wds:Q28594824-3fade18b-48b8-299a-6eb5-0e072ce14b12 wds:Q28594824-506FC497-F628-42E0-9D13-4FF761A210CD wds:Q28594824-52CB551A-FE0E-414A-AC6F-81DD9360FEE9 wds:Q28594824-590AAEB4-7174-4660-A0F4-A697CADEC8C4 wds:Q28594824-956D7B75-DF1A-4D8A-8A15-05BCA12A5618 wds:Q28594824-B85419EF-BDDA-4DBD-BDE8-98709E548C5E wds:Q28594824-BA308F32-03B3-4F29-8D69-9AF326157BB2 wds:Q28594824-DD5947A8-92E4-40BC-8325-BD2636F46471 wds:Q28594824-E79D16D6-05C1-4109-8224-0D86470255EF wds:Q28594824-FC1B584E-9342-4D94-AD0C-633146F6EA3F
main subject	wd:Q21494041 parasitic helminthiasis infectious disease Interleukin 13 Interleukin 5 Interleukin 6 Interleukin 10 CD40 antigen Interleukin 12b Interferon gamma Interleukin 2 receptor, alpha chain CD86 antigen CD80 antigen
PubMed ID	wds:Q28594824-07DA1428-068B-445F-BF89-07F61A307EBD
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/15240714
PubMed ID	15240714
published in	wds:Q28594824-912D0AA4-8A42-400F-8D32-257827113BAE
published in	Journal of Immunology
issue	wds:Q28594824-5D982419-CBCA-453E-B3CA-ED7E23D2A084
volume	wds:Q28594824-769821A3-D462-4534-8B29-49B2FC0C183C
issue	2
volume	173
DOI	wds:Q28594824-CD9EC154-BE50-430E-B792-44DAA4C9AE2C
DOI	http://dx.doi.org/10.4049/JIMMUNOL.173.2.1224
DOI	10.4049/JIMMUNOL.173.2.1224
on focus list of Wikimedia project	wds:Q28594824-4278E5F3-5118-4E9D-A508-B593089DE116
on focus list of Wikimedia project	ScienceSource
ResearchGate publication ID	wds:Q28594824-B65C2A5C-D7A2-47AA-ABDD-AA5114CC5352
ResearchGate publication ID	8467980
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28594824
is cites work of	S. mansoni bolsters anti-viral immunity in the murine respiratory tract Concurrent bacterial stimulation alters the function of helminth-activated dendritic cells, resulting in IL-17 induction. Suppression of TH2-type allergic reactions by helminth infection. A worm's eye view of the immune system: consequences for evolution of human autoimmune disease. Coinfection with the intestinal nematode Heligmosomoides polygyrus markedly reduces hepatic egg-induced immunopathology and proinflammatory cytokines in mouse models of severe schistosomiasis. Regulatory T cell suppressive potency dictates the balance between bacterial proliferation and clearance during persistent Salmonella infection Regulatory T cells in microbial infection The role of natural regulatory T cells in infection Mucosal immunity and allergic responses: lack of regulation and/or lack of microbial stimulation? Parasitic worms and inflammatory diseases Worms and allergy Host cytokine production, lymphoproliferation, and antibody responses during the course of Ancylostoma ceylanicum infection in the Golden Syrian hamster Suppression of allergic airway inflammation by helminth-induced regulatory T cells Regulatory T cells and infection: a dangerous necessity Distinct sources and targets of IL-10 during dendritic cell-driven Th1 and Th2 responses in vivo. Helicobacter infection and gastric neoplasia The novel CD4+CD25+ regulatory T cell effector molecule fibrinogen-like protein 2 contributes to the outcome of murine fulminant viral hepatitis Comparative immunology of human and animal models of hookworm infection Current status of vaccines for schistosomiasis Immunopathology of schistosomiasis Helminth infection suppresses T-cell immune response to HIV-DNA-based vaccine in mice

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