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| - 1998 թուականի Ապրիլին հրատարակուած գիտական յօդուած (hyw)
- artículu científicu espublizáu en 1998 (ast)
- наукова стаття, опублікована у квітні 1998 (uk)
- 1998 թվականի ապրիլին հրատարակված գիտական հոդված (hy)
- vědecký článek publikovaný v roce 1998 (cs)
- vedecký článok (publikovaný 1998/04/15) (sk)
- مقالة علمية (نشرت في 15-4-1998) (ar)
- article scientifique publié en 1998 (fr)
- wetenschappelijk artikel (gepubliceerd op 1998/04/15) (nl)
- scientific journal article (en)
- im April 1998 veröffentlichter wissenschaftlicher Artikel (de)
- 1998 ഏപ്രില് 15 നു പ്രസിദ്ധീകരിച്ച ശാസ്ത്ര ലേഖനം (ml)
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| - E. N. Olson
- D. Srivastava
- P. A. Overbeek
- T. Thomas
- H. Yamagishi
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| - The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (en)
- The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (nl)
- The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (ast)
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| skos:prefLabel
| - The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (en)
- The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (nl)
- The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (ast)
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| name
| - The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (en)
- The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (nl)
- The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (ast)
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| title
| - The bHLH factors, dHAND and eHAND, specify pulmonary and systemic cardiac ventricles independent of left-right sidedness (en)
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of | - Disruption of hyaluronan synthase-2 abrogates normal cardiac morphogenesis and hyaluronan-mediated transformation of epithelium to mesenchyme
- Comparative gene expression analysis and fate mapping studies suggest an early segregation of cardiogenic lineages in Xenopus laevis.
- Efficient array-based identification of novel cardiac genes through differentiation of mouse ESCs
- Differences in the function of three conserved E-boxes of the muscle creatine kinase gene in cultured myocytes and in transgenic mouse skeletal and cardiac muscle
- Wnt5a and Wnt11 are essential for second heart field progenitor development.
- Human Dermo-1 has attributes similar to twist in early bone development
- Gene knock-outs of inositol 1,4,5-trisphosphate receptors types 1 and 2 result in perturbation of cardiogenesis
- Pitx2 regulates lung asymmetry, cardiac positioning and pituitary and tooth morphogenesis
- MicroRNA regulation of cardiovascular development
- The right ventricle, outflow tract, and ventricular septum comprise a restricted expression domain within the secondary/anterior heart field
- A twist of insight - the role of Twist-family bHLH factors in development
- Nfat and miR-25 cooperate to reactivate the transcription factor Hand2 in heart failure
- Msx1 and Msx2 are required for endothelial-mesenchymal transformation of the atrioventricular cushions and patterning of the atrioventricular myocardium
- Association of TWIST1 gene polymorphisms with bone mineral density in postmenopausal women
- Cited2 controls left-right patterning and heart development through a Nodal-Pitx2c pathway
- PIP5KI gamma is required for cardiovascular and neuronal development
- Isl1 identifies a cardiac progenitor population that proliferates prior to differentiation and contributes a majority of cells to the heart
- Hand2 loss-of-function in Hand1-expressing cells reveals distinct roles in epicardial and coronary vessel development
- Transcriptional pathways in second heart field development
- Cardiac chamber formation: development, genes, and evolution.
- MicroRNAs in cardiac development
- Irx4 forms an inhibitory complex with the vitamin D and retinoic X receptors to regulate cardiac chamber-specific slow MyHC3 expression.
- MicroRNA-363 negatively regulates the left ventricular determining transcription factor HAND1 in human embryonic stem cell-derived cardiomyocytes
- Left-right asymmetry and congenital cardiac defects: getting to the heart of the matter in vertebrate left-right axis determination
- Left-right asymmetry and cardiac looping: implications for cardiac development and congenital heart disease
- Heart development: molecular insights into cardiac specification and early morphogenesis
- Targeted deletion of Hand2 in cardiac neural crest-derived cells influences cardiac gene expression and outflow tract development
- Transcription factors in cardiogenesis: the combinations that unlock the mysteries of the heart
- Right versus left ventricular failure: differences, similarities, and interactions
- Suppression of atrial myosin gene expression occurs independently in the left and right ventricles of the developing mouse heart.
- Microdeletions and microduplications in patients with congenital heart disease and multiple congenital anomalies.
- Molecular regulation of cardiomyocyte differentiation
- Single-Cell Resolution of Temporal Gene Expression during Heart Development
- The basic helix-loop-helix transcription factor, dHAND, is required for vascular development
- Left-asymmetric expression of Galanin in the linear heart tube of the mouse embryo is independent of the nodal co-receptor gene cryptic.
- A positive GATA element and a negative vitamin D receptor-like element control atrial chamber-specific expression of a slow myosin heavy-chain gene during cardiac morphogenesis
- Cardiac phenotypes in chromosome 4q- syndrome with and without a deletion of the dHAND gene.
- Characterization of sequences in human TWIST required for nuclear localization
- Dilated cardiomyopathy presenting during fetal life
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