About: Impaired β-adrenergic responsiveness accentuates dysfunctional excitation-contraction coupling in an ovine model of tachypacing-induced heart failure

Facets (new session)
Description
Metadata
Settings
- owl:sameAs
- Inference Rule:

About: Impaired β-adrenergic responsiveness accentuates dysfunctional excitation-contraction coupling in an ovine model of tachypacing-induced heart failure Goto Sponge NotDistinct Permalink

An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

article by Sarah J Briston et al published 15 March 2011 in Journal of Physiology

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в березні 2011 (uk) im März 2011 veröffentlichter wissenschaftlicher Artikel (de) article by Sarah J Briston et al published 15 March 2011 in Journal of Physiology (en)
publication date	wds:Q60182009-CC40FD56-CF16-42BD-9748-0E87151B4585
publication date	2011-03-15 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q60182009-739CE654-E933-4A63-8FA2-E8E6B29FB688
language of work or name	English
cites work	wds:Q60182009-0558E196-74AE-457E-B586-5B9ADC1B6B8F wds:Q60182009-0790CBAE-83AD-4D18-ADAB-58364768F55F wds:Q60182009-085AF217-684C-494D-A75C-0DC90C0E69D1 wds:Q60182009-19554D9B-5E92-4205-B65A-4DBB396439DC wds:Q60182009-1A0C1791-F49E-44D3-B596-83C0AC17AC4A wds:Q60182009-1BFB8DEF-BCE8-4525-9394-6ACE0345AC08 wds:Q60182009-2014EBF1-4621-4963-A108-0CA7D506A428 wds:Q60182009-219D1069-65A3-49D9-848F-E2736A0FC969 wds:Q60182009-225D9EC5-A8A2-42CF-94C8-A8E3078757E7 wds:Q60182009-27532AA6-0A37-45B1-BDC4-E79C1BB1F641 wds:Q60182009-2787E9CE-737B-49E5-8A73-2C0291C197E5 wds:Q60182009-29F130F0-96A7-4A4E-8891-0C99B7F2EAFD wds:Q60182009-343931EB-0329-47D4-8202-BF2D2DB33476 wds:Q60182009-361EFBE8-6079-4E3D-8A26-DE7DF16D7B6D wds:Q60182009-36A506B1-2489-42A7-9DC0-585F992B1BAD wds:Q60182009-390F4B89-6CF1-4CD1-926E-372E5C173B36 wds:Q60182009-3B5BD931-47E0-4D08-B903-24ACD383D13A wds:Q60182009-3E18781C-FED6-4BA9-97DA-771F1DF8F5B1 wds:Q60182009-41DA2B6B-BC6E-4869-BD41-77BB5880A349 wds:Q60182009-422716F5-019B-438C-A664-70212114F113 wds:Q60182009-46CEEFC6-5A32-41B3-8D1F-334BAEF5F163 wds:Q60182009-4B95998C-46A9-45A4-89AD-73C1E6D83FBE wds:Q60182009-51CDEDC9-1C3D-4D4B-9048-5B8458CE79B3 wds:Q60182009-5246B18E-FF60-493E-BD6A-D2B4E654E259 wds:Q60182009-529C7A22-E93B-4CAA-81C3-8A98DAA4564F wds:Q60182009-52D7E90E-DDBC-4C64-B907-6EBB11702AB9 wds:Q60182009-53C20F3D-CDB9-4350-BF19-637A418D53B2 wds:Q60182009-58107364-D67E-4F19-BEA2-5B996FC0A3BE wds:Q60182009-5C6462A8-8A9E-4427-91D4-148B360A9EC4 wds:Q60182009-5EF2F8C8-08F2-4905-986D-4F751BD78E8B wds:Q60182009-5F5D7F8B-3FC8-4D1F-A494-7CD3AC414BC4 wds:Q60182009-60C4735E-3605-42E8-AA0E-31727CC4673B wds:Q60182009-61EFE5B2-CBDC-4235-A092-3191DA03BD35 wds:Q60182009-642E9F0E-078C-4304-A3D0-8983CDF91617 wds:Q60182009-64F5BA22-8CF1-4464-B3C0-DCC9B844CF69 wds:Q60182009-6932C265-7DF6-4362-9F8C-8D7697527A37 wds:Q60182009-6C95F7A4-9F82-4F4E-965F-249C7ED34809 wds:Q60182009-7010E3BB-6097-4EA5-AE38-1AD0EF056D0D wds:Q60182009-78A2062D-7452-4FD8-938F-13B4A28BE158 wds:Q60182009-81EFCFD1-9E05-4A27-9A2C-208BA5219EE9 wds:Q60182009-837C3C44-74B3-4575-85BF-1ECBC89A65DE wds:Q60182009-8D7B7EDC-21B6-4CB0-969A-48599E3253CB wds:Q60182009-9B637830-0EEE-48D6-9291-B4525767793B wds:Q60182009-A1646FFE-862A-4B3A-83C7-16AA771CC94D wds:Q60182009-A57971D8-B0FD-4C31-8BC1-396B637B6A71 wds:Q60182009-AF9A8D7B-CBE8-485B-A3D9-3522489C4B3D wds:Q60182009-AFE52615-6044-42FC-81FE-EF39D1E5E001 wds:Q60182009-B7D32B4E-FA4F-425D-8B02-7F48012A973A wds:Q60182009-B9764933-68B0-418F-A9F9-8211844AA1C1 wds:Q60182009-BF1FAAD2-954B-46FE-B6F5-FC6BAFA9B725 wds:Q60182009-C2D55BBB-E158-4F07-91C9-F1C29BE802B0 wds:Q60182009-C4647061-2CB9-4948-8126-0A1E0AA68970 wds:Q60182009-C5E79C9C-BFC5-451B-8CDA-0535A8566F43 wds:Q60182009-C70D6364-E44A-49C3-9147-BB0859659F82 wds:Q60182009-C823397A-416E-4266-A931-9E18509BE33C wds:Q60182009-C89009FE-8D18-4DA9-9A09-BF9FD51C232D wds:Q60182009-CA025D11-45E5-47D3-847F-48AFED26762F wds:Q60182009-CA2BC1B4-1A6A-4311-8F3F-19A7697CCC52 wds:Q60182009-D6D652AF-F53F-4116-8552-FCC5E7CB3808 wds:Q60182009-E0763C96-D3FA-4207-AF6A-2C80E25D2FE5 wds:Q60182009-E79DC245-8FBC-43AA-B72D-DE6BB99C2341 wds:Q60182009-E9A1E90E-DBA8-4385-88AE-BC1ACA293088 wds:Q60182009-EB407378-276D-44C1-BAC8-42B9A7AB3F87 wds:Q60182009-F2401957-962B-4DCC-B05E-6350D86A14C9 wds:Q60182009-F8DDA187-2C76-4643-93BA-54912C36E4A1 wds:Q60182009-FA5056A5-41F3-4109-8607-779FB9B33F3C wds:Q60182009-FAC78387-BF67-4C70-ADCA-8A6F18AEC3FE
cites work	The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure Lifetime Risk for Developing Congestive Heart Failure Mechanism of beta-adrenergic receptor desensitization in cardiac hypertrophy is increased beta-adrenergic receptor kinase Enhanced sarcolemmal Ca2+ efflux reduces sarcoplasmic reticulum Ca2+ content and systolic Ca2+ in cardiac hypertrophy. Modulation of CICR has no maintained effect on systolic Ca2+: simultaneous measurements of sarcoplasmic reticulum and sarcolemmal Ca2+ fluxes in rat ventricular myocytes. Defective beta-adrenergic receptor signaling precedes the development of dilated cardiomyopathy in transgenic mice with calsequestrin overexpression Decreased Catecholamine Sensitivity and β-Adrenergic-Receptor Density in Failing Human Hearts Alterations in cardiac adrenergic signaling and calcium cycling differentially affect the progression of cardiomyopathy Level of beta-adrenergic receptor kinase 1 inhibition determines degree of cardiac dysfunction after chronic pressure overload-induced heart failure Physiological induction of a beta-adrenergic receptor kinase inhibitor transgene preserves ss-adrenergic responsiveness in pressure-overload cardiac hypertrophy Impaired chronotropic and vasodilator reserves limit exercise capacity in patients with heart failure and a preserved ejection fraction Protein kinase A phosphorylation of the cardiac calcium release channel (ryanodine receptor) in normal and failing hearts. Role of phosphatases and response to isoproterenol Decreased sarcoplasmic reticulum calcium content is responsible for defective excitation-contraction coupling in canine heart failure Deficient production of cyclic AMP: pharmacologic evidence of an important cause of contractile dysfunction in patients with end-stage heart failure The sarcoplasmic reticulum and arrhythmogenic calcium release Ca2+ handling and sarcoplasmic reticulum Ca2+ content in isolated failing and nonfailing human myocardium. L-type Ca2+ channel density and regulation are altered in failing human ventricular myocytes and recover after support with mechanical assist devices Arrhythmogenesis and contractile dysfunction in heart failure: Roles of sodium-calcium exchange, inward rectifier potassium current, and residual beta-adrenergic responsiveness Repolarization abnormalities in cardiomyocytes of dogs with chronic heart failure: role of sustained inward current. Abnormal myocyte Ca2+ homeostasis in rabbits with pacing-induced heart failure Reduced synchrony of Ca2+ release with loss of T-tubules-a comparison to Ca2+ release in human failing cardiomyocytes Decreased protein and phosphorylation level of the protein phosphatase inhibitor-1 in failing human hearts Myocardial adeno-associated virus serotype 6-betaARKct gene therapy improves cardiac function and normalizes the neurohormonal axis in chronic heart failure Regulation of systolic [Ca2+]i and cellular Ca2+ flux balance in rat ventricular myocytes by SR Ca2+, L-type Ca2+ current and diastolic [Ca2+]i.

Faceted Search & Find service v1.16.117 as of May 05 2024

Alternative Linked Data Documents: ODE Content Formats:

RDF

ODATA

Microdata

About

OpenLink Virtuoso version 07.20.3239 as of May 5 2024, on Linux (x86_64-centos_6-linux-gnu), Single-Server Edition (378 GB total memory, 180 GB memory in use)
Data on this page belongs to its respective rights holders.
Virtuoso Faceted Browser Copyright © 2009-2025 OpenLink Software