About: Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors

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About: Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors Goto Sponge NotDistinct Permalink

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rdf:type	Item
description	artículu científicu espublizáu en 2009 (ast) vědecký článek publikovaný v roce 2009 (cs) 2009 թվականի հունվարին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в січні 2009 (uk) 2009 թուականի Յունուարին հրատարակուած գիտական յօդուած (hyw) im Juni 2008 veröffentlichter wissenschaftlicher Artikel (de) scientific journal article (en) wetenschappelijk artikel (gepubliceerd op 2009/01/12) (nl) vedecký článok (publikovaný 2009/01/12) (sk) مقالة علمية (نشرت في 12-1-2009) (ar)
publication date	wds:Q28583420-5B6B3094-9144-4C88-9407-0EC32AF576A5
publication date	2008-06-14 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28583420-F0D922B2-B400-41C2-B62D-3CC15307BFF4
language of work or name	English
author name string	wds:Q28583420-0ABEEF93-F51D-4DD3-AC6E-57EBA4C14030 wds:Q28583420-157F7A55-8787-4342-AF87-376BDCD95AD8 wds:Q28583420-15DE26ED-A545-45C1-9CE4-E40E9F437A87 wds:Q28583420-2F939480-32D5-434D-94CC-459A3911F21B wds:Q28583420-D08A54FB-6AAE-46FA-BC4D-7A8A18DC1261 wds:Q28583420-E4AE574A-554E-4DA1-B516-7091F5E5158A wds:Q28583420-EF67B3ED-979B-49D8-9A75-A9EC23F58FB1
author name string	H Bading F Rode H E Freitag A-S Wahl B Buchthal L C B Rønn S F Bomholt
rdfs:label	Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (en) Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (nl) Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (ast)
skos:prefLabel	Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (en) Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (nl) Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (ast)
name	Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (en) Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (nl) Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (ast)
author	wds:Q28583420-B07E4335-A0D9-4F27-96BD-A1546FE678C7
author	Giles E. Hardingham
title	wds:Q28583420-1F8C5750-00BB-49F5-8B57-1EB27E971490
title	Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors (en)
page(s)	wds:Q28583420-8FD99400-52EE-4B15-B4F5-95A4147EF9CE
page(s)	344-352
instance of	wds:Q28583420-8B9AEC6C-5755-4B4E-96AB-7C9D667E5038
instance of	scholarly article
main subject	wds:Q28583420-13F3DF7C-F5FF-49DB-AA9F-0C99E0386B94
main subject	hypoxia
PubMed ID	wds:Q28583420-98C9DF18-F381-451D-9974-7171427AEAFD
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/18616988
PubMed ID	18616988
published in	wds:Q28583420-99BAC0C0-83DE-4156-B0B0-CB7637371C8A
published in	Neuroscience
issue	wds:Q28583420-3D273B01-3230-4EF4-9DEE-3B6A5382658E
volume	wds:Q28583420-1CE49335-C10D-47B0-AF42-0965C875487E
issue	1
volume	158
DOI	wds:Q28583420-B5B415EE-FF4F-496E-AE4E-7D5C5FC5D702
DOI	http://dx.doi.org/10.1016/J.NEUROSCIENCE.2008.06.018
DOI	10.1016/J.NEUROSCIENCE.2008.06.018
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28583420
is cites work of	Synaptic and extrasynaptic NMDA receptors differentially modulate neuronal cyclooxygenase-2 function, lipid peroxidation, and neuroprotection Influence of GluN2 subunit identity on NMDA receptor function. Selective dendritic susceptibility to bioenergetic, excitotoxic and redox perturbations in cortical neurons G-protein-coupled receptor 30 mediates rapid neuroprotective effects of estrogen via depression of NR2B-containing NMDA receptors Pathophysiology of Huntington's disease: time-dependent alterations in synaptic and receptor function. Extrasynaptic NMDA receptors couple preferentially to excitotoxicity via calpain-mediated cleavage of STEP Calcium signaling in synapse-to-nucleus communication Coupling of the NMDA receptor to neuroprotective and neurodestructive events Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders Neuronal development is promoted by weakened intrinsic antioxidant defences due to epigenetic repression of Nrf2 Synaptic NMDA receptor activity is coupled to the transcriptional control of the glutathione system Environmental experience modulates ischemia-induced amyloidogenesis and enhances functional recovery Mechanisms of specificity in neuronal activity-regulated gene transcription Modes of Neuronal Calcium Entry and Homeostasis following Cerebral Ischemia Leptin counteracts the hypoxia-induced inhibition of spontaneously firing hippocampal neurons: a microelectrode array study Enhanced SUMOylation and SENP-1 protein levels following oxygen and glucose deprivation in neurones Aspartate release and signalling in the hippocampus The subtype of GluN2 C-terminal domain determines the response to excitotoxic insults Peripheral nerve injury is accompanied by chronic transcriptome-wide changes in the mouse prefrontal cortex Chloride co-transporters as possible therapeutic targets for stroke Protection in glutamate-induced neurotoxicity by imidazoline receptor agonist moxonidine The role of PSD-95 and cypin in morphological changes in dendrites following sublethal NMDA exposure. Neuroprotective effects of hydroxysafflor yellow A against excitotoxic neuronal death partially through down-regulation of NR2B-containing NMDA receptors Expression of mRNA Encoding Mcu and Other Mitochondrial Calcium Regulatory Genes Depends on Cell Type, Neuronal Subtype, and Ca2+ Signaling Recovery of NMDA receptor currents from MK-801 blockade is accelerated by Mg2+ and memantine under conditions of agonist exposure Decreased calcium-activated potassium channels by hypoxia causes abnormal firing in the spontaneous firing medial vestibular nuclei neurons. Loss of Arnt (Hif1β) in mouse epidermis triggers dermal angiogenesis, blood vessel dilation and clotting defects Cyclin D1 induction preceding neuronal death via the excitotoxic NMDA pathway involves selective stimulation of extrasynaptic NMDA receptors and JNK pathway. NMDA receptor C-terminal signaling in development, plasticity, and disease Presenilin-1/γ-secretase controls glutamate release, tyrosine phosphorylation, and surface expression of N-methyl-D-aspartate receptor (NMDAR) subunit GluN2B. Neuronal Activity and Its Role in Controlling Antioxidant Genes
is cites work of	wds:Q28394694-A661C3B2-38B3-4B79-9C0A-2B5D04890A94 wds:Q28567752-DB262762-5EE2-4DD4-A46F-C6CEE79B7B7E wds:Q35100259-0F16602A-DE94-4F41-8CC6-E68819F2784C wds:Q34358581-9256EB21-448B-400F-A9C8-164DDD19FCE4 wds:Q37800822-D974221E-FD5B-4896-8A2B-D7D60B9A90B7 wds:Q42059132-FF11FD11-6C53-4FEE-9B7B-79AC65483446 wds:Q36099382-80F278BB-DDF6-41A1-9BE7-3D9496E4BBAF wds:Q39012818-7ECEEE17-839F-4FBC-BC98-E008F24894E4

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