About: Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity

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scientific article published in Nature

Attributes	Values
rdf:type	Item
description	artículu científicu (ast) wetenschappelijk artikel (nl) article publié dans la revue scientifique Nature (fr) scientific article published in Nature (en) im Juni 1974 veröffentlichter wissenschaftlicher Artikel (de) наукова стаття, опублікована в Nature в червні 1974 (uk)
publication date	wds:Q59074350-1E146C19-C3BF-474E-843C-CD68D119695B
publication date	1974-06-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q59074350-9F0FA7CE-6229-406F-9E0E-0A5EA1F25121
language of work or name	English
exact match	wds:Q59074350-35630690-5496-4FC5-866E-C51457D16B25
exact match	https://scigraph.springernature.com/pub.10.1038/249654a0
cites work	wds:Q59074350-287192AA-CE7A-4379-AD7A-94EB53BF1C34 wds:Q59074350-A1CAB112-A89E-4E0F-BBFF-A5A8C81F7A79 wds:Q59074350-B2EE67D3-8D9A-4893-B085-AE2C4EF4797B wds:Q59074350-D6239E6F-7FBA-42CC-BC99-57FA11FFAA3E wds:Q59074350-E01F4DC8-B254-45F3-8BFD-3CCBCEF7FC2C
cites work	Depression of the T Cell Phenomenon of Contact Sensitivity by T Cells from Unresponsive Mice Suppressor cells in normal immunisation as a basic homeostatic phenomenon Effects of cyclophosphamide on lymphoid tissues labelled with 5-iodo-2-deoxyuridine- 125 I and 51 Cr. Proportional increase in the theta carrying lymphocytes in peripheral lymphoid tissue following treatment with cyclophosphamide. IMMUNOLOGICAL UNRESPONSIVENESS IN ALLERGIC CONTACT DEMATITIS TO DINITROCHLOROBENZENE IN GUINEA PIGS
author name string	wds:Q59074350-E32B3109-2806-457B-879C-D9E6790B3340 wds:Q59074350-F655D950-A468-4CA1-8EF0-98A06A83618E
author name string	J L Turk L Polak
rdfs:label	Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity (en) Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity (nl)
skos:prefLabel	Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity (en) Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity (nl)
name	Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity (en) Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity (nl)
title	wds:Q59074350-251B5158-33E8-4D26-A554-446AC6B193B7
title	Reversal of immunological tolerance by cyclophosphamide through inhibition of suppressor cell activity (en)
page(s)	wds:Q59074350-526E50AD-4B73-4C47-B394-474788418245
page(s)	654-656
instance of	wds:Q59074350-C158B736-C386-43B8-BAE5-046A60B3A270
instance of	scholarly article
main subject	wds:Q59074350-003CE8FB-65A6-4CE8-A792-BB7D39F7F59C
main subject	immunology
PubMed ID	wds:Q59074350-ACACB1CE-D361-4CA9-98FB-EC8CBA4D441C
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/4275846
PubMed ID	4275846
published in	wds:Q59074350-982F5B86-4C3D-43AD-A9A5-F6D5AC2142DD
published in	Nature
issue	wds:Q59074350-1E89405A-BA81-4DC3-8E54-C95265CE897D
volume	wds:Q59074350-84E7F9DC-FBDA-436C-9AA3-12004024C4C1
issue	458
volume	249
DOI	wds:Q59074350-915A71EB-3CF2-4979-B453-1E32DB2B74D4
DOI	http://dx.doi.org/10.1038/249654A0
DOI	10.1038/249654A0
Dimensions Publication ID	wds:Q59074350-7F924B78-0EB8-40B8-9E3D-EC34513A66DB
Dimensions Publication ID	1034095312
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q59074350
is cites work of	Epigallocatechin-3-gallate enhances CD8+ T cell-mediated antitumor immunity induced by DNA vaccination. Combinations of immunotherapy and radiation in cancer therapy. Effects of cyclophosphamide and a metabolite, acrolein, on Naegleria fowleri in vitro and in vivo Cancer immunotherapy: the role regulatory T cells play and what can be done to overcome their inhibitory effects. Regulatory T-cell modulation using cyclophosphamide in vaccine approaches: a current perspective Suppressive mechanisms involving sensitization and tolerance in contact allergy Prospects for exosomes in immunotherapy of cancer Manipulation of indoleamine 2,3 dioxygenase; a novel therapeutic target for treatment of diseases Clinical development of combination strategies in immunotherapy: are we ready for more than one investigational product in an early clinical trial? Immunotherapy and chemotherapy--a practical partnership Comprehensive analysis of current approaches to inhibit regulatory T cells in cancer Immunomodulation in cancer therapeutics Anti-tumour synergy of cytotoxic chemotherapy and anti-CD40 plus CpG-ODN immunotherapy through repolarization of tumour-associated macrophages Rat erythrocyte-induced anti-erythrocyte autoantibody production and control in normal mice Regulation of delayed-type hypersensitivity. III. Effect of cyclophosphamide on the suppressor cells for delayed-type hypersensitivity to sheep erythrocytes in mice Enhancement of delayed hypersensitivity by depletion of suppressor T cells with cyclophosphamide in mice Effect of cyclophosphamide on the response of chickens to a virulent strain of Marek's disease virus Activation of human B lymphocytes. XII. Differential effects of in vitro cyclophosphamide on human lymphocyte subpopulations involved in B-cell activation Depression of contact sensitivity by enhancement of suppressor cell activity in Pseudomonas aeruginosa-injected mice The Effect of Cyclophospkamide on the Immune Response Chemotherapy enhances CD8(+) T cell-mediated antitumor immunity induced by vaccination with vaccinia virus Autoimmunity by pesticides: a critical review of the state of the science Augmentation of delayed-type hypersensitivity by doses of cyclophosphamide which do not affect antibody responses Alkaline phosphate in the differentiation of guinea pig T lymphocytes The tumor microenvironment expression of p-STAT3 influences the efficacy of cyclophosphamide with WP1066 in murine melanoma models. Analysis of Autoimmunity through Experimental Models of Thyroiditis and Allergic Encephalomyelitis A New Animal Model for Contact Dermatitis: The Hairless Guinea Pig The effects of sodium diethyldithiocarbamate, azathioprine, cyclophosphamide, or hydrocortisone acetate administered alone or in association for 4 weeks on the immune responses of mice Combined therapy with cyclophosphamide and DNA preparation inhibits the tumor growth in mice Special regulatory T-cell review: A rose by any other name: from suppressor T cells to Tregs, approbation to unbridled enthusiasm Tumour immunogenicity, antigen presentation and immunological barriers in cancer immunotherapy The induction of hapten-specific T cell tolerance using hapten-modified lymphoid membranes II. Relative roles of suppressor T cells and clone inhibition in the tolerant state Situational aldehyde dehydrogenase expression by regulatory T cells may explain the contextual duality of cyclophosphamide as both a pro-inflammatory and tolerogenic agent Combination of cancer immunotherapy with clinically available drugs that can block immunosuppressive cells Inhibition of the humoral response by spleen cells from cyclophosphamide-treated mice Antigen- and receptor-driven regulatory mechanisms. VIII. Suppression of idiotype-negative, p-azobenzenearsonate-specific T cells results from the interaction of an anti-idiotypic second-order T suppressor cell with a cross-reactive-idiotype-positiv The relation between B-cell stimulation and delayed hypersensitivity. The effect of cyclophosphamide pretreatment on antibody production The relation of immune depression and B-cell stimulation during the development of delayed hypersensitivity to soluble antigens Suppression of contact hypersensitivity by short-term ultraviolet irradiation: I. Immunosuppression by serum from irradiated mice Multiple Pathways of B Lymphocyte Tolerance Responses to polyvinyl pyrrolidone and pneumococcal polysaccharide in protein-deficient mice Effects of cytotoxic immunosuppressants on tuberculin-sensitive lymphocytes in guinea pigs Suppressor cells in antigenic competition in contact allergy in mice Immunological Behaviour of Meningeal Exudate Cells in Experimental Allergic Encephalomyelitis

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