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description
| - artículu científicu espublizáu en 2002 (ast)
- vědecký článek publikovaný v roce 2002 (cs)
- наукова стаття, опублікована в грудні 2002 (uk)
- 2002 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած (hyw)
- 2002 թվականի դեկտեմբերին հրատարակված գիտական հոդված (hy)
- vedecký článok (publikovaný 2002/12/20) (sk)
- مقالة علمية (نشرت في 20-12-2002) (ar)
- wetenschappelijk artikel (gepubliceerd op 2002/12/20) (nl)
- article publié dans la revue scientifique Journal of Biological Chemistry (fr)
- im Dezember 2002 veröffentlichter wissenschaftlicher Artikel (de)
- scientific journal article (en)
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cites work
| - Identification of a novel inhibitor of mitogen-activated protein kinase kinase
- Bim: a novel member of the Bcl-2 family that promotes apoptosis
- Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery
- Phosphorylation of the pro-apoptotic protein BIK: mapping of phosphorylation sites and effect on apoptosis
- Synthesis and function of 3-phosphorylated inositol lipids
- Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)
- PUMA, a novel proapoptotic gene, is induced by p53
- Induction of BIM, a proapoptotic BH3-only BCL-2 family member, is critical for neuronal apoptosis
- The proapoptotic BH3-only protein bim is expressed in hematopoietic, epithelial, neuronal, and germ cells
- FKHR-L1 can act as a critical effector of cell death induced by cytokine withdrawal: protein kinase B-enhanced cell survival through maintenance of mitochondrial integrity
- Apoptosis signaling
- The MLK family mediates c-Jun N-terminal kinase activation in neuronal apoptosis
- c-Jun and the transcriptional control of neuronal apoptosis
- The Ras/phosphatidylinositol 3-kinase and Ras/ERK pathways function as independent survival modules each of which inhibits a distinct apoptotic signaling pathway in sympathetic neurons.
- Regulation of neuronal survival and death by E2F-dependent gene repression and derepression.
- Establishment of a noradrenergic clonal line of rat adrenal pheochromocytoma cells which respond to nerve growth factor
- Downregulation of Bim, a proapoptotic relative of Bcl-2, is a pivotal step in cytokine-initiated survival signaling in murine hematopoietic progenitors
- Induction and endoplasmic reticulum location of BIK/NBK in response to apoptotic signaling by E1A and p53.
- The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex
- Dominant-negative c-Jun promotes neuronal survival by reducing BIM expression and inhibiting mitochondrial cytochrome c release
- The phosphatidylinositol 3-kinase (PI3K)-Akt pathway suppresses Bax translocation to mitochondria
- Up-regulation of Hrk, a regulator of cell death, in retinal ganglion cells of axotomized rat retina
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author name string
| - Lloyd A. Greene
- Subhas C. Biswas
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rdfs:label
| - Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (en)
- Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (nl)
- Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (ast)
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skos:prefLabel
| - Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (en)
- Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (nl)
- Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (ast)
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name
| - Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (en)
- Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (nl)
- Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (ast)
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title
| - Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylation (en)
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