About: Jens Mogensen

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) researcher (en) 연구원 (ko) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy)
rdfs:label	Jens Mogensen (en) Jens Mogensen (es) Jens Mogensen (it) Jens Mogensen (nl) Jens Mogensen (ast)
skos:prefLabel	Jens Mogensen (en) Jens Mogensen (es) Jens Mogensen (it) Jens Mogensen (nl) Jens Mogensen (ast)
name	Jens Mogensen (en) Jens Mogensen (es) Jens Mogensen (it) Jens Mogensen (nl) Jens Mogensen (ast)
Scopus author ID	wds:Q63806848-98F66DE5-866E-4E3E-A240-E06EB8585F89
Scopus author ID	7006575943
given name	wds:Q63806848-1F29B252-7DDF-495F-96D0-0329F44AF3D0
given name	Jens
instance of	wds:Q63806848-55534C79-8FAA-44A4-AA5D-C9514B19B452
instance of	Homo sapiens
occupation	wds:Q63806848-D2AC7E30-969D-40E8-9433-9379DEB24933
employer	wds:Q63806848-B7CFEE06-A23E-41D4-A3F3-9AA99AE55364
sex or gender	wds:Q63806848-4D5BD1E5-E3AA-442A-92E1-D6B181DAEF26
VIAF ID	wds:Q63806848-b527e2a1-4f24-4f0b-8347-ad6363b6ce3c
GND ID	wds:Q63806848-d3d4caf4-debb-4a9e-8409-c61584c3bb28
official website	wds:Q63806848-82F09BF6-1AFE-47C1-A144-35422801641B
VIAF ID	http://viaf.org/viaf/307259526
GND ID	https://d-nb.info/gnd/1211606333
occupation	researcher
employer	Odense University
sex or gender	male
VIAF ID	307259526
GND ID	1211606333
official website	https://portal.findresearcher.sdu.dk/en/persons/6dff3b3a-abb9-4624-a24d-f713b09c0e7b
ORCID iD	wds:Q63806848-E3D850FC-90DF-4354-A061-31489D29E8EE
ORCID iD	https://orcid.org/0000-0002-8813-573X
ORCID iD	0000-0002-8813-573X
Padua Research Archive author ID	wds:Q63806848-C72EE839-3536-476A-AF3E-7AB041180226
Padua Research Archive author ID	32048
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q63806848
is author of	Left ventricular outflow tract obstruction and sudden death risk in patients with hypertrophic cardiomyopathy. Prevalence, Clinical Significance, and Genetic Basis of Hypertrophic Cardiomyopathy With Restrictive Phenotype Restrictive cardiomyopathy Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathy Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype Current state of knowledge on aetiology, diagnosis, management, and therapy of myocarditis: a position statement of the European Society of Cardiology Working Group on Myocardial and Pericardial Diseases. Mutated desmoglein-2 proteins are incorporated into desmosomes and exhibit dominant-negative effects in arrhythmogenic right ventricular cardiomyopathy The LMNA mutation p.Arg321Ter associated with dilated cardiomyopathy leads to reduced expression and a skewed ratio of lamin A and lamin C proteins 2014 ESC Guidelines on diagnosis and management of hypertrophic cardiomyopathy: the Task Force for the Diagnosis and Management of Hypertrophic Cardiomyopathy of the European Society of Cardiology (ESC). Triage strategy for urgent management of cardiac tamponade: a position statement of the European Society of Cardiology Working Group on Myocardial and Pericardial Diseases Risk Factors for Malignant Ventricular Arrhythmias in Lamin A/C Mutation Carriers Atlas of the clinical genetics of human dilated cardiomyopathy Frequency and clinical expression of cardiac troponin I mutations in 748 consecutive families with hypertrophic cardiomyopathy. Long-term outcomes in hypertrophic cardiomyopathy caused by mutations in the cardiac troponin T gene. An improved method for chromosome-specific labeling of alpha satellite DNA in situ by using denatured double-stranded DNA probes as primers in a primed in situ labeling (PRINS) procedure. Fast one-step procedure for the detection of nucleic acids in situ by primer-induced sequence-specific labeling with fluorescein-12-dUTP Gender-specific differences in major cardiac events and mortality in lamin A/C mutation carriers. Functional effects of the DCM mutant Gly159Asp Troponin C in skinned muscle fibres A mutation in the glutamate-rich region of RNA-binding motif protein 20 causes dilated cardiomyopathy through missplicing of titin and impaired Frank-Starling mechanism. Truncating Plakophilin-2 Mutations in Arrhythmogenic Cardiomyopathy Are Associated With Protein Haploinsufficiency in Both Myocardium and Epidermis Prevalence and clinical significance of systolic impairment in hypertrophic cardiomyopathy Wytyczne ESC dotyczące rozpoznawania i postępowania w kardiomiopatii przerostowej w 2014 roku The current role of next-generation DNA sequencing in routine care of patients with hereditary cardiovascular conditions: a viewpoint paper of the European Society of Cardiology working group on myocardial and pericardial diseases and members of the Protein expression studies of desmoplakin mutations in cardiomyopathy patients reveal different molecular disease mechanisms Quantitative Expression of the Mutated Lamin A/C Gene in Patients With Cardiolaminopathy A Systematic Review of Phenotypic Features Associated With Cardiac Troponin I Mutations in Hereditary Cardiomyopathies. Current approaches to unravel the mystery of dilated cardiomyopathy, a common cause of hereditary heart failure The clinical outcome of LMNA missense mutations can be associated with the amount of mutated protein in the nuclear envelope The TMEM43 Newfoundland mutation p.S358L causing ARVC-5 was imported from Europe and increases the stiffness of the cell nucleus Pathogenic RBM20-Variants Are Associated With a Severe Disease Expression in Male Patients With Dilated Cardiomyopathy An International External Validation Study of the 2014 European Society of Cardiology Guideline on Sudden Cardiac Death Prevention in Hypertrophic Cardiomyopathy (Evidence from HCM). DOK7 congenital myasthenia may be associated with severe mitral valve insufficiency Nonradioactive, sequence-specific detection of RNA in situ by primed in situ labeling (PRINS) Formin Homology 2 Domain Containing 3 (FHOD3) Is a Genetic Basis for Hypertrophic Cardiomyopathy Role of genotyping in risk factor assessment for sudden death in hypertrophic cardiomyopathy Left ventricular longitudinal systolic function after alcohol septal ablation for hypertrophic obstructive cardiomyopathy: a long-term follow-up study focused on speckle tracking echocardiography. Troponin mutations in cardiomyopathies. Hypertrophic cardiomyopathy-the clinical challenge of managing a hereditary heart condition Development of a Novel Risk Prediction Model for Sudden Cardiac Death in Childhood Hypertrophic Cardiomyopathy (HCM Risk-Kids)
is author of	wds:Q81529484-881BD763-9E38-4CE0-BE56-15E20E3EE752 wds:Q24314590-2135196D-8AE7-4C0C-97E5-2CADA74CA30C wds:Q48546767-3121D7F6-C73D-4CE6-A0E7-622AB5086FCE wds:Q58046364-AB4BDDEC-CA4A-407C-BA6B-1EC9109E003A wds:Q56877622-DB63964B-D98B-4DA2-BB53-795C06FF1CEC wds:Q58455949-90648CB2-B2E4-44A7-8893-49732230BC63 wds:Q64449715-6ECB21A4-96A0-4ACA-B954-3F53001BED83 wds:Q68120040-F849AA74-AD6B-412F-B083-E2218FD50E55

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