About: Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide

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About: Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide Goto Sponge NotDistinct Permalink

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scientific article published on 01 February 2002

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) 2002 թվականին հրատարակված գիտական հոդված (hy) наукова стаття, опублікована в лютому 2002 (uk) im Februar 2002 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu en febreru de 2002 (ast) artikull shkencor i botuar më 01 shkurt 2002 (sq) scientific article published on 01 February 2002 (en)
publication date	wds:Q60489559-D7DEEC68-46E9-43FA-9692-2B3E5038B297
publication date	2002-02-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q60489559-C8B9AC35-30EC-41D8-8AE6-2BD11ACC1808
language of work or name	English
author name string	wds:Q60489559-08542257-EC40-472C-8820-B08003C46CBD wds:Q60489559-48E69B47-7905-4C47-8B63-F55857ADC4AF wds:Q60489559-657E8964-0FED-40D0-B326-FB4EBDC6EAF0 wds:Q60489559-796850F2-3AFD-4582-A00F-D43FC94D3C69
author name string	Kazuyoshi Takeda Konstantinos Kyparissoudis Janice M Kelly Nadine Y Crowe
rdfs:label	Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (en) Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (nl) Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (sq)
skos:prefLabel	Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (en) Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (nl) Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (sq)
name	Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (en) Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (nl) Sequential production of interferon-gamma by NK1.1+ T cells and natural killer cells is essential for the antimetastatic effect of alpha -galactosylceramide (sq)
author	wds:Q60489559-0A08EE21-C359-41E5-A364-B7DBB469FBC8 wds:Q60489559-6AC4C439-30E8-4841-AE06-F81BBC4C7B8B wds:Q60489559-6D397D21-730A-492A-A3B9-940EB0079C0B wds:Q60489559-D907AE03-CB00-4120-897E-A40B2BE82240
author	Mark J. Smyth Hideo Yagita Dale I Godfrey Daniel G Pellicci
title	wds:Q60489559-7891EF34-1668-414B-B2F9-A7D3441C6F73
title	Sequential production of interferon-gamma by NK1.1(+) T cells and natural killer cells is essential for the antimetastatic effect of alpha-galactosylceramide (en)
page(s)	wds:Q60489559-67D8330E-FC97-4A62-A20C-801B30CE43EA
page(s)	1259-1266
instance of	wds:Q60489559-EEE591B7-5D11-4BAB-A778-EEBDC6FF30CA
instance of	scholarly article
PubMed ID	wds:Q60489559-52F6CF0E-D997-4691-A81D-E77395C63206
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/11830474
PubMed ID	11830474
published in	wds:Q60489559-6B5D3491-8894-4F68-82BF-6EBB4CEF0C98
published in	Blood
issue	wds:Q60489559-1850B85A-EA42-41BA-82AF-2CDEA6F386A1
volume	wds:Q60489559-4C643C75-66FC-486D-8AF4-D17EBACEBAC1
issue	4
volume	99
number of pages	wds:Q60489559-9872E69F-20AE-4867-BC36-970F72F6983F
number of pages	8 (xsd:decimal)
DOI	wds:Q60489559-5EC7B3A9-3E20-45C8-B52F-014F32872450
DOI	http://dx.doi.org/10.1182/BLOOD.V99.4.1259
DOI	10.1182/BLOOD.V99.4.1259
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q60489559
is cites work of	Presentation of alpha-galactosylceramide by murine CD1d to natural killer T cells is facilitated by plasma membrane glycolipid rafts NKT cell-dependent leukemia eradication following stem cell mobilization with potent G-CSF analogs. The role of innate immunity in HBV infection Widespread natural variation in murine natural killer T-cell number and function Prolonged IFN-gamma-producing NKT response induced with alpha-galactosylceramide-loaded DCs. Sequential activation of NKT cells and NK cells provides effective innate immunotherapy of cancer New aspects of natural-killer-cell surveillance and therapy of cancer Alpha-galactosylceramide (KRN7000) suppression of chemical- and oncogene-dependent carcinogenesis Antitumor Responses of Invariant Natural Killer T Cells Antigen-specific cytotoxicity by invariant NKT cells in vivo is CD95/CD178-dependent and is correlated with antigenic potency Natural killer T cells infiltrate neuroblastomas expressing the chemokine CCL2 NKG2D performs two functions in invariant NKT cells: direct TCR-independent activation of NK-like cytolysis and co-stimulation of activation by CD1d Valpha24-invariant NKT cells mediate antitumor activity via killing of tumor-associated macrophages The contrasting roles of NKT cells in tumor immunity Virus-specific CD8+ T cells in the liver: armed and ready to kill The bovine CD1D gene has an unusual gene structure and is expressed but cannot present α-galactosylceramide with a C26 fatty acid Molecular basis of lipid antigen presentation by CD1d and recognition by natural killer T cells Sustained expansion of NKT cells and antigen-specific T cells after injection of alpha-galactosyl-ceramide loaded mature dendritic cells in cancer patients Therapeutic activation of Vα24+Vβ11+ NKT cells in human subjects results in highly coordinated secondary activation of acquired and innate immunity Autoreactive natural killer T cells: promoting immune protection and immune tolerance through varied interactions with myeloid antigen-presenting cells NK cell-based immunotherapy for malignant diseases Transcriptional regulator Bhlhe40 works as a cofactor of T-bet in the regulation of IFN-γ production in iNKT cells Structural and Functional Characterization of a Novel Nonglycosidic Type I NKT Agonist with Immunomodulatory Properties Gene Therapy against Murine Melanoma B16F10-Nex2 Using IL-13Ralpha2-Fc Chimera and Interleukin 12 in Association with a Cyclopalladated Drug To be or not to be NKT: natural killer T cells in the liver. Activated NKT cells increase dendritic cell migration and enhance CD8+ T cell responses in the skin Therapeutic effect of repeated natural killer T cell stimulation in mouse cholangitis complicated by colitis The role of NKT cells in tumor immunity NKT cell networks in the regulation of tumor immunity PD-1/PD-L blockade prevents anergy induction and enhances the anti-tumor activities of glycolipid-activated invariant NKT cells iNKT cells suppress the CD8+ T cell response to a murine Burkitt's-like B cell lymphoma Mouse and human iNKT cell agonist β-mannosylceramide reveals a distinct mechanism of tumor immunity Differential antitumor immunity mediated by NKT cell subsets in vivo Going both ways: immune regulation via CD1d-dependent NKT cells A critical role for natural killer T cells in immunosurveillance of methylcholanthrene-induced sarcomas Superior protection against malaria and melanoma metastases by a C-glycoside analogue of the natural killer T cell ligand alpha-Galactosylceramide β-mannosylceramide activates type I natural killer t cells to induce tumor immunity without inducing long-term functional anergy NKT cells as an ideal anti-tumor immunotherapeutic Tuberous sclerosis 1 promotes invariant NKT cell anergy and inhibits invariant NKT cell-mediated antitumor immunity IL-10-producing NKT10 cells are a distinct regulatory invariant NKT cell subset NK cell-like behavior of Valpha14i NK T cells during MCMV infection Activation of natural killer (NK) T cells during murine cytomegalovirus infection enhances the antiviral response mediated by NK cells

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