About: Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin

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About: Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin Goto Sponge NotDistinct Permalink

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rdf:type	Item
description	artículu científicu espublizáu en 2004 (ast) vědecký článek publikovaný v roce 2004 (cs) 2004 թվականի օգոստոսին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в серпні 2004 (uk) 2004 թուականի Օգոստոսին հրատարակուած գիտական յօդուած (hyw) مقالة علمية (نشرت في أغسطس 2004) (ar) wetenschappelijk artikel (gepubliceerd op 2004/08/01) (nl) vedecký článok (publikovaný 2004/08/01) (sk) im August 2004 veröffentlichter wissenschaftlicher Artikel (de) scientific journal article (en)
publication date	wds:Q28577283-19CE2D50-481B-4AAA-84AF-A0178AD49A4D
publication date	2004-08-01 00:00:00Z (xsd:dateTime)
author name string	wds:Q28577283-24FE3290-175C-4AA4-AE68-7C24F2E1A35D wds:Q28577283-D3E4756E-73A0-4EB8-B6F7-521B9A30649F wds:Q28577283-EC3C6AA8-D9D2-4679-994D-27792F438D9B
OpenCitations bibliographic resource ID	wds:Q28577283-FF1F5D2A-C2E0-4C28-A9F8-D344616B6547
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/578920
author name string	Jack E. Bodwell Joshua W. Hamilton Lauren A. Kingsley
OpenCitations bibliographic resource ID	578920
rdfs:label	Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (en) Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (nl) Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (ast)
skos:prefLabel	Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (en) Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (nl) Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (ast)
name	Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (en) Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (nl) Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (ast)
title	wds:Q28577283-CD1065A6-F5FD-47BB-A7D9-855B3946DD3E
title	Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin (en)
page(s)	wds:Q28577283-ECA48438-90FA-4ACF-9BB8-666AE26AE4E6
page(s)	1064–1076
instance of	wds:Q28577283-AA5E374A-2DE2-4D82-8D53-1BA8E9979EFD
instance of	scholarly article
main subject	wds:Q28577283-3CC9F3CD-0553-402E-A693-88EFD6AED418 wds:Q28577283-CFDA203C-9883-4CF2-8523-45B29DA8B653
main subject	arsenic glucocorticoid
PubMed ID	wds:Q28577283-538EFBE8-715D-4461-973D-AEDF795489EB
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/15310238
PubMed ID	15310238
published in	wds:Q28577283-23C1D8D2-2701-4B75-8BC2-D28ADB263E0A
published in	Chemical Research in Toxicology
issue	wds:Q28577283-8DC50ABA-D566-4633-918D-15DCC64D642D
volume	wds:Q28577283-19D9FB86-5E5B-41B6-8D56-6D5220A28772
issue	8
volume	17
DOI	wds:Q28577283-C1F03CD4-6993-40E8-A042-039C0CE5FC7D
DOI	http://dx.doi.org/10.1021/TX0499113
DOI	10.1021/TX0499113
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28577283
is cites work of	Arsenic as an endocrine disruptor: effects of arsenic on estrogen receptor-mediated gene expression in vivo and in cell culture Arsenic as an endocrine disruptor: arsenic disrupts retinoic acid receptor-and thyroid hormone receptor-mediated gene regulation and thyroid hormone-mediated amphibian tail metamorphosis Arsenic exposure and type 2 diabetes: a systematic review of the experimental and epidemiological evidence Arsenic disruption of steroid receptor gene activation: Complex dose-response effects are shared by several steroid receptors Disruption of histone modification and CARM1 recruitment by arsenic represses transcription at glucocorticoid receptor-regulated promoters Analysis of genomic dose-response information on arsenic to inform key events in a mode of action for carcinogenicity The Effects of Arsenic Exposure on Neurological and Cognitive Dysfunction in Human and Rodent Studies: A Review Perturbation of defense pathways by low-dose arsenic exposure in zebrafish embryos Prenatal exposure to arsenic and cadmium impacts infectious disease-related genes within the glucocorticoid receptor signal transduction pathway The steroidogenic acute regulatory protein as a target of endocrine disruption in male reproduction In utero exposure to low dose arsenic via drinking water impairs early life lung mechanics in mice Arsenic exposure during pregnancy and size at birth: a prospective cohort study in Bangladesh Endocrine-disrupting chemicals: associated disorders and mechanisms of action Human health effects from chronic arsenic poisoning--a review Relation between in Utero Arsenic Exposure and Birth Outcomes in a Cohort of Mothers and Their Newborns from New Hampshire Environmental exposure to arsenic may reduce human semen quality: associations derived from a Chinese cross-sectional study Chronic exposure to arsenic in the drinking water alters the expression of immune response genes in mouse lung. Perinatal exposure to 50 ppb sodium arsenate induces hypothalamic-pituitary-adrenal axis dysregulation in male C57BL/6 mice Prenatal arsenic exposure alters the programming of the glucocorticoid signaling system during embryonic development The effects of metals as endocrine disruptors. Pilot-scale demonstration of phytofiltration for treatment of arsenic in New Mexico drinking water Mechanisms pertaining to arsenic toxicity Lung tumors in mice induced by "whole-life" inorganic arsenic exposure at human-relevant doses Induction of cytoplasmic accumulation of p53: a mechanism for low levels of arsenic exposure to predispose cells for malignant transformation. Role of glucocorticoid receptor in acclimation of killifish (Fundulus heteroclitus) to seawater and effects of arsenic. Moderate perinatal arsenic exposure alters neuroendocrine markers associated with depression and increases depressive-like behaviors in adult mouse offspring Learning deficits in C57BL/6J mice following perinatal arsenic exposure: consequence of lower corticosterone receptor levels? Sex-specific associations of arsenic exposure with global DNA methylation and hydroxymethylation in leukocytes: results from two studies in Bangladesh Arsenic induced progesterone production in a caspase-3-dependent manner and changed redox status in preovulatory granulosa cells. Arsenic-induced cancer cell phenotype in human breast epithelia is estrogen receptor-independent but involves aromatase activation mot-2-Mediated cross talk between nuclear factor-B and p53 is involved in arsenite-induced tumorigenesis of human embryo lung fibroblast cells Understanding arsenic dynamics in agronomic systems to predict and prevent uptake by crop plants. Systems biology and birth defects prevention: blockade of the glucocorticoid receptor prevents arsenic-induced birth defects Monomethylated trivalent arsenic species disrupt steroid receptor interactions with their DNA response elements at non-cytotoxic cellular concentrations The repressive effect of NF-kappaB on p53 by mot-2 is involved in human keratinocyte transformation induced by low levels of arsenite Effect of sodium arsenite on reproductive organs of female Wistar rats. Environmental endocrine disruption of energy metabolism and cardiovascular risk Thyroid hormone- and estrogen receptor interactions with natural ligands and endocrine disruptors in the cerebellum. Endocrine active metals, prenatal stress and enhanced neurobehavioral disruption. The glucocorticoid receptor: a revisited target for toxins Mutual interaction between nutritional status and chronic arsenic toxicity due to groundwater contamination in an area of Terai, lowland Nepal The hijacking of cellular signaling and the diabetes epidemic: mechanisms of environmental disruption of insulin action and glucose homeostasis Risk assessment of endocrine disrupters: the role of toxicological studies. Comparison of Individual and Combined Effects of Four Endocrine Disruptors on Estrogen Receptor Beta Transcription in Cerebellar Cell Culture: The Modulatory Role of Estradiol and Triiodo-Thyronine Contribution of breast milk and formula to arsenic exposure during the first year of life in a US prospective cohort CD44v6 expression in human skin keratinocytes as a possible mechanism for carcinogenesis associated with chronic arsenic exposure

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