About: insulitis

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illness

Attributes	Values
rdf:type	Item
description	illness (en) entzündliche Infiltration von Immunzellen in die Langerhans-Inseln der Bauchspeicheldrüse (de)
rdfs:label	Insulitis (de) infiltration des îlots de Langerhans (fr) insulite (pt) insulitis (ca) insulitis (en) insulitis (es) insulitis (nl) אינסוליטיס (he) التهاب الجزر البنكرياسية (ar) 胰岛炎 (zh) 胰岛炎 (zh-cn) 胰岛炎 (zh-hans) 胰島炎 (zh-hant)
skos:prefLabel	Insulitis (de) infiltration des îlots de Langerhans (fr) insulite (pt) insulitis (ca) insulitis (en) insulitis (es) insulitis (nl) אינסוליטיס (he) التهاب الجزر البنكرياسية (ar) 胰岛炎 (zh) 胰岛炎 (zh-cn) 胰岛炎 (zh-hans) 胰島炎 (zh-hant)
name	Insulitis (de) infiltration des îlots de Langerhans (fr) insulite (pt) insulitis (ca) insulitis (en) insulitis (es) insulitis (nl) אינסוליטיס (he) التهاب الجزر البنكرياسية (ar) 胰岛炎 (zh) 胰岛炎 (zh-cn) 胰岛炎 (zh-hans) 胰島炎 (zh-hant)
Freebase ID	wds:Q922215-536842ED-2C56-47C9-AF7D-4CBFFD2AEA57
Freebase ID	/m/0407f5y
instance of	wds:Q922215-d31c3778-40b0-95dd-2ebe-99d056fd1881
instance of	disease
subclass of	wds:Q922215-b6c599a5-4db6-b09a-ff11-30b40eebeaaa
subclass of	endocrine pancreas disease
anatomical location	wds:Q922215-82d2144a-481e-4346-f67a-8a4e4abd921e
anatomical location	islet of Langerhans
Commons category	wds:Q922215-e216c786-402c-4a87-81fc-5aa6f492562f
Commons category	Insulitis
skos:altLabel	insulite (fr)
OpenAlex ID	wds:Q922215-6CC18368-7669-4E74-82D1-4203BE949583
Microsoft Academic ID	wds:Q922215-4B6602A5-074D-4C1B-96A0-D968579CCBDD
Microsoft Academic ID	https://makg.org/entity/2779511750
OpenAlex ID	C2779511750
Microsoft Academic ID	2779511750
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q922215 Insulitis https://de.wikipedia.org/wiki/Insulitis Category:Insulitis https://ar.wikipedia.org/wiki/%D8%A7%D9%84%D8%AA%D9%87%D8%A7%D8%A8_%D8%A7%D9%84%D8%AC%D8%B2%D8%B1_%D8%A7%D9%84%D8%A8%D9%86%D9%83%D8%B1%D9%8A%D8%A7%D8%B3%D9%8A%D8%A9 https://he.wikipedia.org/wiki/%D7%90%D7%99%D7%A0%D7%A1%D7%95%D7%9C%D7%99%D7%98%D7%99%D7%A1 https://pt.wikipedia.org/wiki/Insulite
is main subject of	Systemic production of interferon-gamma inducing factor (IGIF) versus local IFN-gamma expression involved in the development of Th1 insulitis in NOD mice Increased expression of CCL2 in insulin-producing cells of transgenic mice promotes mobilization of myeloid cells from the bone marrow, marked insulitis, and diabetes Inhibition of insulitis and prevention of diabetes in nonobese diabetic mice by blocking L-selectin and very late antigen 4 adhesion receptors Effect of infection by lactic dehydrogenase virus on expression of intercellular adhesion molecule-1 on vascular endothelial cells of pancreatic islets in streptozotocin-induced insulitis of CD-1 mice Reduced streptozotocin-induced insulitis in CD-1 mice by treatment with anti-intercellular adhesion molecule-1 and anti-lymphocyte function associated antigen-1 monoclonal antibodies together with lactic dehydrogenase virus infection Pathogenicity of a reovirus type 2 in suckling mice: insulitis and lymphoid atrophy Expression of genetically determined diabetes and insulitis in the nonobese diabetic (NOD) mouse at the level of bone marrow-derived cells. Transfer of diabetes and insulitis to nondiabetic (NOD X B10) F1 mice with bone marrow cells from NOD mice Insulitis in type 1 diabetes: a sticky problem Remodeling of the innervation of pancreatic islets accompanies insulitis preceding onset of diabetes in the NOD mouse Killer Treg cells ameliorate inflammatory insulitis in non-obese diabetic mice through local and systemic immunomodulation Expression of Ly-6C by T lymphocytes of NOD mice after CD3-complex stimulation. Identification of activated cells during insulitis of prediabetic mice Demonstration of a TH1 cytokine profile in the late phase of NOD insulitis Intra-islet proliferation of cytotoxic T lymphocytes contributes to insulitis progression Insulitis and diabetes in NOD mice reduced by prophylactic insulin therapy Vascular addressins are induced on islet vessels during insulitis in nonobese diabetic mice and are involved in lymphoid cell binding to islet endothelium Co-expression of HLA DR3 and DQ8 results in the development of spontaneous insulitis and loss of tolerance to GAD65 in transgenic mice CD40 ligand-CD40 interactions are necessary for the initiation of insulitis and diabetes in nonobese diabetic mice Direct evidence for the contribution of B cells to the progression of insulitis and the development of diabetes in non-obese diabetic mice Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice Hyperresponsiveness, resistance to B-cell receptor-dependent activation-induced cell death, and accumulation of hyperactivated B-cells in islets is associated with the onset of insulitis but not type 1 diabetes. Flow cytometric enumeration of mononuclear cell populations infiltrating the islets of Langerhans in prediabetic NOD mice: development of a model of autoimmune insulitis for type I diabetes B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice Inhibition of VEGFR-2 reverses type 1 diabetes in NOD mice by abrogating insulitis and restoring islet function Treatment with anti-T-lymphocyte antibodies prevents induction of insulitis in mice given multiple doses of streptozocin Neonatal injection of CD3 antibody into nonobese diabetic mice reduces the incidence of insulitis and diabetes Reduced incidence of insulitis in NOD mice following anti-CD3 injection: requirement for neonatal injection NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humans. Insulitis in transgenic mice expressing tumor necrosis factor beta (lymphotoxin) in the pancreas Transgenic tumor necrosis factor (TNF)-alpha production in pancreatic islets leads to insulitis, not diabetes. Distinct patterns of inflammation in TNF-alpha and TNF-beta transgenic mice A case of recurrent type 1 diabetes mellitus with insulitis of transplanted pancreas in simultaneous pancreas-kidney transplantation from cardiac death donor Autoimmune syndromes in major histocompatibility complex (MHC) congenic strains of nonobese diabetic (NOD) mice. The NOD MHC is dominant for insulitis and cyclophosphamide-induced diabetes. Coxsackievirus B3 infection and type 1 diabetes development in NOD mice: insulitis determines susceptibility of pancreatic islets to virus infection Expression of cholera toxin B-proinsulin fusion protein in lettuce and tobacco chloroplasts--oral administration protects against development of insulitis in non-obese diabetic mice Monoclonal anti-tumor necrosis factor antibody renders non-obese diabetic mice hypersensitive to irradiation and enhances insulitis development Coxsackie B4 virus infection of beta cells and natural killer cell insulitis in recent-onset type 1 diabetic patients

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