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description
| - artículu científicu espublizáu en 2005 (ast)
- vědecký článek publikovaný v roce 2005 (cs)
- 2005 թուականի Յունիսին հրատարակուած գիտական յօդուած (hyw)
- наукова стаття, опублікована в червні 2005 (uk)
- مقالة علمية (نشرت في يونيو 2005) (ar)
- 2005 թվականի հունիսին հրատարակված գիտական հոդված (hy)
- vedecký článok (publikovaný 2005/06/01) (sk)
- wetenschappelijk artikel (gepubliceerd op 2005/06/01) (nl)
- scientific journal article (en)
- im Juni 2005 veröffentlichter wissenschaftlicher Artikel (de)
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publication date
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language of work or name
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author name string
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author name string
| - Osamu Nakagawa
- Eric N. Olson
- John McAnally
- Paul D. Gottlieb
- Philip W. Tucker
- Dillon Phan
- Tara L. Rasmussen
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rdfs:label
| - BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (en)
- BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (nl)
- BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (ast)
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skos:prefLabel
| - BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (en)
- BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (nl)
- BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (ast)
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name
| - BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (en)
- BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (nl)
- BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (ast)
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author
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author
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title
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title
| - BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart (en)
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is about
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is cites work
of | - skNAC, a Smyd1-interacting transcription factor, is involved in cardiac development and skeletal muscle growth and regeneration
- Smyd3 is required for the development of cardiac and skeletal muscle in zebrafish
- The tale of two domains: proteomics and genomics analysis of SMYD2, a new histone methyltransferase
- Centronuclear myopathy in mice lacking a novel muscle-specific protein kinase transcriptionally regulated by MEF2
- Genetic pathways to mammalian heart development: Recent progress from manipulation of the mouse genome
- Covalent modifications of histones during development and disease pathogenesis
- PDE5A inhibition attenuates bleomycin-induced pulmonary fibrosis and pulmonary hypertension through inhibition of ROS generation and RhoA/Rho kinase activation.
- Cardiac deletion of Smyd2 is dispensable for mouse heart development
- SET/MYND Lysine Methyltransferases Regulate Gene Transcription and Protein Activity
- Do we have two hearts? New insights in right ventricular function supported by myocardial imaging echocardiography
- Myomaxin is a novel transcriptional target of MEF2A that encodes a Xin-related alpha-actinin-interacting protein
- A threshold of GATA4 and GATA6 expression is required for cardiovascular development
- SMYD1, the myogenic activator, is a direct target of serum response factor and myogenin
- SmyD1, a histone methyltransferase, is required for myofibril organization and muscle contraction in zebrafish embryos.
- smyd1 and smyd2 are expressed in muscle tissue in Xenopus laevis
- Muscle-specific expression of the smyd1 gene is controlled by its 5.3-kb promoter and 5'-flanking sequence in zebrafish embryos.
- Enhancer identification through comparative genomics
- Building the mammalian heart from two sources of myocardial cells
- Transcriptional pathways in second heart field development
- Regulation of skeletal muscle sarcomere integrity and postnatal muscle function by Mef2c
- Ca2+-operated transcriptional networks: molecular mechanisms and in vivo models.
- SMYD proteins: key regulators in skeletal and cardiac muscle development and function
- Expression and functional characterization of Smyd1a in myofibril organization of skeletal muscles
- Regulation of HDAC9 gene expression by MEF2 establishes a negative-feedback loop in the transcriptional circuitry of muscle differentiation
- Research Resource: The androgen receptor modulates expression of genes with critical roles in muscle development and function
- Web-based analysis of the mouse transcriptome using Genevestigator
- Zebrafish Mef2ca and Mef2cb are essential for both first and second heart field cardiomyocyte differentiation.
- Myoglobin-deficient mice activate a distinct cardiac gene expression program in response to isoproterenol-induced hypertrophy
- Smyd1 facilitates heart development by antagonizing oxidative and ER stress responses
- MEF2C is required for the normal allocation of cells between the ventricular and sinoatrial precursors of the primary heart field
- Whole exome sequencing of microdissected splenic marginal zone lymphoma: a study to discover novel tumor-specific mutations
- Defective myogenesis in the absence of the muscle-specific lysine methyltransferase SMYD1.
- Inositol 1,4,5-trisphosphate receptors are essential for the development of the second heart field
- A provisional gene regulatory atlas for mouse heart development
- Still Heart Encodes a Structural HMT, SMYD1b, with Chaperone-Like Function during Fast Muscle Sarcomere Assembly
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