About: Jean-Luc Puel

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researcher

Attributes	Values
rdf:type	Item
description	researcher (en) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy)
rdfs:label	Jean-Luc Puel (en) Jean-Luc Puel (es) Jean-Luc Puel (nl) Jean-Luc Puel (ast)
skos:prefLabel	Jean-Luc Puel (en) Jean-Luc Puel (es) Jean-Luc Puel (nl) Jean-Luc Puel (ast)
name	Jean-Luc Puel (en) Jean-Luc Puel (es) Jean-Luc Puel (nl) Jean-Luc Puel (ast)
given name	wds:Q63442217-458993D6-EED3-4419-A8D9-7DF084A8EF83
given name	Jean-Luc
instance of	wds:Q63442217-97F97A5E-E34C-48C9-86B0-C6CDF6BC236E
instance of	Homo sapiens
doctoral student	wds:Q63442217-38AD97E3-0755-41B5-A26E-73AFE24C4164 wds:Q63442217-7FD9B703-E234-4474-AB7F-4D353FAFD94D
doctoral student	Régis Nouvian Matthieu Guitton
occupation	wds:Q63442217-F26EF3EA-5E83-4E7F-A057-CD4E558A1D52
employer	wds:Q63442217-1360529B-17F3-4BEF-85EF-5DA7E17E303F wds:Q63442217-66FB058A-2144-48EB-BEB9-81721F9FABEF wds:Q63442217-75CB9F01-C097-4DAD-99A2-60E29035AA18 wds:Q63442217-A2518FC2-794D-4186-B4CC-9DCE0FA44E1A wds:Q63442217-E63D56CF-E7B5-40BA-9DE3-35F2CCF8C80D wds:Q63442217-E66B8FC8-8196-4AD9-82D2-AA528E9E83A2
sex or gender	wds:Q63442217-C5B18156-7EE8-4039-8BC7-8C97AE7D332E
ISNI	wds:Q63442217-C20FF5D9-3A10-41A9-A171-697770709E9D
VIAF ID	wds:Q63442217-734DE2E9-59D5-4DDC-A3FC-53380B43D7C0
IdRef ID	wds:Q63442217-925BCE08-9A1C-4644-BD36-886C8F2C9A4B
date of birth	wds:Q63442217-863E93A0-31D5-4743-8B6A-432D948BAD31
VIAF ID	http://viaf.org/viaf/189529056
IdRef ID	http://www.idref.fr/075000008/id
occupation	researcher
employer	University of Montpellier Louisiana State University Institute of Human Genetics Délégation Languedoc Roussillon
sex or gender	male
ISNI	0000000356553715
VIAF ID	189529056
IdRef ID	075000008
date of birth	1958-01-01 00:00:00Z (xsd:dateTime)
ORCID iD	wds:Q63442217-371FD88D-E8D4-40AF-B610-E145A23C63C2
ORCID iD	https://orcid.org/0000-0002-3772-5190
ORCID iD	0000-0002-3772-5190
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q63442217
is author of	AMPA-preferring glutamate receptors in cochlear physiology of adult guinea-pig. Control of hair cell excitability by vestibular primary sensory neurons Inhibition of the c-Jun N-terminal kinase-mediated mitochondrial cell death pathway restores auditory function in sound-exposed animals. Deafness and cochlear fibrocyte alterations in mice deficient for the inner ear protein otospiralin. Degeneration of sensory outer hair cells following pharmacological blockade of cochlear KCNQ channels in the adult guinea pig. A peptide inhibitor of c-Jun N-terminal kinase protects against both aminoglycoside and acoustic trauma-induced auditory hair cell death and hearing loss. Tmprss3, a transmembrane serine protease deficient in human DFNB8/10 deafness, is critical for cochlear hair cell survival at the onset of hearing m-Chlorophenylpiperazine exacerbates perception of salicylate-induced tinnitus in rats. Blocking c-Jun-N-terminal kinase signaling can prevent hearing loss induced by both electrode insertion trauma and neomycin ototoxicity. FXYD6 is a novel regulator of Na,K-ATPase expressed in the inner ear. A novel dual inhibitor of calpains and lipid peroxidation (BN82270) rescues the cochlea from sound trauma. Development of 2f1-f2 otoacoustic emissions in the rat Excitotoxicity and repair of cochlear synapses after noise-trauma induced hearing loss. Excitatory amino acid antagonists protect cochlear auditory neurons from excitotoxicity. Macrophage contribution to the response of the rat organ of Corti to amikacin. Caspase inhibitors, but not c-Jun NH2-terminal kinase inhibitor treatment, prevent cisplatin-induced hearing loss. Correlative development of cochlear action potential sensitivity, latency, and frequency selectivity. New pharmacological strategies to restore hearing and treat tinnitus. Synaptic repair mechanisms responsible for functional recovery in various cochlear pathologies. Synaptic regeneration and functional recovery after excitotoxic injury in the guinea pig cochlea Impairment of SLC17A8 encoding vesicular glutamate transporter-3, VGLUT3, underlies nonsyndromic deafness DFNA25 and inner hair cell dysfunction in null mice. Effect of contralateral sound stimulation on the distortion product 2F1-F2: evidence that the medial efferent system is involved. Synaptic connections and putative functions of the dopaminergic innervation of the guinea pig cochlea. Effects of a dopaminergic agonist in the guinea pig cochlea Knockout of the ASIC2 channel in mice does not impair cutaneous mechanosensation, visceral mechanonociception and hearing Transient Ca2+-permeable AMPA receptors in postnatal rat primary auditory neurons. Oxidative stress, inflammation, and autophagic stress as the key mechanisms of premature age-related hearing loss in SAMP8 mouse Cochlea. Glutamate transporters in the guinea-pig cochlea: partial mRNA sequences, cellular expression and functional implications. Salicylate induces tinnitus through activation of cochlear NMDA receptors Selective attention modifies the active micromechanical properties of the cochlea OPA1 R445H mutation in optic atrophy associated with sensorineural deafness. Contribution of auditory nerve fibers to compound action potential of the auditory nerve Salicylate enables cochlear arachidonic-acid-sensitive NMDA receptor responses. Dopamine inhibition of auditory nerve activity in the adult mammalian cochlea. Characteristics of tinnitus and etiology of associated hearing loss: a study of 123 patients. Salicylate-induced tinnitus: molecular mechanisms and modulation by anxiety. Cochlear NMDA receptor blockade prevents salicylate-induced tinnitus. Electrophysiological evidence for the presence of NMDA receptors in the guinea pig cochlea Protein kinase C may be involved in synaptic repair of auditory neuron dendrites after AMPA injury in the cochlea. Cochlear origin of 2f1-f2 distortion products assessed by using 2 types of mutant mice The human OPA1delTTAG mutation induces premature age-related systemic neurodegeneration in mouse. Efficient and specific transduction of cochlear supporting cells by adeno-associated virus serotype 5. Clinical and angiographic outcome of paclitaxel-eluting stent implantation for unprotected left main coronary artery bifurcation narrowing.

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