About: c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion

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About: c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion Goto Sponge NotDistinct Permalink

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scientific article published on 01 February 2007

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в лютому 2007 (uk) article scientifique publié en 2007 (fr) im Februar 2007 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu en febreru de 2007 (ast) artikull shkencor i botuar më 01 shkurt 2007 (sq) scientific article published on 01 February 2007 (en)
publication date	wds:Q64377234-7E14F580-7C23-4DA1-A62B-7365B25B4F6D
publication date	2007-02-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q64377234-2D5F8C2B-1F76-41C1-8862-7EB16F4249AE
language of work or name	English
cites work	wds:Q64377234-D45239D5-87A0-4FB3-BC9D-216FF5844C15
cites work	COX-2 inhibitors suppress integrin alpha5 expression in human lung carcinoma cells through activation of Erk: involvement of Sp1 and AP-1 sites
author name string	wds:Q64377234-1D0E68D4-CC34-4B82-B063-98CDE33426CD wds:Q64377234-4FE2C4C9-9632-4C48-9CD0-714118D61ECE wds:Q64377234-529B3694-1421-44A2-816B-D07B71BF0467 wds:Q64377234-62107931-92F4-479D-BB3A-7C7F9544A820 wds:Q64377234-66EFF98A-E94B-4C64-99C5-04F3C451BE1A wds:Q64377234-85EAE80F-2F2A-4865-A156-E8AF4CE055CF wds:Q64377234-C5DFD6CB-8A17-48E2-BD6B-0ED97AE3BFC7 wds:Q64377234-C7A7E8CA-3817-46F9-A1ED-2A874A96721B wds:Q64377234-CDE9B7E1-21FE-4E43-BF32-E5F092767350 wds:Q64377234-E522FE83-9CA0-4A96-A394-EA3B1791C493 wds:Q64377234-F949B745-D2CE-444E-B03A-5716BFF49DFF
author name string	Ravi Salgia S Diane Yamada George F Vande Woude Nariyoshi Shinomiya Kenjiro Sawada Emily Kistner A Reza Radjabi Maria S Tretiakova Amy R Becker Hilary Kenny Muge A Turkyilmaz
rdfs:label	c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (en) c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (nl) c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (sq)
skos:prefLabel	c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (en) c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (nl) c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (sq)
name	c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (en) c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (nl) c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (sq)
author	wds:Q64377234-74CFF66E-6891-495A-8EDC-AF5E32DF1906
author	Ernst Lengyel
title	wds:Q64377234-DF705DB4-3ECA-48B9-9F85-91D854526C7A
title	c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion (en)
page(s)	wds:Q64377234-F4762ECE-E166-4DFF-9CF2-A6ABA2AC8AFE
page(s)	1670-1679
instance of	wds:Q64377234-5ACC1279-4C57-485F-BF1F-539246606430
instance of	scholarly article
main subject	wds:Q64377234-F0620649-7527-4E69-8F22-57DE60495E98
main subject	ovarian neoplasm
PubMed ID	wds:Q64377234-031764BE-8B5A-40F4-8888-6A9E12B91AE4
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/17308108
PubMed ID	17308108
published in	wds:Q64377234-EABB2FFC-1DC2-4353-AF87-64C76297153E
published in	Cancer Research
issue	wds:Q64377234-438750EB-6A95-42D0-BAF2-D4303D8452DB
volume	wds:Q64377234-44CB4235-0F36-4D61-A808-6F35486116F4
issue	4
volume	67
DOI	wds:Q64377234-A4F2912A-CA60-4E1F-8AA7-0F63099750D5
DOI	http://dx.doi.org/10.1158/0008-5472.CAN-06-1147
DOI	10.1158/0008-5472.CAN-06-1147
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q64377234
is cites work of	Ligand-independent activation of c-Met by fibronectin and α(5)β(1)-integrin regulates ovarian cancer invasion and metastasis MET: a promising anticancer therapeutic target Suppression of tumor invasion and metastasis by concurrent inhibition of c-Met and VEGF signaling in pancreatic neuroendocrine tumors Dual MET-EGFR combinatorial inhibition against T790M-EGFR-mediated erlotinib-resistant lung cancer. Let-7 expression defines two differentiation stages of cancer The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2 Prognostic significance of overexpression of c-Met oncoprotein in cholangiocarcinoma p70 S6 kinase drives ovarian cancer metastasis through multicellular spheroid-peritoneum interaction and P-cadherin/b1 integrin signaling activation. P-cadherin cooperates with insulin-like growth factor-1 receptor to promote metastatic signaling of gonadotropin-releasing hormone in ovarian cancer via p120 catenin Induction of a tumor-metastasis-receptive microenvironment as an unwanted and underestimated side effect of treatment by chemotherapy or radiotherapy Wild-type p53 controls cell motility and invasion by dual regulation of MET expression An orally available small-molecule inhibitor of c-Met, PF-2341066, reduces tumor burden and metastasis in a preclinical model of ovarian cancer metastasis c-Met overexpression contributes to the acquired apoptotic resistance of nonadherent ovarian cancer cells through a cross talk mediated by phosphatidylinositol 3-kinase and extracellular signal-regulated kinase 1/2. Gene amplification and protein overexpression of MET are common events in ovarian clear-cell adenocarcinoma: their roles in tumor progression and prognostication of the patient Tolfenamic acid inhibits ovarian cancer cell growth and decreases the expression of c-Met and survivin through suppressing specificity protein transcription factors. Expression and significance of CD44, CD47 and c-met in ovarian clear cell carcinoma A novel c-Met inhibitor, MK8033, synergizes with carboplatin plus paclitaxel to inhibit ovarian cancer cell growth c-MET as a Potential Therapeutic Target in Ovarian Clear Cell Carcinoma Small molecule inhibitor of c-Met (PHA665752) suppresses the growth of ovarian cancer cells and reverses cisplatin resistance HGF/c-Met pathway has a prominent role in mediating antiapoptotic signals through AKT in epithelial ovarian carcinoma. Ovarian cancer development and metastasis Thrombin induces tumor invasion through the induction and association of matrix metalloproteinase-9 and beta1-integrin on the cell surface c-Met inhibitor synergizes with tumor necrosis factor-related apoptosis-induced ligand to induce papillary thyroid carcinoma cell death Expression and mutational analysis of MET in human solid cancers The initial steps of ovarian cancer cell metastasis are mediated by MMP-2 cleavage of vitronectin and fibronectin. Downregulation of miRNA-31 induces taxane resistance in ovarian cancer cells through increase of receptor tyrosine kinase MET. Predictive and prognostic protein biomarkers in epithelial ovarian cancer: recommendation for future studies Let-7 modulates acquired resistance of ovarian cancer to Taxanes via IMP-1-mediated stabilization of multidrug resistance 1 Pathophysiology of colorectal peritoneal carcinomatosis: Role of the peritoneum Melanoma proteoglycan modifies gene expression to stimulate tumor cell motility, growth, and epithelial-to-mesenchymal transition Frequent downregulation of miR-34 family in human ovarian cancers Assessment of hepatocyte growth factor in ovarian cancer mortality

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