About: David Michael Aronoff

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About: David Michael Aronoff Goto Sponge NotDistinct Permalink

An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

physician and infectious disease researcher

Attributes	Values
rdf:type	Item
field of work	wds:Q61108096-10f99255-4f03-e2ef-c623-86aeaa9948b2 wds:Q61108096-28e8ae98-4e94-f1b9-b2f5-8b2cfea4de6f wds:Q61108096-2b1ab7db-4e58-c619-562d-f12d8477e15b wds:Q61108096-4d27e5dd-481d-226b-1859-93de17f54777 wds:Q61108096-750d97b5-42de-3893-20c0-112ca90f6d29 wds:Q61108096-ac82f13e-4a03-7116-5c30-2ca441946e25 wds:Q61108096-bb0ea9d0-46c2-4695-48f4-25afe071c592 wds:Q61108096-e735152b-4a6e-3cfb-1948-e0f7c9540004
field of work	Clostridium maternal health human gut flora Streptococcus bacterial infectious disease Clostridium difficile innate immune response reproductive tract infection
description	wetenschapper (nl) дослідник (uk) physician and infectious disease researcher (en)
rdfs:label	David M Aronoff (nl) David M Aronoff (ast) David Michael Aronoff (en) David Michael Aronoff (sq)
skos:prefLabel	David M Aronoff (nl) David M Aronoff (ast) David Michael Aronoff (en) David Michael Aronoff (sq)
name	David M Aronoff (nl) David M Aronoff (ast) David Michael Aronoff (en) David Michael Aronoff (sq)
affiliation	wds:Q61108096-9a766baf-42d3-e9de-74da-262aee8392cb wds:Q61108096-FAA476A5-BE1A-47A0-9968-67EC04C44871 wds:Q61108096-b213bb23-4dbe-faa5-be0d-5dd25da443fb
affiliation	Vanderbilt University School of Medicine Vanderbilt Department of Medicine Vanderbilt Institute for Infection, Immunology, and Inflammation
given name	wds:Q61108096-B35107C0-53E5-461A-A534-6168C42B3BDA
given name	David
instance of	wds:Q61108096-4F2C8620-9EE3-425B-9AEF-3BE276FB5805
instance of	Homo sapiens
skos:altLabel	DM Aronoff (en) David Aronoff (en)
occupation	wds:Q61108096-1b910116-41c9-be34-a7f4-697c2288e4fb wds:Q61108096-d1ac6cd8-4c5c-3bf9-00cd-9a8435e38ee4 wds:Q61108096-e7fda52b-4675-f3dd-0f99-5b11fafb865c
employer	wds:Q61108096-4E43F39E-2EF0-41E7-A8F8-3170A398422F wds:Q61108096-C6B76A9F-299A-4CED-8B39-7EB001906B83
sex or gender	wds:Q61108096-BB8F65E6-7637-4290-B813-B73C779BD861
on focus list of Wikimedia project	wds:Q61108096-DC885F13-28B2-4A00-B817-B09167232FE5
educated at	wds:Q61108096-a6dbdd1f-4f0b-5aca-37e9-1529d0c27f78 wds:Q61108096-c2d5e51f-478b-8e3e-cbb8-089ea8d70f6a
family name	wds:Q61108096-884e3d26-4c64-61ca-b8d0-eff3eb3a5985
occupation	university teacher physician medical researcher
employer	Indiana University School of Medicine Vanderbilt University
sex or gender	male
on focus list of Wikimedia project	WikiProject COVID-19
educated at	Tufts University Indiana University
family name	Aronoff
ORCID iD	wds:Q61108096-215F591D-26C6-4696-B675-0DF1C8742C00
ORCID iD	https://orcid.org/0000-0003-4587-6121
ORCID iD	0000-0003-4587-6121
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q61108096
is author of	Clostridium difficile ribotype does not predict severe infection Aspirin and Reye's syndrome: discovery of aspirin and paracetamol Fever and hypothermia in systemic inflammation: recent discoveries and revisions. The influence of non-steroidal anti-inflammatory drugs on the gut microbiome Identification of toxemia in patients with Clostridium difficile infection. Intrauterine group A streptococcal infections are exacerbated by prostaglandin E2. Comparative analysis of the extracellular proteomes of two Clostridium sordellii strains exhibiting contrasting virulence Misoprostol impairs female reproductive tract innate immunity against Clostridium sordellii Clostridium sordellii toxic shock syndrome Storage duration of red blood cell transfusion and Clostridium difficile infection: a within person comparison Leukotriene B4 enhances innate immune defense against the puerperal sepsis agent Streptococcus pyogenes. Postoperative burden of hospital-acquired Clostridium difficile infection. Cardiobacterium hominis endocarditis: Two cases and a review of the literature Leukotrienes enhance the bactericidal activity of alveolar macrophages against Klebsiella pneumoniae through the activation of NADPH oxidase Cyclooxygenase inhibition in sepsis: is there life after death? E-prostanoid 3 receptor deletion improves pulmonary host defense and protects mice from death in severe Streptococcus pneumoniae infection Prostaglandin E2 inhibits alveolar macrophage phagocytosis through an E-prostanoid 2 receptor-mediated increase in intracellular cyclic AMP. Synthetic Prostacyclin Analogs Differentially Regulate Macrophage Function via Distinct Analog-Receptor Binding Specificities Effects of prostaglandin E2 on the subcellular localization of Epac-1 and Rap1 proteins during Fcgamma-receptor-mediated phagocytosis in alveolar macrophages New insights into the mechanism of action of acetaminophen: Its clinical pharmacologic characteristics reflect its inhibition of the two prostaglandin H2 synthases Stimulatory Effects of Peroxisome Proliferator-Activated Receptor-gamma on Fcgamma Receptor-Mediated Phagocytosis by Alveolar Macrophages Short communication: differences between macrophages and dendritic cells in the cyclic AMP-dependent regulation of lipopolysaccharide-induced cytokine and chemokine synthesis. Determinants of the cellular specificity of acetaminophen as an inhibitor of prostaglandin H(2) synthases. Prostaglandin E(2) inhibits fibroblast migration by E-prostanoid 2 receptor-mediated increase in PTEN activity 11,12-epoxyeicosatrienoic acid attenuates synthesis of prostaglandin E2 in rat monocytes stimulated with lipopolysaccharide A clinical and epidemiological review of non-toxigenic Clostridium difficile. Activation of phosphatase and tensin homolog on chromosome 10 mediates the inhibition of FcgammaR phagocytosis by prostaglandin E2 in alveolar macrophages Specific leukotriene receptors couple to distinct G proteins to effect stimulation of alveolar macrophage host defense functions. Phosphorylation by protein kinase a inhibits nuclear import of 5-lipoxygenase. Blunt trauma as a risk factor for group A streptococcal necrotizing fasciitis Assessing the relationship between the use of nonsteroidal antiinflammatory drugs and necrotizing fasciitis caused by group A streptococcus Cyclic AMP: master regulator of innate immune cell function.

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