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| - artículu científicu espublizáu en 1996 (ast)
- vědecký článek publikovaný v roce 1996 (cs)
- vedecký článok (publikovaný 1996/09/01) (sk)
- 1996 թվականի սեպտեմբերին հրատարակված գիտական հոդված (hy)
- 1996 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած (hyw)
- наукова стаття, опублікована у вересні 1996 (uk)
- wetenschappelijk artikel (gepubliceerd op 1996/09/01) (nl)
- scientific journal article (en)
- im September 1996 veröffentlichter wissenschaftlicher Artikel (de)
- مقالة علمية نشرت في سبتمبر 1996 حول موضوع: دماغ (ar)
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| author name string
| - M Baudry
- X Bi
- G Tocco
- H Herschman
- L Vician
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| rdfs:label
| - Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (en)
- Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (nl)
- Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (ast)
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| skos:prefLabel
| - Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (en)
- Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (nl)
- Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (ast)
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| name
| - Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (en)
- Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (nl)
- Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (ast)
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| title
| - Two synaptotagmin genes, Syt1 and Syt4, are differentially regulated in adult brain and during postnatal development following kainic acid-induced seizures (en)
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| DOI
| - 10.1016/0169-328X(96)00055-1
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| is cites work
of | - Potential new antiepileptogenic targets indicated by microarray analysis in a rat model for temporal lobe epilepsy.
- Stress differentially regulates synaptophysin and synaptotagmin expression in hippocampus.
- Astrocytic glutamate is not necessary for the generation of epileptiform neuronal activity in hippocampal slices.
- Nerve growth factor-dependent sorting of synaptotagmin IV protein to mature dense-core vesicles that undergo calcium-dependent exocytosis in PC12 cells.
- Alternative splicing of synaptotagmins involving transmembrane exon skipping
- Synaptotagmin IV: biochemistry, genetics, behavior, and possible links to human psychiatric disease
- Differential mRNA expression patterns of the synaptotagmin gene family in the rodent brain
- Age- and experience-dependent expression of dynamin I and synaptotagmin I in cat visual system.
- Synaptotagmin IV regulates glial glutamate release
- Synaptotagmin IV does not alter excitatory fast synaptic transmission or fusion pore kinetics in mammalian CNS neurons
- The salt-inducible kinase, SIK, is induced by depolarization in brain
- KID-1, a protein kinase induced by depolarization in brain
- rTLE3, a newly identified transducin-like enhancer of split, is induced by depolarization in brain
- Acute administration of cocaine, but not amphetamine, increases the level of synaptotagmin IV mRNA in the dorsal striatum of rat.
- Nurr1 mRNA expression in neonatal and adult rat brain following kainic acid-induced seizure activity.
- Synaptotagmin I and IV define distinct populations of neuronal transport vesicles
- Analysis of synaptotagmin I-IV messenger RNA expression and developmental regulation in the rat hypothalamus and pituitary.
- Temporal characteristics of vesicular fusion in astrocytes: examination of synaptobrevin 2-laden vesicles at single vesicle resolution.
- Kainate-induced genes in the hippocampus: lessons from expression patterns.
- Searching for depolarization-induced genes that modulate synaptic plasticity and neurotrophin-induced genes that mediate neuronal differentiation
- Loss of synaptotagmin IV results in a reduction in synaptic vesicles and a distortion of the Golgi structure in cultured hippocampal neurons
- Late postnatal maturation of excitatory synaptic transmission permits adult-like expression of hippocampal-dependent behaviors
- Kainic acid-induced generalized seizures alter the regional hippocampal expression of the rat m1 and m3 muscarinic acetylcholine receptor genes.
- Altered expression of synaptotagmin I in temporal lobe tissue of patients with refractory epilepsy.
- Kainic acid-induced generalized seizures alter the regional hippocampal expression of the rat Kv4.2 potassium channel gene.
- The calcium sensor synaptotagmin 1 is expressed and regulated in hippocampal postsynaptic spines.
- Synaptotagmin I and IV are differentially regulated in the brain by the recreational drug 3,4-methylenedioxymethamphetamine (MDMA).
- Up-regulation of Synaptotagmin IV within amyloid plaque-associated dystrophic neurons in Tg2576 mouse model of Alzheimer's disease
- Upregulation and axonal transport of synaptotagmin-IV in the direct-pathway medium spiny neurons in hemi-parkinsonian rats induced by dopamine D1 receptor stimulation
- Upregulation of synaptotagmin IV inhibits transmitter release in PC12 cells with targeted synaptotagmin I knockdown
- Abnormal expression of synaptophysin, SNAP-25, and synaptotagmin 1 in the hippocampus of kainic acid-exposed rats with behavioral deficits
- Effect of acute and chronic treatment with methamphetamine on mRNA expression of synaptotagmin IV and 25 KDa-synaptic-associated protein in the rat brain
- Amphiphysin I but not dynamin I nor synaptojanin mRNA expression increased after repeated methamphetamine administration in the rat cerebrum and cerebellum
- Putting a brake on synaptic vesicle endocytosis.
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