About: Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals

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scientific journal article

Attributes	Values
rdf:type	Item
description	artículu científicu espublizáu en 2009 (ast) vedecký článok (publikovaný 2009/01/01) (sk) vědecký článek publikovaný v roce 2009 (cs) wetenschappelijk artikel (gepubliceerd op 2009/01/01) (nl) مقالة علمية (نشرت عام 2009) (ar) article scientifique publié en 2009 (fr) 2009 թվականի հունվարին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в січні 2009 (uk) 2009 թուականի Յունուարին հրատարակուած գիտական յօդուած (hyw) im Januar 2009 veröffentlichter wissenschaftlicher Artikel (de) scientific journal article (en)
publication date	wds:Q28511672-C5B196D7-DD2A-44B9-9F9A-964A1FE7A4CA
publication date	2009-01-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q28511672-6CCF4022-59BA-440E-A1F4-C19CF2C95B6F
language of work or name	English
cites work	wds:Q28511672-2696A1C8-599D-4098-B048-2BE49EDA38F9 wds:Q28511672-9747E906-AB6F-4CDC-819B-60C3AB83B7D5
cites work	Critical roles of c-Jun signaling in regulation of NFAT family and RANKL-regulated osteoclast differentiation Effects of blocking platelet-derived growth factor-receptor signaling in a mouse model of experimental prostate cancer bone metastases
author name string	wds:Q28511672-0B1734B2-79A4-4FC7-89C8-08CDC253D6C3 wds:Q28511672-37F5869F-46A2-4003-8AB0-1628BEB09B87
OpenCitations bibliographic resource ID	wds:Q28511672-ED04F787-F524-4434-8E02-0EEEF9487E77
OpenCitations bibliographic resource ID	https://w3id.org/oc/corpus/br/3001089
author name string	Hiroaki Nakamura Toru Hiraga
OpenCitations bibliographic resource ID	3001089
rdfs:label	Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (en) Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (nl) Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (ast)
skos:prefLabel	Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (en) Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (nl) Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (ast)
name	Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (en) Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (nl) Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (ast)
title	wds:Q28511672-9C6203EA-A1E7-4ADE-8985-74AA35AC828D
title	Imatinib mesylate suppresses bone metastases of breast cancer by inhibiting osteoclasts through the blockade of c-Fms signals (en)
page(s)	wds:Q28511672-22820BEB-9218-4884-A726-674B95290768
page(s)	215–222
instance of	wds:Q28511672-D65ECF8D-51DC-422B-8113-C01D502C74F2
instance of	scholarly article
main subject	wds:Q28511672-6C2AE8A5-F37C-4ACB-B10C-3F5402C2C4AA wds:Q28511672-84B0BE7D-AF74-4000-B333-F1235C6BB4FC wds:Q28511672-997497CD-541B-4DCB-A71B-40856F3CF8F4
main subject	wd:Q177094 breast cancer Colony stimulating factor 1 receptor
PubMed ID	wds:Q28511672-B8A50113-76CD-421C-92EE-5F78AFF422BE
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/18814279
PubMed ID	18814279
published in	wds:Q28511672-ECF2B9D3-7B3F-489B-8E4A-6A355A9A164D
published in	International Journal of Cancer
issue	wds:Q28511672-1DA1C313-D228-4689-BECC-61FFA5D71080
volume	wds:Q28511672-56D96F2E-2DFC-483E-9FA3-5A23E6467D9F
issue	1
volume	124
DOI	wds:Q28511672-61E43D05-A76C-4D96-BD22-7E975F7F717D
DOI	http://dx.doi.org/10.1002/IJC.23903
DOI	10.1002/IJC.23903
ResearchGate publication ID	wds:Q28511672-7DB27675-81D0-405A-B808-518E33D27D3F
ResearchGate publication ID	23279949
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q28511672
is cites work of	Tumor-associated macrophages in breast cancer as potential biomarkers for new treatments and diagnostics Growth factors as active participants in carcinogenesis: a perspective Bone-derived IGF mediates crosstalk between bone and breast cancer cells in bony metastases Breast tumor stroma: A driving force in the development of resistance to therapies FMS Kinase Inhibitors: Current Status and Future Prospects. Evaluation of the novel USPIO GEH121333 for MR imaging of cancer immune responses Emerging role of mucins in epithelial to mesenchymal transition Amplification of F-Actin Disassembly and Cellular Repulsion by Growth Factor Signaling. Therapeutic Antibodies Targeting CSF1 Impede Macrophage Recruitment in a Xenograft Model of Tenosynovial Giant Cell Tumor Platelet-derived growth factors induced lymphangiogenesis: evidence, unanswered questions and upcoming challenges. Autocrine inhibition of the c-fms proto-oncogene reduces breast cancer bone metastasis assessed with in vivo dual-modality imaging Inhibition of the c-fms proto-oncogene autocrine loop and tumor phenotype in glucocorticoid stimulated human breast carcinoma cells Imatinib inhibits proliferation of human mesenchymal stem cells and promotes early but not late osteoblast differentiation in vitro Characterization and Targeting of Platelet-Derived Growth Factor Receptor alpha (PDGFRA) in Inflammatory Breast Cancer (IBC) Stromal fibroblasts induce metastatic tumor cell clusters via epithelial-mesenchymal plasticity Tyrosine Kinase Inhibitors Regulate OPG through Inhibition of PDGFRβ. The M-CSF receptor in osteoclasts and beyond Malignant stroma increases luminal breast cancer cell proliferation and angiogenesis through platelet-derived growth factor signaling The Effects of Imatinib Mesylate on Cellular Viability, Platelet Derived Growth Factor and Stem Cell Factor in Mouse Testicular Normal Leydig Cells The Therapeutic Potential of Targeting Tumor Microenvironment in Breast Cancer: Rational Strategies and Recent Progress
is cites work of	wds:Q39064583-967E39CC-AF73-4D48-9284-4D6AC06837EC wds:Q39633737-994BEA15-6776-4F2F-B1B3-43567C16EE71 wds:Q36941590-7ACE9797-6E64-4C73-9821-C1DD01108207 wds:Q39459161-9F3DAA57-2541-4AEE-ABE0-505483ED6CC0 wds:Q98499841-FB1147E1-94DC-4FF5-B3C9-4B3AC7C7ABC6 wds:Q37584296-A25AE6C0-44B2-4C73-A58B-44368DD2D1BB wds:Q36223424-C0C23F1C-5CC8-4B6F-B9F7-F331AFACEFB1 wds:Q38872199-AC631455-0E6D-41F1-81F3-6707812E3368 wds:Q39346175-0BBC6F65-0B52-4355-925E-84CD113513E2 wds:Q42668157-0AD3F8BE-12F4-49AD-861F-3586F861D854 wds:Q92090616-C49A14A5-EA85-497B-9696-3C8DE0827823 wds:Q36162943-5BCA4B5F-1CD4-47D9-8DAE-A655C3F72D85 wds:Q37676697-70765B9B-61B1-4EBC-892C-7753502315BB wds:Q36872172-D43F2A80-5C93-41EF-9711-DB19F4BD7639 wds:Q37995562-BA971798-4E09-41B3-BE1F-35E146E70801 wds:Q50901053-49D1F2F1-6D7E-4D65-9234-28089DD1AB10 wds:Q34309316-66973F0C-6B87-4DD2-AEF2-CFB63982823C

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