About: Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats

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About: Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats Goto Sponge NotDistinct Permalink

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scientific article published on 01 August 1998

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в серпні 1998 (uk) article scientifique publié en 1998 (fr) im August 1998 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'agostu de 1998 (ast) scientific article published on 01 August 1998 (en)
publication date	wds:Q77156044-EE584783-EB7B-4DD0-8D4E-1858E768D68F
publication date	1998-08-01 00:00:00Z (xsd:dateTime)
author name string	wds:Q77156044-0E7BF3A4-5286-4E33-B44E-3D334796E34C wds:Q77156044-42ABCB57-A697-4A44-B751-9F2596502040 wds:Q77156044-4B05ED16-6565-4763-AC89-0A0FD8C5DFD7 wds:Q77156044-545259CA-7680-4B75-B6CD-2AE83915D69A wds:Q77156044-5D23F7D8-6E96-4D93-B202-4E12C1EE469B wds:Q77156044-70C52A8D-77E2-44FA-93F3-3F3943FBFD7C wds:Q77156044-73AAFC76-2E22-4252-982F-F9DAC2B8E7DE wds:Q77156044-9F3BE95B-4429-42C8-A32F-E13D639D6ABA wds:Q77156044-CAB69EF5-9C2B-4A6B-8CC6-0D66E7A6D543 wds:Q77156044-D86DD17A-35E8-4D2E-A4B0-66C946816F02
author name string	H Yamamoto M Katoh K Tamaki A Takeshita H Tomita H Shimokawa K Egashira M Koyanagi M Takemoto Y Ohara
rdfs:label	Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats (en) Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats (nl)
skos:prefLabel	Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats (en) Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats (nl)
name	Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats (en) Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats (nl)
title	wds:Q77156044-EB6BB24F-96F7-45C1-A76B-F394BDC0254E
title	Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats (en)
page(s)	wds:Q77156044-5C980CFA-5004-4E14-ACA6-92FDB63F1FAE
page(s)	273-279
instance of	wds:Q77156044-FC1545C4-A175-4CA8-B072-4A0E5613634E
instance of	scholarly article
PubMed ID	wds:Q77156044-42A18249-EE6A-4FBC-A87E-84BB72B4C36C
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/9719054
PubMed ID	9719054
published in	wds:Q77156044-43C2170F-043B-4B04-9E6E-DA0806B4421F
published in	Hypertension
issue	wds:Q77156044-6C85CB1F-70FB-4237-9642-430EA2B0F783
volume	wds:Q77156044-454D415B-4ACD-4302-A709-C847DA86726A
issue	2
volume	32
DOI	wds:Q77156044-E190443B-528E-4DAF-B19B-598826E7A0B4
DOI	http://dx.doi.org/10.1161/01.HYP.32.2.273
DOI	10.1161/01.HYP.32.2.273
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q77156044
is cites work of	TGF-beta-dependent pathogenesis of mitral valve prolapse in a mouse model of Marfan syndrome TGF-beta and fibrosis Endothelial-to-mesenchymal transition contributes to cardiac fibrosis Fibrosis in heart disease: understanding the role of transforming growth factor-beta in cardiomyopathy, valvular disease and arrhythmia Dysregulation of TGF-beta activation contributes to pathogenesis in Marfan syndrome Matrix metalloproteinases: pathways of induction by bioactive molecules Control of liver cell fate decision by a gradient of TGF beta signaling modulated by Onecut transcription factors Ac-SDKP reverses inflammation and fibrosis in rats with heart failure after myocardial infarction. Angiotensin II and nitric oxide interaction. Deletion of phd2 in myeloid lineage attenuates hypertensive cardiovascular remodeling Olmesartan medoxomil: a review of its use in the management of hypertension Differential expression of embryonic epicardial progenitor markers and localization of cardiac fibrosis in adult ischemic injury and hypertensive heart disease Blockade of transforming growth factor-beta1 accelerates lymphatic regeneration during wound repair Clinical implications of matrix metalloproteinases Autophagy is a regulator of TGF-β1-induced fibrogenesis in primary human atrial myofibroblasts Angiotensin II and connective tissue: homeostasis and reciprocal regulation Sulfatases are determinants of alveolar formation Angiotensin II blockade reverses myocardial fibrosis in a transgenic mouse model of human hypertrophic cardiomyopathy Diastolic dysfunction of aging is independent of myocardial structure but associated with plasma advanced glycation end-product levels Impact of type 2 diabetes and the metabolic syndrome on myocardial structure and microvasculature of men with coronary artery disease Transforming growth factor beta1 genotype and change in left ventricular mass during antihypertensive treatment--results from the Swedish Irbesartan Left Ventricular Hypertrophy Investigation versus Atenolol (SILVHIA). Fibrosis and heart failure Angiotensin II and the pathophysiology of cardiovascular remodeling Novel therapy for myocardial infarction: can HGF/Met be beneficial? Inhibition of transforming growth factor-beta signaling induces left ventricular dilation and dysfunction in the pressure-overloaded heart Olmesartan medoxomil: current status of its use in monotherapy Ac-SDKP inhibits transforming growth factor-beta1-induced differentiation of human cardiac fibroblasts into myofibroblasts Olmesartan medoxomil Paradoxes of nitric oxide in the diabetic kidney Olmesartan medoxomil for the treatment of hypertension in children and adolescents Differential effects of arginine methylation on diastolic dysfunction and disease progression in patients with chronic systolic heart failure Fibrosis in hypertensive heart disease: role of the renin-angiotensin-aldosterone system The association between systemic sclerosis, arginine and asymmetric dimethylarginine Counter-regulation by atorvastatin of gene modulations induced by L-NAME hypertension is associated with vascular protection Provinol prevents CsA-induced nephrotoxicity by reducing reactive oxygen species, iNOS, and NF-kB expression Delivery of an engineered HGF fragment in an extracellular matrix-derived hydrogel prevents negative LV remodeling post-myocardial infarction Thrombospondins in the heart: potential functions in cardiac remodeling Extracellular matrix alterations in cardiomyopathy: The possible crucial role in the dilative form. CCAAT/enhancer-binding protein β overexpression alleviates myocardial remodelling by regulating angiotensin-converting enzyme-2 expression in diabetes. Myocardial remodeling in viral heart disease: possible interactions between inflammatory mediators and MMP-TIMP system Mechanisms underlying the cardiac antifibrotic effects of losartan metabolites. Fibroblasts in an endocardial fibroelastosis disease model mainly originate from mesenchymal derivatives of epicardium. Time course of cardiac inflammation during nitric oxide synthase inhibition in SHR: impact of prior transient ACE inhibition Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α' Serum concentrations of asymmetric and symmetric dimethylarginine are associated with mortality in acute heart failure patients. The axis of local cardiac endogenous Klotho-TGF-β1-Wnt signaling mediates cardiac fibrosis in human

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