About: Janusz Witowski

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

Polish physician, researcher, pathophysiologist

Attributes	Values
rdf:type	Item
description	研究者 (ja) wetenschapper (nl) изилдөөчү (ky) польський дослідник (uk) polski lekarz, patofizjolog (pl) Polish physician, researcher, pathophysiologist (en)
rdfs:label	Janusz Witowski (en) Janusz Witowski (nl) Janusz Witowski (pl) Janusz Witowski (ast)
skos:prefLabel	Janusz Witowski (en) Janusz Witowski (nl) Janusz Witowski (pl) Janusz Witowski (ast)
name	Janusz Witowski (en) Janusz Witowski (nl) Janusz Witowski (pl) Janusz Witowski (ast)
Polish scientist ID (deprecated)	wds:Q90787050-314331EE-4E73-4E5A-87AA-F5E4AF81C59A
Polish scientist ID (deprecated)	11323
given name	wds:Q90787050-AB75625D-DD8B-4FDD-90B1-083A40E8205F wds:Q90787050-DF5D3C23-2E4A-4EB6-BD56-9DD07C3EBFAF
given name	Cezary Janusz
instance of	wds:Q90787050-82E58E0D-3C09-42AC-86EF-A43FE6AC512A
instance of	Homo sapiens
doctoral advisor	wds:Q90787050-C7891C6A-7FF7-4F68-9669-B037B8471172
doctoral student	wds:Q90787050-D544DD55-7271-4DA3-BCA6-9EAE893962CC
doctoral advisor	Andrzej Bręborowicz
doctoral student	Krzysztof Książek
skos:altLabel	Janusz Cezary Witowski (pl)
occupation	wds:Q90787050-42608298-B4CF-43FA-8229-AA5123B55100 wds:q90787050-952be169-42b8-123b-2711-e9aafa77bc9b
NUKAT ID	wds:q90787050-0daabf51-474d-5a94-6f71-75c916ff6a42
sex or gender	wds:Q90787050-3883A0ED-8857-4B11-ABB5-85E2C483A52E
ISNI	wds:q90787050-bcff7011-4c89-4a78-cb84-2dd8ed3928d5
VIAF ID	wds:Q90787050-f8f113fe-4100-0438-eade-b7a5b7c96711
GND ID	wds:q90787050-279a6501-4f49-8c62-9877-eb9a4f0366f2
country of citizenship	wds:q90787050-5a674b30-45d0-b6e0-d375-d9f68fb5a24f
academic degree	wds:Q90787050-1F06816C-E8E5-4BF6-89F0-AE9ED6B6B736 wds:Q90787050-9EE6C122-1A71-4742-8C98-5465282F40A8 wds:q90787050-7867c0fa-4a2a-ed2c-476e-0118f3a26643
date of birth	wds:Q90787050-3ae13dae-4c02-ad46-1f7c-5b2bb4600e57
PLWABN ID	wds:q90787050-159fadc0-4060-4991-834f-a63600ef69cd
family name	wds:q90787050-ac97fd76-4d58-5759-2513-3ef4f24d9485
VIAF ID	http://viaf.org/viaf/162015659
GND ID	https://d-nb.info/gnd/128921935
occupation	physician researcher
NUKAT ID	n01090280
sex or gender	male
ISNI	0000000111449178
VIAF ID	162015659
GND ID	128921935
country of citizenship	Poland
academic degree	doctorate habilitation scientific professorship degree
date of birth	1964-01-01 00:00:00Z (xsd:dateTime)
PLWABN ID	9810551087205606
family name	Witowski
ORCID iD	wds:Q90787050-2EF43C0F-4FAA-41E3-8350-9392A5DB58CF
ORCID iD	https://orcid.org/0000-0002-1093-6027
ORCID iD	0000-0002-1093-6027
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q90787050
is author of	Interplay between IFN-gamma and IL-6 signaling governs neutrophil trafficking and apoptosis during acute inflammation New developments in peritoneal fibroblast biology: implications for inflammation and fibrosis in peritoneal dialysis Peritoneal cell culture: fibroblasts Regulation of chemokine CCL5 synthesis in human peritoneal fibroblasts: a key role of IFN-γ. Glucose-mediated induction of TGF-beta 1 and MCP-1 in mesothelial cells in vitro is osmolality and polyol pathway dependent Human peritoneal fibroblasts are a potent source of neutrophil-targeting cytokines: a key role of IL-1beta stimulation The proto-oncogene c-Fos transcriptionally regulates VEGF production during peritoneal inflammation. Activation of nuclear factor of activated T cells 5 in the peritoneal membrane of uremic patients. Oxidative stress-dependent increase in ICAM-1 expression promotes adhesion of colorectal and pancreatic cancers to the senescent peritoneal mesothelium. Senescence induces a proangiogenic switch in human peritoneal mesothelial cells Identification of IGFBP-7 by urinary proteomics as a novel prognostic marker in early acute kidney injury Glucose degradation products in peritoneal dialysis fluids: do they harm? Biocompatibility of a bicarbonate-buffered amino-acid-based solution for peritoneal dialysis L-2-oxothiazolidine-4-carboxylic acid reduces in vitro cytotoxicity of glucose degradation products Early loss of proliferative potential of human peritoneal mesothelial cells in culture: the role of p16INK4a-mediated premature senescence. Correlation between the donor age and the proliferative lifespan of human peritoneal mesothelial cells in vitro: is TGF-beta1 a link? Peritoneal dialysis with solutions low in glucose degradation products is associated with improved biocompatibility profile towards peritoneal mesothelial cells. Impaired response to oxidative stress in senescent cells may lead to accumulation of DNA damage in mesothelial cells from aged donors PD membrane: biological responses to different PD fluids Relation of salivary antioxidant status and cytokine levels to clinical parameters of oral health in pregnant women with diabetes Senescent peritoneal mesothelial cells promote ovarian cancer cell adhesion: the role of oxidative stress-induced fibronectin New insights into the biology of peritoneal mesothelial cells: the roles of epithelial-to-mesenchymal transition and cellular senescence Mesothelial toxicity of peritoneal dialysis fluids is related primarily to glucose degradation products, not to glucose per se. Vulnerability to oxidative stress and different patterns of senescence in human peritoneal mesothelial cell strains Quality assurance in peritoneal dialysis Association of serum VEGF with clinical response to anti-TNFα therapy for Crohn's disease Effects of peritoneal dialysis solutions on the peritoneal membrane: clinical consequences. Oxidative stress-mediated early senescence contributes to the short replicative life span of human peritoneal mesothelial cells Glucose-induced mesothelial cell senescence and peritoneal neoangiogenesis and fibrosis Angiogenesis-related proteins--their role in the pathogenesis and treatment of inflammatory bowel disease Oxidative stress contributes to accelerated development of the senescent phenotype in human peritoneal mesothelial cells exposed to high glucose Accelerated senescence of human peritoneal mesothelial cells exposed to high glucose: the role of TGF-beta1 An increase in serum tumour necrosis factor-α during anti-tumour necrosis factor-α therapy for Crohn's disease - A paradox or a predictive index? Peritoneal dialysis fluids can alter HSP expression in human peritoneal mesothelial cells.

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