About: Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium

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About: Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium Goto Sponge NotDistinct Permalink

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scientific article published on 01 August 1994

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в серпні 1994 (uk) im August 1994 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'agostu de 1994 (ast) scientific article published on 01 August 1994 (en)
publication date	wds:Q72092902-6C2FEE46-81CB-41F9-82FA-CF574179B58F
publication date	1994-08-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q72092902-B7446203-6365-4B8A-821B-BCD32A6F8EB9
language of work or name	English
author name string	wds:Q72092902-19BE0302-FBF8-41D2-B13D-D2BF332C7BA8 wds:Q72092902-511EE1C9-3F89-4E74-9415-54C0F258BB49 wds:Q72092902-B1CD134D-DE25-4E86-BFD9-AB2C1E0A1310 wds:Q72092902-D5A5A3DD-B317-4472-85CF-A4FFAD0D5D11
author name string	L R Jones M Karimi K Green M A Movsesian
rdfs:label	Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium (en) Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium (nl)
skos:prefLabel	Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium (en) Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium (nl)
name	Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium (en) Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium (nl)
title	wds:Q72092902-C0D90335-A4AE-41CD-A92E-3D0BE36B233D
title	Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium (en)
page(s)	wds:Q72092902-2358C224-4860-40C0-8BC0-355CA12D64F1
page(s)	653-657
instance of	wds:Q72092902-7DC87606-796D-45CD-8784-1F8A252499B6
instance of	scholarly article
PubMed ID	wds:Q72092902-B974A89D-9FD1-4D19-A32A-8B65C5DC14C5
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/8044934
PubMed ID	8044934
published in	wds:Q72092902-C7DBAFB3-D179-40D6-A4AD-7177D6428053
published in	Circulation
issue	wds:Q72092902-3B41C469-24B0-4DC1-A8BC-7C2839856085
volume	wds:Q72092902-6E8C0566-BECA-452D-85A6-2E07FF7838F8
issue	2
volume	90
DOI	wds:Q72092902-32138DD2-E473-43B1-9C16-41FD6154352B
DOI	http://dx.doi.org/10.1161/01.CIR.90.2.653
DOI	10.1161/01.CIR.90.2.653
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q72092902
is cites work of	Expression of a Gi-coupled receptor in the heart causes impaired Ca2+ handling, myofilament injury, and dilated cardiomyopathy Interleukin-1 beta modulates the growth and phenotype of neonatal rat cardiac myocytes Heart failure with preserved ejection fraction: molecular pathways of the aging myocardium Human SERCA2a levels correlate inversely with age in senescent human myocardium. Cellular and molecular determinants of altered Ca2+ handling in the failing rabbit heart: primary defects in SR Ca2+ uptake and release mechanisms. Modulation of gene expression in transgenic mouse hearts overexpressing calsequestrin Altered myocardial calcium cycling and energetics in heart failure--a rational approach for disease treatment Enhanced myocardial relaxation in vivo in transgenic mice overexpressing the beta2-adrenergic receptor is associated with reduced phospholamban protein Diminished post-rest potentiation of contractile force in human dilated cardiomyopathy. Functional evidence for alterations in intracellular Ca2+ handling Regulation of Ca2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin Adrenergic and muscarinic receptor regulation and therapeutic implications in heart failure The sarco(endo)plasmic reticulum Ca(2+)-ATPase gene is regulated at the transcriptional level during compensated left ventricular hypertrophy in the rat Reduced level of serine(16) phosphorylated phospholamban in the failing rat myocardium: a major contributor to reduced SERCA2 activity Alterations of calcium-regulatory proteins in heart failure. Molecular aspects and gene therapy prospects for diastolic failure Sarcoplasmic reticulum Ca2+ATPase and phospholamban mRNA and protein levels in end-stage heart failure due to ischemic or dilated cardiomyopathy Right ventricular upregulation of the Ca(2+) binding protein S100A1 in chronic pulmonary hypertension Integrin-linked kinase mediates force transduction in cardiomyocytes by modulating SERCA2a/PLN function. Phospholamban: a crucial regulator of cardiac contractility Pathological ventricular remodeling: mechanisms: part 1 of 2 Nitric oxide synthase regulation of cardiac excitation-contraction coupling in health and disease Calcium regulatory proteins and their alteration by transgenic approaches Electrical and structural remodeling of the failing ventricle Sarcoplasmic reticulum Ca(2+)-ATPase overexpression by adenovirus mediated gene transfer and in transgenic mice Enhanced ryanodine receptor-mediated calcium leak determines reduced sarcoplasmic reticulum calcium content in chronic canine heart failure Transmural heterogeneity and remodeling of ventricular excitation-contraction coupling in human heart failure Differential regulation of two types of intracellular calcium release channels during end-stage heart failure Comparative analysis of the isoform expression pattern of Ca(2+)-regulatory membrane proteins in fast-twitch, slow-twitch, cardiac, neonatal and chronic low-frequency stimulated muscle fibers Calcium cycling proteins and force-frequency relationship in heart failure Kinetics studies of the cardiac Ca-ATPase expressed in Sf21 cells: new insights on Ca-ATPase regulation by phospholamban Beta 2-adrenoceptor activation by zinterol causes protein phosphorylation, contractile effects and relaxant effects through a cAMP pathway in human atrium Ca2+-ATPases in non-failing and failing heart: evidence for a novel cardiac sarco/endoplasmic reticulum Ca2+-ATPase 2 isoform (SERCA2c) Altered sarcoplasmic reticulum calcium cycling--targets for heart failure therapy Injury to the Ca2+ ATPase of the sarcoplasmic reticulum in anesthetized dogs contributes to myocardial reperfusion injury. Electrophysiological remodeling in heart failure Calcium transport proteins in the nonfailing and failing heart: gene expression and function Sarcoplasmic reticulum in heart failure: central player or bystander? Pathophysiology of Heart Failure Altered inotropism in the failing human myocardium Oxidative posttranslational modifications mediate decreased SERCA activity and myocyte dysfunction in Galphaq-overexpressing mice Characterizing phospholamban to sarco(endo)plasmic reticulum Ca2+-ATPase 2a (SERCA2a) protein binding interactions in human cardiac sarcoplasmic reticulum vesicles using chemical cross-linking The molecular and cellular pathophysiology of heart failure. Transcription factor AP-2alpha triggers apoptosis in cardiac myocytes Gene transfer in cardiac myocytes. Advances in gene-based therapy for heart failure Protein phosphorylation in isolated trabeculae from nonfailing and failing human hearts Improvement of cardiac sarcoplasmic reticulum calcium cycling in dogs with heart failure following long-term therapy with the Acorn Cardiac Support Device The ryanodine binding sarcoplasmic reticulum calcium release channel in nonfailing and in failing human myocardium Calcium channel blockers in cardiac failure Calcium handling proteins: structure, function, and modulation by exercise Myocyte contractile dysfunction with hypertrophy and failure: relevance to cardiac surgery Noblesse oblige Calcium and the Failing Heart: Phospholamban, Good Guy or Bad Guy? Human cardiomyocyte calcium handling and transverse tubules in mid-stage of post-myocardial-infarction heart failure. Association with SERCA2a directs phospholamban trafficking to sarcoplasmic reticulum from a nuclear envelope pool Effects of stimulation frequency on calcium transients in noninfarcted myocardium: modulation by chronic captopril treatment
is cites work of	wds:Q36022786-5660D32B-F370-4D15-AFE1-F9FFA5F1CD5F

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