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| - wetenschappelijk artikel (nl)
- наукова стаття, опублікована в лютому 2000 (uk)
- im Februar 2000 veröffentlichter wissenschaftlicher Artikel (de)
- wüsseschaftlicher Artikel, wo im Hornig 2000 veröffentlicht worden isch (gsw)
- artículu científicu espublizáu en febreru de 2000 (ast)
- artikull shkencor i botuar më 01 shkurt 2000 (sq)
- scientific article published on 01 February 2000 (en)
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| - K A Fitzgerald
- B S Skeffington
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| - Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (en)
- Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (nl)
- Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (sq)
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| skos:prefLabel
| - Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (en)
- Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (nl)
- Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (sq)
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| name
| - Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (en)
- Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (nl)
- Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (sq)
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| title
| - Ras, protein kinase C zeta, and I kappa B kinases 1 and 2 are downstream effectors of CD44 during the activation of NF-kappa B by hyaluronic acid fragments in T-24 carcinoma cells (en)
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| DOI
| - 10.4049/JIMMUNOL.164.4.2053
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| is cites work
of | - LPS-TLR4 signaling to IRF-3/7 and NF-kappaB involves the toll adapters TRAM and TRIF
- Hyaluronan and its catabolic products in tissue injury and repair
- Interaction of CD44 with Different Forms of Hyaluronic Acid. Its Role in Adhesion and Migration of Tumor Cells
- The interaction of versican with its binding partners
- Heart-valve mesenchyme formation is dependent on hyaluronan-augmented activation of ErbB2-ErbB3 receptors
- Identification of sequence, protein isoforms, and distribution of the hyaluronan-binding protein RHAMM in adult and developing rat brain
- A crucial role for CD44 in inflammation
- CD44 signaling through focal adhesion kinase and its anti-apoptotic effect
- Hyaluronan fragments activate nitric oxide synthase and the production of nitric oxide by articular chondrocytes
- MEK5, a new target of the atypical protein kinase C isoforms in mitogenic signaling
- Protein kinase C iota protects neural cells against apoptosis induced by amyloid beta-peptide.
- The dual functions of GPI-anchored PH-20: hyaluronidase and intracellular signaling
- Regulation of interferon-gamma during innate and adaptive immune responses
- Cortactin underpins CD44-promoted invasion and adhesion of breast cancer cells to bone marrow endothelial cells
- Molecular prognostication in bladder cancer--a current perspective.
- Hyaluronan: a multifunctional, megaDalton, stealth molecule
- Hyaluronan blocks human neutrophil elastase (HNE)-induced airway responses in sheep
- Interactions between Hyaluronan and Its Receptors (CD44, RHAMM) Regulate the Activities of Inflammation and Cancer
- Hyaluronan activates cell motility of v-Src-transformed cells via Ras-mitogen-activated protein kinase and phosphoinositide 3-kinase-Akt in a tumor-specific manner
- The role of hyaluronan synthase 3 in ventilator-induced lung injury.
- Hyaluronan is not just a goo!
- The role of Toll-like receptors in non-infectious lung injury
- Suppressor activity and potency among regulatory T cells is discriminated by functionally active CD44
- Threat matrix: low-molecular-weight hyaluronan (HA) as a danger signal
- Prognostic value of the expression of phosphatase and tensin homolog and CD44 in elderly patients with refractory acute myeloid leukemia
- Hyaluronate fragments reverse skin atrophy by a CD44-dependent mechanism
- Hyaluronan oligosaccharides induce matrix metalloproteinase 13 via transcriptional activation of NFkappaB and p38 MAP kinase in articular chondrocytes
- Regulatory pathways involved in the infection-induced production of IFN-gamma by NK cells
- Hyaluronan oligosaccharide-induced activation of transcription factors in bovine articular chondrocytes
- Mechanisms involved in enhancement of the expression and function of aggrecanases by hyaluronan oligosaccharides
- Intracellular domain fragment of CD44 alters CD44 function in chondrocytes
- Intracellular signaling of cardiac fibroblasts
- Inhibition of HA synthase 3 mRNA expression, with a phosphodiesterase 3 inhibitor, blocks lung injury in a septic ventilated rat model.
- Hyaluronan - a functional and structural sweet spot in the tissue microenvironment
- Proteins involved in the production and perception of oligosaccharides in relation to plant and animal development.
- CD44 is a potential target of amplification within the 11p13 amplicon detected in gastric cancer cell lines
- TLRs play good cop, bad cop in the lung
- Chemically modified N-acylated hyaluronan fragments modulate proinflammatory cytokine production by stimulated human macrophages
- CD44 stimulation by fragmented hyaluronic acid induces upregulation and tyrosine phosphorylation of c-Met receptor protein in human chondrosarcoma cells
- Sorafenib controls the epithelial‑mesenchymal transition of ovarian cancer cells via EGF and the CD44‑HA signaling pathway in a cell type‑dependent manner
- Serglycin in tumor microenvironment promotes non-small cell lung cancer aggressiveness in a CD44-dependent manner
- Essential role of protein kinase C zeta in transducing a motility signal induced by superoxide and a chemotactic peptide, fMLP.
- Liver Cancer Initiation Requires p53 Inhibition by CD44-Enhanced Growth Factor Signaling
- Planning, evaluating and vetting receptor signaling studies to assess hyaluronan size-dependence and specificity
- What is special about 200 kDa hyaluronan that activates hyaluronan receptor signaling?
- A role for Akt in the rapid regulation of inflammatory and apoptotic pathways in mouse bladder
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