About: NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure

Facets (new session)
Description
Metadata
Settings
- owl:sameAs
- Inference Rule:

About: NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure Goto Sponge NotDistinct Permalink

An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

scientific article published on 12 May 2008

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в травні 2008 (uk) scientific article published on 12 May 2008 (en) 2008 թվականի մայիսի 12-ին հրատարակված գիտական հոդված (hy) artículu científicu espublizáu en mayu de 2008 (ast) 2008 മെയ് 12 നു പ്രസിദ്ധീകരിച്ച ശാസ്ത്ര ലേഖനം (ml)
publication date	wds:Q81269550-DB15B012-DF9B-4668-BAD7-D1635B431154
publication date	2008-05-12 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q81269550-E8135898-7DD0-4C8D-8F14-7EF28DA42FB9
language of work or name	English
author name string	wds:Q81269550-1AF39912-CFC1-458A-86EF-D726EC957496 wds:Q81269550-48389740-4EAF-4AB7-8451-82F98C6EFBB3 wds:Q81269550-4A295E9C-1AB5-49AD-ACBC-5C12B2CF8472 wds:Q81269550-4DE782A0-3C94-49DD-B396-17523E06401F wds:Q81269550-9E3ECCA7-1CAB-460C-BB9F-F4C4F23DA90D wds:Q81269550-EE13F6E6-0014-4AF8-899B-31AFA060CBD2 wds:Q81269550-F9FA5F20-8F91-4ABE-9E9D-CB323708D7E9
author name string	Anne-Sophie Armand Leon J De Windt Sylvia Heeneman Roel van der Nagel Paula A da Costa Martins Meriem Bourajjaj Xander H Wehrens
rdfs:label	NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure (en) NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure (nl)
skos:prefLabel	NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure (en) NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure (nl)
name	NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure (en) NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure (nl)
author	wds:Q81269550-25FB75AC-7FA8-4B57-AA90-EF3DA8C5D1FE
author	Bart Weijts
title	wds:Q81269550-582DF480-6619-412D-A476-ABD44E8FCB44
title	NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure (en)
page(s)	wds:Q81269550-18AD6F7F-DC88-4BD2-AADD-81928268DF38
page(s)	22295-22303
instance of	wds:Q81269550-F60F89DD-D925-4AFE-8D6F-A8E4A4B45E62
instance of	scholarly article
main subject	wds:Q81269550-BB09A626-8667-4722-A877-EF630F21B239
main subject	heart failure
PubMed ID	wds:Q81269550-A3D21D0F-0194-447D-AF6E-7503C39A22C4
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/18477567
PubMed ID	18477567
published in	wds:Q81269550-44EEE194-9789-4944-A037-D2871E9A8D13
published in	Journal of Biological Chemistry
issue	wds:Q81269550-E7B28D72-9C71-4287-AE81-861E7D53EF7A
volume	wds:Q81269550-4E11F912-9555-4BA4-A9FE-998E7CAE2B60
issue	32
volume	283
DOI	wds:Q81269550-7E3EFC39-97CF-46FF-9E9E-06EB29A081A5
DOI	http://dx.doi.org/10.1074/JBC.M801296200
DOI	10.1074/JBC.M801296200
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q81269550
is cites work of	MicroRNA-199b targets the nuclear kinase Dyrk1a in an auto-amplification loop promoting calcineurin/NFAT signalling. The functional role of calcineurin in hypertrophy, regeneration, and disorders of skeletal muscle. MEK1 inhibits cardiac PPARα activity by direct interaction and prevents its nuclear localization What causes a broken heart--molecular insights into heart failure Molecular pathways underlying cardiac remodeling during pathophysiological stimulation Cooperative synergy between NFAT and MyoD regulates myogenin expression and myogenesis Nfat and miR-25 cooperate to reactivate the transcription factor Hand2 in heart failure miR-222 is necessary for exercise-induced cardiac growth and protects against pathological cardiac remodeling Targeted inhibition of calpain reduces myocardial hypertrophy and fibrosis in mouse models of type 1 diabetes Parsing good versus bad signaling pathways in the heart: role of calcineurin-nuclear factor of activated T-cells Syndecan-4 is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT pathway Chapter 2. Calcineurin signaling and the slow oxidative skeletal muscle fiber type The scaffold protein muscle A-kinase anchoring protein β orchestrates cardiac myocyte hypertrophic signaling required for the development of heart failure. Syndecan-4 signaling via NFAT regulates extracellular matrix production and cardiac myofibroblast differentiation in response to mechanical stress Interaction between NFκB and NFAT coordinates cardiac hypertrophy and pathological remodeling Counteracting Protein Kinase Activity in the Heart: The Multiple Roles of Protein Phosphatases Cardiomyocyte-specific deletion of the vitamin D receptor gene results in cardiac hypertrophy Prox1 maintains muscle structure and growth in the developing heart The transcription factor Eya2 prevents pressure overload-induced adverse cardiac remodeling CIB1 is a regulator of pathological cardiac hypertrophy Small changes can make a big difference - microRNA regulation of cardiac hypertrophy Variation at the NFATC2 locus increases the risk of thiazolidinedione-induced edema in the Diabetes REduction Assessment with ramipril and rosiglitazone Medication (DREAM) study AKAPs: the architectural underpinnings of local cAMP signaling. mAKAP-a master scaffold for cardiac remodeling. Transverse aortic constriction in mice Peroxisome proliferator-activated receptor (PPAR) gene profiling uncovers insulin-like growth factor-1 as a PPARalpha target gene in cardioprotection Regulation of the stability and transcriptional activity of NFATc4 by ubiquitination Cardiomyocyte calcineurin signaling in subcellular domains: from the sarcolemma to the nucleus and beyond Cardiac anti-remodelling effect of aerobic training is associated with a reduction in the calcineurin/NFAT signalling pathway in heart failure mice. Protein kinase D3 is a pivotal activator of pathological cardiac hypertrophy by selectively increasing the expression of hypertrophic transcription factors Association of promoter methylation statuses of congenital heart defect candidate genes with Tetralogy of Fallot MicroRNAs in the onset and development of cardiovascular disease Extracellular signal-regulated kinase activation during cardiac hypertrophy reduces sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) transcription Molecular and Cellular Mechanisms of Cardiovascular Disorders in Diabetes Identification of exercise capacity QTL using association mapping in inbred mice The role of the paracrine/autocrine mediator endothelin-1 in regulation of cardiac contractility and growth. Cardiac fibroblasts on the fast track--scleraxis: from Achilles' heel to anti-fibrotic therapy. Beyond the cardiac myofilament: hypertrophic cardiomyopathy- associated mutations in genes that encode calcium-handling proteins Adenylyl cyclase type 5 in cardiac disease, metabolism, and aging. In vitro and in vivo direct monitoring of miRNA-22 expression in isoproterenol-induced cardiac hypertrophy by bioluminescence imaging. Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac Growth. Cathepsin K knockout protects against cardiac dysfunction in diabetic mice LncRNA ZFAS1 as a SERCA2a Inhibitor to Cause Intracellular Ca2+ Overload and Contractile Dysfunction in a Mouse Model of Myocardial Infarction. Calcineurin signaling in the heart: The importance of time and place. SIRT2 deacetylase represses NFAT transcription factor to maintain cardiac homeostasis. Alteration in microRNA-25 expression regulate cardiac function via renin secretion.

Faceted Search & Find service v1.16.117 as of May 05 2024

Alternative Linked Data Documents: ODE Content Formats:

RDF

ODATA

Microdata

About

OpenLink Virtuoso version 07.20.3239 as of May 5 2024, on Linux (x86_64-centos_6-linux-gnu), Single-Server Edition (378 GB total memory, 172 GB memory in use)
Data on this page belongs to its respective rights holders.
Virtuoso Faceted Browser Copyright © 2009-2024 OpenLink Software