About: Eddie C. Wang

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	ikertzailea (eu) researcher (en) 연구원 (ko) 研究者 (ja) araştırmacı (tr) дослідник (uk) изилдөөчү (ky) 研究者 (zh) onderzoeker (nl)
rdfs:label	Eddie C. Wang (en) Eddie C. Wang (tr) Wang EC (nl)
skos:prefLabel	Eddie C. Wang (en) Eddie C. Wang (tr) Wang EC (nl)
name	Eddie C. Wang (en) Eddie C. Wang (tr) Wang EC (nl)
given name	wds:Q71829139-AB999509-A17B-40FA-9FBC-7831D0D34E77
given name	Eddie
instance of	wds:Q71829139-650D85FB-C665-45B0-85B6-DE07F729CEB5
instance of	Homo sapiens
Loop ID	wds:Q71829139-50B7DB04-243B-4DAF-88CA-14670F995B96
Loop ID	588461
ResearchGate profile ID	wds:Q71829139-04A527BA-0343-470C-914F-F7AB5FCDB6A2
ResearchGate profile ID	Eddie_Wang
Google Scholar author ID	wds:Q71829139-4AB5C476-DA35-4917-B985-D2E1DF61BE44
Google Scholar author ID	DgvpthYAAAAJ
skos:altLabel	Eddie Wang (en) Wang EC (en)
occupation	wds:Q71829139-A5FE2292-900A-4703-9B67-10B66AE953D1
employer	wds:Q71829139-6F4EB7B3-0AD1-49EA-B986-B6051A709F76 wds:Q71829139-702D7651-27C6-4775-BDE8-5495CE0F4EA1 wds:Q71829139-A7F941C2-C7D3-46BD-A1E8-E0F1605E6288 wds:Q71829139-D194A252-FDD7-4476-AD9F-52FBF721DBCC
sex or gender	wds:Q71829139-BAD52D8A-DAD8-4E89-89A4-EE422AC363EB
educated at	wds:Q71829139-01ACA6F4-CF0C-42A8-90B6-F11DB4B429EF wds:Q71829139-2C16A1E4-74B5-4A63-AEA8-A0F26BE757A7 wds:Q71829139-E5AB8A3E-D400-4209-B8E5-E8D467066BC5
occupation	researcher
employer	Cardiff University School of Medicine Imperial Cancer Research Fund
sex or gender	male
educated at	John Ffowcs Williams
ORCID iD	wds:Q71829139-41B6E946-136E-4FEB-ADBA-AE71271BFF85
ORCID iD	https://orcid.org/0000-0002-2243-4964
ORCID iD	0000-0002-2243-4964
Gateway to Research person ID	wds:Q71829139-414D1239-F212-47ED-A05C-0B2C63A3C76B
Gateway to Research person ID	C7C0FC1F-2464-4404-9921-CAFD2EB08C64
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q71829139
is author of	The TNF-like protein 1A-death receptor 3 pathway promotes macrophage foam cell formation in vitro. Human cytomegalovirus: taking the strain UL40-mediated NK evasion during productive infection with human cytomegalovirus. The human cytomegalovirus MHC class I homolog UL18 inhibits LIR-1+ but activates LIR-1- NK cells. The paradox of NKp46+ natural killer cells: drivers of severe hepatitis C virus-induced pathology but in-vivo resistance to interferon α treatment. Genomic structure, expression, and chromosome mapping of the mouse homologue for the WSL-1 (DR3, Apo3, TRAMP, LARD, TR3, TNFRSF12) gene. The switch from latent to productive infection in epstein-barr virus-infected B cells is associated with sensitization to NK cell killing. Increased CD200 expression in acute myeloid leukemia is linked with an increased frequency of FoxP3+ regulatory T cells. Quantitative temporal viromics: an approach to investigate host-pathogen interaction CD200 expression suppresses natural killer cell function and directly inhibits patient anti-tumor response in acute myeloid leukemia Analysis of the human T-cell response to picornaviruses: identification of T-cell epitopes close to B-cell epitopes in poliovirus. Sustained TL1A expression modulates effector and regulatory T-cell responses and drives intestinal goblet cell hyperplasia. DR3 regulates negative selection during thymocyte development The TNF-family receptor DR3 is essential for diverse T cell-mediated inflammatory diseases Adenovirus E3/19K promotes evasion of NK cell recognition by intracellular sequestration of the NKG2D ligands major histocompatibility complex class I chain-related proteins A and B. Proliferation and interleukin 5 production by CD8hi CD57+ T cells. HCMV pUL135 remodels the actin cytoskeleton to impair immune recognition of infected cells Cytomegalovirus destruction of focal adhesions revealed in a high-throughput Western blot analysis of cellular protein expression. Human cytomegalovirus glycoprotein UL141 targets the TRAIL death receptors to thwart host innate antiviral defenses. The death receptor 3/TL1A pathway is essential for efficient development of antiviral CD4⁺ and CD8⁺ T-cell immunity. A central role for alpha beta T cells in the pathogenesis of murine lupus A Novel Role for TL1A/DR3 in Protection against Intestinal Injury and Infection. Human cytomegalovirus infection downregulates expression of the cellular aminopeptidases CD10 and CD13. Functional differences between influenza A-specific cytotoxic T lymphocyte clones expressing dominant and subdominant TCR Adenovirus vector delivery stimulates natural killer cell recognition. The Death Receptor 3-TNF-like protein 1A pathway drives adverse bone pathology in inflammatory arthritis. CD8highCD57+ T lymphocytes in normal, healthy individuals are oligoclonal and respond to human cytomegalovirus. Two novel human cytomegalovirus NK cell evasion functions target MICA for lysosomal degradation The measurement of stress-related immune dysfunction in psychoneuroimmunology. Naive and activated T cells display differential responsiveness to TL1A that affects Th17 generation, maintenance, and proliferation. DR3 signaling protects against cisplatin nephrotoxicity mediated by tumor necrosis factor. Human cytomegalovirus UL40 signal peptide regulates cell surface expression of the NK cell ligands HLA-E and gpUL18 Regulation of early cartilage destruction in inflammatory arthritis by death receptor 3 A high-efficiency system of natural killer cell cloning. An essential role for death receptor 3 in experimental autoimmune uveoretinitis. Death Receptor 3 Promotes Chemokine-Directed Leukocyte Recruitment in Acute Resolving Inflammation and Is Essential for Pathological Development of Mesothelial Fibrosis in Chronic Disease. Expression of CD200 on AML blasts directly suppresses memory T-cell function. Inhibitory killer cell immunoglobulin-like receptors strengthen CD8 T cell-mediated control of HIV-1, HCV, and HTLV-1 Subsets of CD8+, CD57+ cells in normal, healthy individuals: correlations with human cytomegalovirus (HCMV) carrier status, phenotypic and functional analyses RORα-expressing T regulatory cells restrain allergic skin inflammation. The inhibitor of cyclin-dependent kinases, olomoucine II, exhibits potent antiviral properties. HCMV-Encoded NK Modulators: Lessons From and Genetic Variation High-Definition Analysis of Host Protein Stability during Human Cytomegalovirus Infection Reveals Antiviral Factors and Viral Evasion Mechanisms Review: Novel Insights Into Tumor Necrosis Factor Receptor, Death Receptor 3, and Progranulin Pathways in Arthritis and Bone Remodeling Death Receptor 3 (TNFRSF25) Increases Mineral Apposition by Osteoblasts and Region Specific New Bone Formation in the Axial Skeleton of Male DBA/1 Mice Oestrogen-deficiency induces bone loss by modulating CD14+ monocyte and CD4+ T cell DR3 expression and serum TL1A levels Suppression of costimulation by human cytomegalovirus promotes evasion of cellular immune defenses. Age-dependent maintenance of motor control and corticostriatal innervation by death receptor 3. Characterization of a highly glycosylated form of the human cytomegalovirus HLA class I homologue gpUL18

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