About: Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair

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About: Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair Goto Sponge NotDistinct Permalink

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scientific article published on 01 August 2000

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в серпні 2000 (uk) article scientifique publié en 2000 (fr) im August 2000 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'agostu de 2000 (ast) scientific article published on 01 August 2000 (en)
publication date	wds:Q74199089-3FC89B41-4DFA-423D-BDEB-59F62D590D6B
publication date	2000-08-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q74199089-E6B9F4E1-51A2-4DCB-A0E3-069621501DB5
language of work or name	English
exact match	wds:Q74199089-91211191-427A-4015-B629-A5773473A556
exact match	https://scigraph.springernature.com/pub.10.1046/j.1523-1747.2000.00029.x
author name string	wds:Q74199089-44A59F86-8C52-4878-BF5C-C4338FF0BD0A wds:Q74199089-483BB7EB-93B5-4CFA-BCFF-6A9B5BAB8AAA wds:Q74199089-8B644191-12D1-4018-856A-7A08FF2934A8 wds:Q74199089-A5ED5565-D940-451A-A14A-25D8C7DDFF64 wds:Q74199089-CFE0FD67-BCFE-4655-B3B2-D706B6AD23C9
author name string	Frank S Pfeilschifter J Kämpfer H Stallmeyer B Wetzler C
rdfs:label	Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair (en) Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair (nl)
skos:prefLabel	Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair (en) Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair (nl)
name	Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair (en) Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair (nl)
title	wds:Q74199089-C2AE16D5-8682-4D77-95E4-F0F481AD099F
title	Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair (en)
page(s)	wds:Q74199089-86E6698E-D479-4E5A-B218-4E30E7652800
page(s)	245-253
instance of	wds:Q74199089-FD41EAC7-8F21-49A8-BC75-8BF75F67432C
instance of	scholarly article
main subject	wds:Q74199089-82B7843A-2149-428E-B964-F8D54DCDA5EA
main subject	wound healing
PubMed ID	wds:Q74199089-F9FB52E6-FD8C-449F-B8BD-DA0EB294C5F9
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/10951242
PubMed ID	10951242
published in	wds:Q74199089-EAC35F8D-B465-4FEC-9CA4-9FC394A336F5
published in	Journal of Investigative Dermatology
issue	wds:Q74199089-FBA16954-8D7F-4AB7-AEDF-12BF243C7A7D
volume	wds:Q74199089-714AAAC7-C719-4916-889F-C45511C4838D
issue	2
volume	115
DOI	wds:Q74199089-DD852709-2FE7-4310-8E03-780F3943B48B
DOI	http://dx.doi.org/10.1046/J.1523-1747.2000.00029.X
DOI	10.1046/J.1523-1747.2000.00029.X
Dimensions Publication ID	wds:Q74199089-CBA5EC69-B80A-4336-86DE-7ECE84CF4894
Dimensions Publication ID	1002429506
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q74199089
is cites work of	Combining experimental and mathematical modeling to reveal mechanisms of macrophage-dependent left ventricular remodeling Tissue regeneration using macrophage migration inhibitory factor-impregnated gelatin microbeads in cutaneous wounds Chemokine expression of CCL2, CCL3, CCL5 and CXCL10 during early inflammatory tendon healing precedes nerve regeneration: an immunohistochemical study in the rat Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury Chemokines in Wound Healing and as Potential Therapeutic Targets for Reducing Cutaneous Scarring Enhanced lymph vessel density, remodeling, and inflammation are reflected by gene expression signatures in dermal lymphatic endothelial cells in type 2 diabetes Nitric oxide releasing nanoparticles are therapeutic for Staphylococcus aureus abscesses in a murine model of infection Blockade of receptor for advanced glycation end-products restores effective wound healing in diabetic mice. Restraint stress alters neutrophil and macrophage phenotypes during wound healing Altered CXCR2 signaling in beta-arrestin-2-deficient mouse models Glucose toxic effects on granulation tissue productive cells: the diabetics' impaired healing Vascular endothelial growth factor-C accelerates diabetic wound healing Low-intensity vibration improves angiogenesis and wound healing in diabetic mice HOXA3 modulates injury-induced mobilization and recruitment of bone marrow-derived cells Microbial symbionts accelerate wound healing via the neuropeptide hormone oxytocin Macrophage dysfunction impairs resolution of inflammation in the wounds of diabetic mice Inflammation and wound healing: the role of the macrophage Exercise, Obesity, and Cutaneous Wound Healing: Evidence from Rodent and Human Studies Wound healing in mice with high-fat diet- or ob gene-induced diabetes-obesity syndromes: a comparative study Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha Systemic anti-TNFalpha treatment restores diabetes-impaired skin repair in ob/ob mice by inactivation of macrophages. Impaired wound healing in mouse models of diabetes is mediated by TNF-alpha dysregulation and associated with enhanced activation of forkhead box O1 (FOXO1). Osteopontin: a multifunctional protein at the crossroads of inflammation, atherosclerosis, and vascular calcification A transgenic mouse model of inducible macrophage depletion: effects of diphtheria toxin-driven lysozyme M-specific cell lineage ablation on wound inflammatory, angiogenic, and contractive processes. Epigenetic changes in bone marrow progenitor cells influence the inflammatory phenotype and alter wound healing in type 2 diabetes. Macrophage PPARγ and impaired wound healing in type 2 diabetes The role of microRNA-146a in the pathogenesis of the diabetic wound-healing impairment: correction with mesenchymal stem cell treatment TLR2 expression and signaling-dependent inflammation impair wound healing in diabetic mice Topical vascular endothelial growth factor accelerates diabetic wound healing through increased angiogenesis and by mobilizing and recruiting bone marrow-derived cells Diabetes induces stable intrinsic changes to myeloid cells that contribute to chronic inflammation during wound healing in mice Longitudinal shift in diabetic wound microbiota correlates with prolonged skin defense response Quercetin protects against diabetes-induced exaggerated vasoconstriction in rats: effect on low grade inflammation. The functional behavior of a macrophage/fibroblast co-culture model derived from normal and diabetic mice with a marine gelatin-oxidized alginate hydrogel Maggot secretions suppress pro-inflammatory responses of human monocytes through elevation of cyclic AMP. Proresolution therapy for the treatment of delayed healing of diabetic wounds. Resolvins, specialized proresolving lipid mediators, and their potential roles in metabolic diseases CXC chemokines and their receptors: a case for a significant biological role in cutaneous wound healing Increased saturated fatty acids in obesity alter resolution of inflammation in part by stimulating prostaglandin production Plasmin modulates vascular endothelial growth factor-A-mediated angiogenesis during wound repair Decreased macrophage number and activation lead to reduced lymphatic vessel formation and contribute to impaired diabetic wound healing. A novel primate model of delayed wound healing in diabetes: dysregulation of connective tissue growth factor Age-dependent impairment of HIF-1alpha expression in diabetic mice: Correction with electroporation-facilitated gene therapy increases wound healing, angiogenesis, and circulating angiogenic cells Wound administration of M2-polarized macrophages does not improve murine cutaneous healing responses. Pivotal role for alpha1-antichymotrypsin in skin repair The innate immune system, toll-like receptors and dermal wound healing: A review Diabetic complications and dysregulated innate immunity Blocking interleukin-1β induces a healing-associated wound macrophage phenotype and improves healing in type 2 diabetes Comparison of the effect of topical versus systemic L-arginine on wound healing in acute incisional diabetic rat model

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