About: Ferdinando Nicoletti

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

Italian researcher

Attributes	Values
rdf:type	Item
description	araştırmacı (tr) эрдэмтэн (mn) Italian researcher (en) باحث إيطالي (ar) ricercatore italiano (it)
rdfs:label	Ferdinando Nicoletti (en) Ferdinando Nicoletti (it)
skos:prefLabel	Ferdinando Nicoletti (en) Ferdinando Nicoletti (it)
name	Ferdinando Nicoletti (en) Ferdinando Nicoletti (it)
Scopus author ID	wds:Q84521195-EF2FC5B1-2879-4768-90B2-05D27AD10160
Scopus author ID	7102021660
given name	wds:Q84521195-84CA75DA-B225-4E20-9A5F-634C93BDBBBE
given name	Ferdinando
instance of	wds:Q84521195-E7866896-641A-4F0A-A2C2-90B3335D0F99
instance of	Homo sapiens
occupation	wds:Q84521195-6F9A0E2C-98D2-43C2-B6BB-4348DC9729C2
employer	wds:Q84521195-E0DBF261-31A9-48EA-88E1-A7D07F04C7CC
languages spoken, written or signed	wds:q84521195-90880828-4a82-6be4-93b0-851afe7e1290
sex or gender	wds:Q84521195-E3CAF31C-C0B9-47C2-A715-F57FDEEA2911
VIAF ID	wds:Q84521195-63a245b4-3b9d-400d-8160-cfc2d84baf8c
GND ID	wds:Q84521195-7e8b7a9f-3f51-4a61-b0ed-f7f349ff9f3b
country of citizenship	wds:Q84521195-1F486567-BC50-48C6-82F6-8801955A0C0A
family name	wds:Q84521195-E5C3B447-62B7-4A11-A80A-07F63F58BDE6
VIAF ID	http://viaf.org/viaf/36409798
GND ID	https://d-nb.info/gnd/1222202069
occupation	researcher
employer	University of Milan-Bicocca
languages spoken, written or signed	Italian
sex or gender	male
VIAF ID	36409798
GND ID	1222202069
country of citizenship	Italy
family name	Nicoletti
ORCID iD	wds:Q84521195-DBAEE396-4508-4BEA-A9EE-FD64E87A288D
ORCID iD	https://orcid.org/0000-0002-4570-8462
ORCID iD	0000-0002-4570-8462
ResearcherID	wds:Q84521195-3E39D16C-B058-4688-BF1A-3B2189063A36
ResearcherID	M-4428-2016
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q84521195 https://it.wikipedia.org/wiki/Ferdinando_Nicoletti
is author of	Insulin lispro is as effective as regular insulin in optimising metabolic control and preserving beta-cell function at onset of type 1 diabetes mellitus Ras/Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR inhibitors: rationale and importance to inhibiting these pathways in human health Therapeutic resistance resulting from mutations in Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR signaling pathways. Akt as a therapeutic target in cancer Emerging Raf inhibitors. Biphasic effect of interferon-gamma in murine collagen-induced arthritis Nondepleting anti-CD4 and soluble interleukin-1 receptor prevent autoimmune destruction of syngeneic islet grafts in diabetic NOD mice Critical role of macrophage migration inhibitory factor activity in experimental autoimmune diabetes ISO-1 binding to the tautomerase active site of MIF inhibits its pro-inflammatory activity and increases survival in severe sepsis Macrophage migration inhibitory factor (MIF) seems crucially involved in Guillain–Barré syndrome and experimental allergic neuritis Chitotriosidase gene expression in Kupffer cells from patients with non-alcoholic fatty liver disease Targeting the leukemic stem cell: the Holy Grail of leukemia therapy Therapeutic potential of carbon monoxide in multiple sclerosis Prevention of clinical and histological signs of proteolipid protein (PLP)-induced experimental allergic encephalomyelitis (EAE) in mice by the water-soluble carbon monoxide-releasing molecule (CORM)-A1. Blood levels of transforming growth factor-beta 1 (TGF-beta1) are elevated in both relapsing remitting and chronic progressive multiple sclerosis (MS) patients and are further augmented by treatment with interferon-beta 1b (IFN-beta1b). The analysis of IL-1 beta and its naturally occurring inhibitors in multiple sclerosis: The elevation of IL-1 receptor antagonist and IL-1 receptor type II after steroid therapy Circulating serum levels of IL-1ra in patients with relapsing remitting multiple sclerosis are normal during remission phases but significantly increased either during exacerbations or in response to IFN-beta treatment Short-term treatment of relapsing remitting multiple sclerosis patients with interferon (IFN)-beta1B transiently increases the blood levels of interleukin (IL)-6, IL-10 and IFN-gamma without significantly modifying those of IL-1beta, IL-2, IL-4 and Anticancer effects of the nitric oxide-modified saquinavir derivative saquinavir-NO against multidrug-resistant cancer cells Elevated Serum Levels of Osteopontin in HCV-Associated Lymphoproliferative Disorders. Emerging MEK inhibitors. Involvement of Akt-1 and mTOR in sensitivity of breast cancer to targeted therapy Roles of the Ras/Raf/MEK/ERK pathway in leukemia therapy. Antibodies to endothelial cells in primary vasculitides mediate in vitro endothelial cytotoxicity in the presence of normal peripheral blood mononuclear cells. Serum concentrations of the interferon-gamma-inducible chemokine IP-10/CXCL10 are augmented in both newly diagnosed Type I diabetes mellitus patients and subjects at risk of developing the disease Interleukin-13 prevents autoimmune diabetes in NOD mice Ras/Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR cascade inhibitors: how mutations can result in therapy resistance and how to overcome resistance GSK-3 as potential target for therapeutic intervention in cancer Multifaceted roles of GSK-3 and Wnt/β-catenin in hematopoiesis and leukemogenesis: opportunities for therapeutic intervention. Molecular mechanisms involved in NAFLD progression Endothelial activation by aPL: a potential pathogenetic mechanism for the clinical manifestations of the syndrome Parkinson’s disease is associated with increased serum levels of macrophage migration inhibitory factor Murine concanavalin A-induced hepatitis is prevented by interleukin 12 (IL-12) antibody and exacerbated by exogenous IL-12 through an interferon-gamma-dependent mechanism Macrophage migration inhibitory factor (MIF) is necessary for progression of autoimmune diabetes mellitus Deregulation of the EGFR/PI3K/PTEN/Akt/mTORC1 pathway in breast cancer: possibilities for therapeutic intervention Advances in targeting signal transduction pathways The Raf/MEK/ERK pathway can govern drug resistance, apoptosis and sensitivity to targeted therapy Roles of the Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR pathways in controlling growth and sensitivity to therapy-implications for cancer and aging PIK3CA mutations in human solid tumors: role in sensitivity to various therapeutic approaches Effects of tumour necrosis factor-alpha on human sperm motility and apoptosis Protection from experimental autoimmune diabetes in the non-obese diabetic mouse with soluble interleukin-1 receptor Increased serum levels of interleukin-18 in patients with multiple sclerosis IL-18 binding protein fusion construct delays the development of diabetes in adoptive transfer and cyclophosphamide-induced diabetes in NOD mouse. Extrahepatic disorders of HCV infection: a distinct entity of B-cell neoplasia? Association of t(14;18) translocation with HCV infection in gastrointestinal MALT lymphomas. Prevalence of hepatitis C virus infection among health-care workers: A 10-year survey. MIF in autoimmunity and novel therapeutic approaches. In vitro type-1 and type-2 cytokine production in systemic lupus erythematosus: lack of relationship with clinical disease activity Beneficial effects of r-h-CLU on disease severity in different animal models of peripheral neuropathies Serum levels of the interferon-gamma-inducing cytokine interleukin-18 are increased in individuals at high risk of developing type I diabetes Combination of cyclophosphamide and interferon-beta halts progression in patients with rapidly transitional multiple sclerosis Resistance to TRAIL and how to surmount it. Apotransferrin inhibits interleukin-2 expression and protects mice from experimental autoimmune encephalomyelitis. BRAF inhibition improves tumor recognition by the immune system: Potential implications for combinatorial therapies against melanoma involving adoptive T-cell transfer Preventive and curative effects of cyclophosphamide in an animal model of Guillain Barrè syndrome Ectopic NGAL expression can alter sensitivity of breast cancer cells to EGFR, Bcl-2, CaM-K inhibitors and the plant natural product berberine Targeting the translational apparatus to improve leukemia therapy: roles of the PI3K/PTEN/Akt/mTOR pathway

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