About: H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection

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About: H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection Goto Sponge NotDistinct Permalink

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scientific article published on 01 November 1992

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в листопаді 1992 (uk) im November 1992 veröffentlichter wissenschaftlicher Artikel (de) scientific article published on 01 November 1992 (en) artículu científicu espublizáu en payares de 1992 (ast)
publication date	wds:Q67577416-32B2FC33-0CA2-4530-926E-5E739C6D3327
publication date	1992-11-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q67577416-86D06F85-0462-4519-80B0-8D9EB185F3B3
language of work or name	English
author name string	wds:Q67577416-8274B9A8-A0F4-4268-B435-732AEE6B3C98 wds:Q67577416-A447E2FB-59EC-4CBB-8500-05E173C02659
author name string	D Plaksin C Gelber
rdfs:label	H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection (en) H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection (nl)
skos:prefLabel	H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection (en) H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection (nl)
name	H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection (en) H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection (nl)
author	wds:Q67577416-52CDF5B7-A5FD-494B-8A74-A3482385698B
author	Lea Eisenbach
title	wds:Q67577416-B647CD1C-4C8D-4A93-8BCD-EA7008C8F71A
title	H-2Db gene transfer into highly metastatic D122 cells results in tumor rejection in allogeneic recipients, but does not affect metastasis in syngeneic recipients. Implications for mechanisms of allorejection (en)
page(s)	wds:Q67577416-647BA4A5-1821-4180-B9D2-0F8A2553EFCF
page(s)	771-777
instance of	wds:Q67577416-AE759487-DC76-4ADD-87CB-57D4D9E49FAF
instance of	scholarly article
PubMed ID	wds:Q67577416-5784CCF2-CED9-4A63-89FA-57DE39B2E7B0
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/1428231
PubMed ID	1428231
published in	wds:Q67577416-96C3C384-D4FA-413E-90FE-FCE744409226
published in	International Journal of Cancer
issue	wds:Q67577416-B2337143-4359-4AFA-9AAE-8FD101C30C31
volume	wds:Q67577416-9E44AA79-A2FF-40EB-8BAD-FE9DB0A25227
issue	5
volume	52
number of pages	wds:Q67577416-60566356-89E4-4CD4-B466-8E5B811F9D51
number of pages	7 (xsd:decimal)
DOI	wds:Q67577416-4934C391-94BE-4422-B5DB-97406371503D
DOI	http://dx.doi.org/10.1002/IJC.2910520517
DOI	10.1002/IJC.2910520517
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q67577416
is cites work of	Tumor endothelial marker 1 (Tem1) functions in the growth and progression of abdominal tumors KBF1 (p50 NF-kappa B homodimer) acts as a repressor of H-2Kb gene expression in metastatic tumor cells CTL induction by a tumour-associated antigen octapeptide derived from a murine lung carcinoma A genetically modified allogeneic cellular vaccine generates MHC class I-restricted cytotoxic responses against tumor-associated antigens and protects against CNS tumors in vivo. Upregulation of HLA Class I Expression on Tumor Cells by the Anti-EGFR Antibody Nimotuzumab. Various stress stimuli rewire the profile of liver secretome in a p53-dependent manner.
is cites work of	wds:Q59069991-35D7E150-61A4-4AA7-94A7-A77B6A5E3155 wds:Q34480359-67C584B5-999D-4F64-B168-98E7D307E45E wds:Q43119856-4F0003A6-E0A5-41A6-A2FB-1F6343E2947E wds:Q48615846-01D8F6BE-1963-473F-A331-8FF76A363A58 wds:Q28269339-7EBB92DB-B6A5-464D-BE3E-F0289CDB0A10 wds:Q55364421-88744B9E-AE7D-491A-9FD2-9EB33663CAF0

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