About: Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways

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About: Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways Goto Sponge NotDistinct Permalink

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rdf:type	Item
description	artículu científicu espublizáu en 2001 (ast) vědecký článek publikovaný v roce 2001 (cs) 2001 թվականի մայիսին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в травні 2001 (uk) 2001 թուականի Մայիսին հրատարակուած գիտական յօդուած (hyw) vedecký článok (publikovaný 2001/05/11) (sk) مقالة علمية (نشرت في 11-5-2001) (ar) article scientifique publié en 2001 (fr) im Mai 2001 veröffentlichter wissenschaftlicher Artikel (de) wetenschappelijk artikel (gepubliceerd op 2001/05/11) (nl) scientific journal article (en)
publication date	wds:Q28571066-F4908BA6-77F4-40C4-9A21-1393B88ED073
publication date	2001-05-11 00:00:00Z (xsd:dateTime)
cites work	wds:Q28571066-ACE4A688-9F06-4338-ACC1-697B3CBD585B
cites work	Cell transformation by the superoxide-generating oxidase Mox1
author name string	wds:Q28571066-4592B3BE-CEFF-48CD-B158-06DF5E50BC90 wds:Q28571066-68900A73-2745-46F1-9686-45694C0485EA wds:Q28571066-6DB5D086-C929-4BCB-AC82-D2B3B5CB2B40 wds:Q28571066-7B41976E-A0BF-41DB-8145-6980B290B7E9 wds:Q28571066-8390B2C8-7AF0-441F-8EA9-37B234B2E6C0 wds:Q28571066-8DAF0031-EA39-468D-A578-B28A623B4D5D wds:Q28571066-B3AE084C-FD55-44B4-8C34-120A4013E2C8 wds:Q28571066-C6C78298-6A02-4E23-97F2-2D587AE56C07 wds:Q28571066-E7BE51C5-AC5B-4D75-BAE7-5AF880D2BB06
OpenCitations bibliographic resource ID	wds:Q28571066-91763272-165C-40F6-A518-1C1CF41E31C6
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/3486540
author name string	Y. Zhang K. K. Griendling J. D. Lambeth Q. Yin B. Lassègue D. Sorescu K. Szöcs M. Akers S. L. Grant
OpenCitations bibliographic resource ID	3486540
rdfs:label	Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (en) Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (nl) Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (ast)
skos:prefLabel	Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (en) Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (nl) Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (ast)
name	Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (en) Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (nl) Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (ast)
title	wds:Q28571066-99EC270D-395F-40D1-A81E-5DBC2DC68B9E
title	Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways (en)
page(s)	wds:Q28571066-1930B535-5AA9-4F18-BAAD-0CE4F650BA0B
page(s)	888–894
instance of	wds:Q28571066-9CBE62B8-C72B-4A71-AC69-CC7C6E69712A
instance of	scholarly article
PubMed ID	wds:Q28571066-7570F0E3-EE32-4F1C-82D7-18D886CACC01
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/11348997
PubMed ID	11348997
published in	wds:Q28571066-ADB20967-7BD8-428C-A705-5BF4337AAFA9
published in	Circulation Research
issue	wds:Q28571066-95E95692-1D58-40D5-9101-CD7BB7925CBB
volume	wds:Q28571066-6C32784B-AEAA-4568-8E1C-B721BF039A0E
issue	9
volume	88
DOI	wds:Q28571066-117BFB20-22BB-4FAD-8CAF-3B4D9896957D
DOI	http://dx.doi.org/10.1161/HH0901.090299
DOI	10.1161/HH0901.090299
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28571066
is cites work of	Novel Human Homologues of p47 and p67 Participate in Activation of Superoxide-producing NADPH Oxidases Poldip2, a novel regulator of Nox4 and cytoskeletal integrity in vascular smooth muscle cells NOXO1, Regulation of Lipid Binding, Localization, and Activation of Nox1 by the Phox Homology (PX) Domain Molecular evolution of the reactive oxygen-generating NADPH oxidase (Nox/Duox) family of enzymes Nox4 oxidase overexpression specifically decreases endogenous Nox4 mRNA and inhibits angiotensin II-induced adventitial myofibroblast migration Nonhematopoietic NADPH oxidase regulation of lung eosinophilia and airway hyperresponsiveness in experimentally induced asthma Inhibition of NADPH oxidase 4 activates apoptosis via the AKT/apoptosis signal-regulating kinase 1 pathway in pancreatic cancer PANC-1 cells. NOX5 NAD(P)H oxidase regulates growth and apoptosis in DU 145 prostate cancer cells NOX1/nicotinamide adenine dinucleotide phosphate, reduced form (NADPH) oxidase promotes proliferation of stellate cells and aggravates liver fibrosis induced by bile duct ligation Redox control of renal function and hypertension. Impaired basal NO activity in patients with glomerular disease and the influence of oxidative stress. The pathobiology of diabetic vascular complications--cardiovascular and kidney disease Salt, Angiotensin II, Superoxide, and Endothelial Function Mitogenic signaling via platelet-derived growth factor beta in metanephric mesenchymal cells Overexpression of SIRT1 in vascular smooth muscle cells attenuates angiotensin II-induced vascular remodeling and hypertension in mice Angiotensin II-induced production of mitochondrial reactive oxygen species: potential mechanisms and relevance for cardiovascular disease Orchestrating redox signaling networks through regulatory cysteine switches The nicotinamide adenine dinucleotide phosphate oxidase (NOX) homologues NOX1 and NOX2/gp91(phox) mediate hepatic fibrosis in mice Inhibition of NADPH oxidase improves impaired reactivity of pial arterioles during chronic exposure to nicotine Retracted: Activation of the AMP-Activated Protein Kinase (AMPK) by Nitrated Lipids in Endothelial Cells The involvement of aldosterone in cyclic stretch-mediated activation of NADPH oxidase in vascular smooth muscle cells NADPH oxidase overexpression in human colon cancers and rat colon tumors induced by 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP). The human ATF1 rs11169571 polymorphism increases essential hypertension risk through modifying miRNA binding Superoxide-dependent cathepsin activation is associated with hypertensive myocardial remodeling and represents a target for angiotensin II type 1 receptor blocker treatment Deficiency of Nox2 prevents angiotensin II-induced inward remodeling in cerebral arterioles Regulation of NAD(P)H oxidases by AMPK in cardiovascular systems Angiotensin and cytoskeletal proteins: role in vascular remodeling Decreased blood pressure in NOX1-deficient mice Ebselen and congeners inhibit NADPH oxidase 2-dependent superoxide generation by interrupting the binding of regulatory subunits Angiotensin II induces a region-specific hyperplasia of the ascending aorta through regulation of inhibitor of differentiation 3. Resveratrol blocks Akt activation in angiotensin II- or EGF-stimulated vascular smooth muscle cells in a redox-independent manner. Reactive oxygen species: roles in blood pressure and kidney function Lipid rafts and caveolin-1 coordinate interleukin-1beta (IL-1beta)-dependent activation of NFkappaB by controlling endocytosis of Nox2 and IL-1beta receptor 1 from the plasma membrane Hypoxia-induced changes in pulmonary and systemic vascular resistance: where is the O2 sensor? Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy

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