About: P. Weydt

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

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rdf:type	Item
description	researcher (en) 연구원 (ko) 研究者 (ja) investigador (es) araştırmacı (tr) Neurologe (de) wetenschapper (nl) дослідник (uk) изилдөөчү (ky) 研究者 (zh)
rdfs:label	P. Weydt (en) P. Weydt (es) P. Weydt (nl) P. Weydt (ast) Patrick Weydt (de)
skos:prefLabel	P. Weydt (en) P. Weydt (es) P. Weydt (nl) P. Weydt (ast) Patrick Weydt (de)
name	P. Weydt (en) P. Weydt (es) P. Weydt (nl) P. Weydt (ast) Patrick Weydt (de)
Scopus author ID	wds:Q67390356-39B4E7B6-A24D-46DF-B661-4A57F40EEA9D
Scopus author ID	6603391824
given name	wds:Q67390356-92A5E118-A4AD-4349-9A0E-DF441B976F5A
given name	Patrick
instance of	wds:Q67390356-329D7FF6-30BC-43C2-8B50-8FB68CD29457
instance of	Homo sapiens
Semantic Scholar author ID	wds:Q67390356-087C6561-9DC5-4B97-82AE-2CB73364336B
Semantic Scholar author ID	4546596
skos:altLabel	Patrick Roman Philipp Weydt (en) Patrick Weydt (en) Patrick Weydt (nl)
occupation	wds:Q67390356-67946757-8092-4E63-B10B-450A68ADA990
employer	wds:Q67390356-D6B63090-9D05-4CDE-BFB1-47C91D121604
sex or gender	wds:Q67390356-C88C8603-05C6-450F-9614-84432C190258
VIAF ID	wds:Q67390356-A311E233-1850-47C8-91ED-11E8452B79D3
GND ID	wds:Q67390356-929CEAFD-907D-4F3E-A878-B55C00264172
VIAF ID	http://viaf.org/viaf/74727958
GND ID	https://d-nb.info/gnd/122170334
occupation	researcher
employer	University Hospital Bonn
sex or gender	male
VIAF ID	74727958
GND ID	122170334
ORCID iD	wds:Q67390356-CBDF8CB2-AD07-4C64-8673-92A515816783
ORCID iD	https://orcid.org/0000-0003-2344-5662
ORCID iD	0000-0003-2344-5662
ResearcherID	wds:Q67390356-2DB35A77-1D13-4020-A004-B67C8DE69D27
ResearcherID	F-5114-2015
is owl:sameAs of	P. Weydt
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q67390356
is author of	Serum microRNAs in patients with genetic amyotrophic lateral sclerosis and pre-manifest mutation carriers A greatly extended PPARGC1A genomic locus encodes several new brain-specific isoforms and influences Huntington disease age of onset Current pharmacological management of amyotrophic [corrected] lateral sclerosis and a role for rational polypharmacy PGC-1α is a male-specific disease modifier of human and experimental amyotrophic lateral sclerosis Thermoregulatory and metabolic defects in Huntington's disease transgenic mice implicate PGC-1alpha in Huntington's disease neurodegeneration Prodromal Huntington disease as a model for functional compensation of early neurodegeneration Medicinal cannabis: rational guidelines for dosing Survey of cannabis use in patients with amyotrophic lateral sclerosis Endocannabinoids accumulate in spinal cord of SOD1 G93A transgenic mice. Haploinsufficiency of TBK1 causes familial ALS and fronto-temporal dementia Can lesions to the motor cortex induce amyotrophic lateral sclerosis? The gene coding for PGC-1alpha modifies age at onset in Huntington's Disease Modafinil to treat fatigue in amyotrophic lateral sclerosis: an open label pilot study Targeting protein aggregation in neurodegeneration--lessons from polyglutamine disorders Neuroinflammation in the pathogenesis of amyotrophic lateral sclerosis Cannabinol delays symptom onset in SOD1 (G93A) transgenic mice without affecting survival. Targeting toxic proteins for turnover Poly-GP in cerebrospinal fluid links C9orf72-associated dipeptide repeat expression to the asymptomatic phase of ALS/FTD. Dynein mutations associated with hereditary motor neuropathies impair mitochondrial morphology and function with age. Mutual exacerbation of peroxisome proliferator-activated receptor γ coactivator 1α deregulation and α-synuclein oligomerization Neurofilaments in the diagnosis of motoneuron diseases: a prospective study on 455 patients Neurofilament levels as biomarkers in asymptomatic and symptomatic familial amyotrophic lateral sclerosis The role of microglial cells in amyotrophic lateral sclerosis Two-point magnitude MRI for rapid mapping of brown adipose tissue and its application to the R6/2 mouse model of Huntington disease Neuro-inflammation as a therapeutic target in amyotrophic lateral sclerosis. The concept and diagnostic criteria of primary lateral sclerosis Clinical Trials in Spinal and Bulbar Muscular Atrophy-Past, Present, and Future NEK1 mutations in familial amyotrophic lateral sclerosis. Comparative biochemical characterization of the monoacylglycerol lipase inhibitor KML29 in brain, spinal cord, liver, spleen, fat and muscle tissue. Full-length PGC-1α salvages the phenotype of a mouse model of human neuropathy through mitochondrial proliferation. Incidence and geographical variation of amyotrophic lateral sclerosis (ALS) in Southern Germany--completeness of the ALS registry Swabia A single nucleotide polymorphism in the coding region of PGC-1α is a male-specific modifier of Huntington disease age-at-onset in a large European cohort. Contrasting effects of selective MAGL and FAAH inhibition on dopamine depletion and GDNF expression in a chronic MPTP mouse model of Parkinson's disease. Peripheral monocytes are functionally altered and invade the CNS in ALS patients Serum microRNAs in sporadic amyotrophic lateral sclerosis. Multicenter evaluation of neurofilaments in early symptom onset amyotrophic lateral sclerosis. Towards a European Registry and Biorepository for Patients with Spinal and Bulbar Muscular Atrophy. The metabolic and endocrine characteristics in spinal and bulbar muscular atrophy. Different CSF protein profiles in amyotrophic lateral sclerosis and frontotemporal dementia with C9orf72 hexanucleotide repeat expansion Comparison of Sirtuin 3 Levels in ALS and Huntington's Disease-Differential Effects in Human Tissue Samples vs. Transgenic Mouse Models ALS-causing mutations differentially affect PGC-1α expression and function in the brain vs. peripheral tissues Novel Blood-Based Biomarkers of Cognition, Stress, and Physical or Cognitive Training in Older Adults at Risk of Dementia: Preliminary Evidence for a Role of BDNF, Irisin, and the Kynurenine Pathway. High-resolution respirometry of fine-needle muscle biopsies in pre-manifest Huntington's disease expansion mutation carriers shows normal mitochondrial respiratory function. Provision of assistive technology devices among people with ALS in Germany: a platform-case management approach.

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