About: Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion

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About: Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion Goto Sponge NotDistinct Permalink

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rdf:type	Item
description	artículu científicu espublizáu en 2005 (ast) vědecký článek publikovaný v roce 2005 (cs) наукова стаття, опублікована в жовтні 2005 (uk) 2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած (hyw) 2005 թվականի հոտեմբերին հրատարակված գիտական հոդված (hy) مقالة علمية (نشرت في أكتوبر 2005) (ar) wetenschappelijk artikel (gepubliceerd op 2005/10/01) (nl) vedecký článok (publikovaný 2005/10/01) (sk) scientific journal article (en) im Oktober 2005 veröffentlichter wissenschaftlicher Artikel (de)
publication date	wds:Q28512256-483887FE-802D-4C4D-ABC9-6BD466F6873E
publication date	2005-10-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28512256-24970D73-F347-4969-97A0-C264F6C8F6B6
language of work or name	English
author name string	wds:Q28512256-0B1BFA7F-EEB5-4163-99C0-D5D6892746A1
OpenCitations bibliographic resource ID	wds:Q28512256-E6729D9E-AD75-402A-9B42-97EB11BED08F
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/1339440
author name string	Fabiana Cerrato
OpenCitations bibliographic resource ID	1339440
rdfs:label	Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (en) Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (nl) Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (ast)
skos:prefLabel	Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (en) Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (nl) Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (ast)
name	Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (en) Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (nl) Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (ast)
author	wds:Q28512256-40D434D7-61FE-496B-B44B-812C265DBA58 wds:Q28512256-BD797D95-3144-4418-85BC-A037430A09BD
author	Huansheng Xu Antonio Baldini
title	wds:Q28512256-7E710A04-6E11-4E34-90B5-9D78B2C7F936
title	Timed mutation and cell-fate mapping reveal reiterated roles of Tbx1 during embryogenesis, and a crucial function during segmentation of the pharyngeal system via regulation of endoderm expansion (en)
page(s)	wds:Q28512256-5B2D2DD5-E2B9-429E-BD5F-226A8F014109
page(s)	4387–4395
instance of	wds:Q28512256-943F6D10-1E1A-4951-9BD8-E1DCBB7733B3
instance of	scholarly article
main subject	wds:Q28512256-5913C5A4-03F7-41C2-844C-F51B67B5C870 wds:Q28512256-F1E55225-23E1-4386-9606-A42DDB1A922C
main subject	embryogenesis T-box 1
PubMed ID	wds:Q28512256-96CFD23B-6109-4040-AF2B-E0E51F42A816
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/16141220
PubMed ID	16141220
published in	wds:Q28512256-F6CACF2D-DB20-4802-B971-FFC8312D9BE3
published in	Development
issue	wds:Q28512256-C5851F9B-AFB1-4F65-B46E-205B04540CCB
volume	wds:Q28512256-93BA83FC-B336-42C2-B1C1-69E9E4C6298B
issue	19
volume	132
DOI	wds:Q28512256-CA465EC7-1967-4B75-B652-8D5C6C3ED343
DOI	http://dx.doi.org/10.1242/DEV.02018
DOI	10.1242/DEV.02018
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28512256
is cites work of	What can vertebrates tell us about segmentation? 22q11 deletion syndrome: a role for TBX1 in pharyngeal and cardiovascular development Cellular and molecular events during early thymus development Mesodermal expression of Tbx1 is necessary and sufficient for pharyngeal arch and cardiac outflow tract development Tbx1 regulates Vegfr3 and is required for lymphatic vessel development Genetic pathways to mammalian heart development: Recent progress from manipulation of the mouse genome Identification and developmental expression of two Tbx1/10-related genes in the agnathan Lethenteron japonicum Hes1 expression is reduced in Tbx1 null cells and is required for the development of structures affected in 22q11 deletion syndrome Patterning of the third pharyngeal pouch into thymus/parathyroid by Six and Eya1 Velocardiofacial syndrome, DiGeorge syndrome: the chromosome 22q11.2 deletion syndromes Extrinsic versus intrinsic cues in avian paraxial mesoderm patterning and differentiation. Tbx1 regulation of myogenic differentiation in the limb and cranial mesoderm. Neonatal Levels of T-cell Receptor Excision Circles (TREC) in Patients with 22q11.2 Deletion Syndrome and Later Disease Features Defective ALK5 signaling in the neural crest leads to increased postmigratory neural crest cell apoptosis and severe outflow tract defects The molecular setup of the avian head mesoderm and its implication for craniofacial myogenesis. Islet 1 is expressed in distinct cardiovascular lineages, including pacemaker and coronary vascular cells Transcription factors in parathyroid development: lessons from hypoparathyroid disorders Cyp26 genes a1, b1 and c1 are down-regulated in Tbx1 null mice and inhibition of Cyp26 enzyme function produces a phenocopy of DiGeorge Syndrome in the chick. Tbx1 is required for second heart field proliferation in zebrafish Isl1 is upstream of sonic hedgehog in a pathway required for cardiac morphogenesis Fate-mapping evidence that hepatic stellate cells are epithelial progenitors in adult mouse livers Tbx1 controls the morphogenesis of pharyngeal pouch epithelia through mesodermal Wnt11r and Fgf8a Endodermal/ectodermal interfaces during pharyngeal segmentation in vertebrates. Tbx1 regulates population, proliferation and cell fate determination of otic epithelial cells In vivo genetic ablation of the periotic mesoderm affects cell proliferation survival and differentiation in the cochlea Tbx1 regulates proliferation and differentiation of multipotent heart progenitors Transcriptional regulation of thymus organogenesis and thymic epithelial cell differentiation Fgf8 expression in the Tbx1 domain causes skeletal abnormalities and modifies the aortic arch but not the outflow tract phenotype of Tbx1 mutants Tbx1 controls cardiac neural crest cell migration during arch artery development by regulating Gbx2 expression in the pharyngeal ectoderm Retinoic acid regulates differentiation of the secondary heart field and TGFbeta-mediated outflow tract septation Identification of downstream genetic pathways of Tbx1 in the second heart field A fate map of Tbx1 expressing cells reveals heterogeneity in the second cardiac field Dynamics of thymus organogenesis and colonization in early human development Congenital anomalies and rhabdoid tumor associated with 22q11 germline deletion and somatic inactivation of the SMARCB1 tumor suppressor Secondary immunologic consequences in chromosome 22q11.2 deletion syndrome (DiGeorge syndrome/velocardiofacial syndrome). Tbx1, subpulmonary myocardium and conotruncal congenital heart defects. Primary immunodeficiency diseases associated with neurologic manifestations Multilineage somatic activating mutations in HRAS and NRAS cause mosaic cutaneous and skeletal lesions, elevated FGF23 and hypophosphatemia A defect in early myogenesis causes Otitis media in two mouse models of 22q11.2 Deletion Syndrome MOZ regulates the Tbx1 locus, and Moz mutation partially phenocopies DiGeorge syndrome Visualization of outflow tract development in the absence of Tbx1 using an FgF10 enhancer trap transgene. Mechanism responsible for D-transposition of the great arteries: Is this part of the spectrum of right isomerism? Lineage tracing reveals conversion of liver sinusoidal endothelial cells into hepatocytes. p53 Suppression partially rescues the mutant phenotype in mouse models of DiGeorge syndrome

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