About: Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes

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About: Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes Goto Sponge NotDistinct Permalink

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scientific article published on 01 August 2006

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rdf:type	Item
description	wetenschappelijk artikel (nl) article scientifique publié en 2006 (fr) наукова стаття, опублікована в липні 2006 (uk) im Juli 2006 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'agostu de 2006 (ast) scientific article published on 01 August 2006 (en)
publication date	wds:Q58426487-A5F37798-70CF-4EC5-B578-2A03946C1298
publication date	2006-08-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q58426487-CC184992-5BF3-4662-8BA6-0C9D0E3139F2
language of work or name	English
author name string	wds:Q58426487-12F56D30-B3D3-4A00-8D05-3FE8994AE85A wds:Q58426487-5B6731BD-B0D0-4C0B-86DD-17D9A5B9AFC4 wds:Q58426487-8A5286E8-AC08-4307-888D-F6B906542F3E wds:Q58426487-9EAECB66-B409-49F0-8AD5-50CA0A77263D
author name string	Michael J Berridge Andrew Proven Jeffrey K Horton Stephen J Capper
rdfs:label	Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes (en) Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes (nl)
skos:prefLabel	Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes (en) Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes (nl)
name	Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes (en) Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes (nl)
author	wds:Q58426487-301D1773-1AE0-486B-832C-1DC671EC5F64 wds:Q58426487-546AC857-1F8F-434A-82A1-577BB4942A9D wds:Q58426487-F3481A1D-8CFD-444A-B3CF-40220969DAF5
author	Stuart J. Conway Martin D Bootman H Llewelyn Roderick
title	wds:Q58426487-15B00E3F-EA33-4425-AD6C-7B24E658E062
title	Inositol 1,4,5-trisphosphate supports the arrhythmogenic action of endothelin-1 on ventricular cardiac myocytes (en)
page(s)	wds:Q58426487-0F823212-B021-4DC9-B696-13ED29149097
page(s)	3363-3375
instance of	wds:Q58426487-F9BC6B0B-CB95-4369-BA7C-52633A984B04
instance of	scholarly article
PubMed ID	wds:Q58426487-8B9AD27C-C42A-469A-B6BF-6BB1FA0675C9
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/16882691
PubMed ID	16882691
published in	wds:Q58426487-67EB3B94-AADB-4B84-B539-2C599B4EFA2E
published in	Journal of Cell Science
issue	wds:Q58426487-4649473A-4DB0-4678-AA3C-F90BD557D070
volume	wds:Q58426487-22F26777-3A2F-4118-8DF9-CBDA98A2A330
issue	Pt 16
volume	119
DOI	wds:Q58426487-7FA99DCC-DBBE-4320-82AD-2C721632923C
DOI	http://dx.doi.org/10.1242/JCS.03073
DOI	10.1242/JCS.03073
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q58426487
is cites work of	Inositol-1,4,5-trisphosphate-mediated spontaneous activity in mouse embryonic stem cell-derived cardiomyocytes Wide long lasting perinuclear Ca2+ release events generated by an interaction between ryanodine and IP3 receptors in canine Purkinje cells Ankyrin-B reduction enhances Ca spark-mediated SR Ca release promoting cardiac myocyte arrhythmic activity. Type 1 inositol (1,4,5)-trisphosphate receptor activates ryanodine receptor 1 to mediate calcium spark signaling in adult mammalian skeletal muscle Ca²+ spark-dependent and -independent sarcoplasmic reticulum Ca²+ leak in normal and failing rabbit ventricular myocytes Optogenetic activation of Gq signalling modulates pacemaker activity of cardiomyocytes. 1,4,5-Inositol trisphosphate-operated intracellular Ca(2+) stores and angiotensin-II/endothelin-1 signaling pathway are functional in human embryonic stem cell-derived cardiomyocytes G protein-dependent and G protein-independent signaling pathways and their impact on cardiac function Caveolae, ion channels and cardiac arrhythmias Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes Activation of purinergic receptors by ATP induces ventricular tachycardia by membrane depolarization and modifications of Ca2+ homeostasis Calcium and IP3 dynamics in cardiac myocytes: experimental and computational perspectives and approaches Role of inositol 1,4,5-trisphosphate in the regulation of ventricular Ca(2+) signaling in intact mouse heart Increased InsP3Rs in the junctional sarcoplasmic reticulum augment Ca2+ transients and arrhythmias associated with cardiac hypertrophy The IP3 receptor regulates cardiac hypertrophy in response to select stimuli Why, where, and when do cardiac myocytes express inositol 1,4,5-trisphosphate receptors? IP3 receptor-dependent Ca2+ release modulates excitation-contraction coupling in rabbit ventricular myocytes IP3-dependent nuclear Ca2+ signalling in the mammalian heart. An integrated mechanism of cardiomyocyte nuclear Ca(2+) signaling Pacemaking, arrhythmias, inotropy and hypertrophy: the many possible facets of IP3 signalling in cardiac myocytes Inhibition of thromboxane A2-induced arrhythmias and intracellular calcium changes in cardiac myocytes by blockade of the inositol trisphosphate pathway. Nicotinic acid adenine dinucleotide phosphate (NAADP)-mediated calcium signaling and arrhythmias in the heart evoked by β-adrenergic stimulation Endothelial dysfunction as a nexus for endothelial cell-cardiomyocyte miscommunication. Chemically Homogenous Compounds with Antagonistic Properties at All α1-Adrenoceptor Subtypes but not β1-Adrenoceptor Attenuate Adrenaline-Induced Arrhythmia in Rats The type 2 inositol 1,4,5-trisphosphate receptor, emerging functions for an intriguing Ca²⁺-release channel Contractile responses to endothelin-1 are regulated by PKC phosphorylation of cardiac myosin binding protein-C in rat ventricular myocytes. The role of the paracrine/autocrine mediator endothelin-1 in regulation of cardiac contractility and growth. Vitamin D: a custodian of cell signalling stability in health and disease Physiologic, Pathologic, and Therapeutic Paracrine Modulation of Cardiac Excitation-Contraction Coupling. Endothelin system and atrial fibrillation post-cardiac surgery. Functionally redundant control of cardiac hypertrophic signaling by inositol 1,4,5-trisphosphate receptors. Calcium in the Pathophysiology of Atrial Fibrillation and Heart Failure Inositol 1,4,5-trisphosphate-mediated sarcoplasmic reticulum-mitochondrial crosstalk influences adenosine triphosphate production via mitochondrial Ca2+ uptake through the mitochondrial ryanodine receptor in cardiac myocytes Endothelin-1 promotes hypertrophic remodelling of cardiac myocytes by activating sustained signalling and transcription downstream of endothelin type A receptors Spatially defined InsP3-mediated signaling in embryonic stem cell-derived cardiomyocytes Multiple H+ sensors mediate the extracellular acidification-induced [Ca2+]i elevation in cultured rat ventricular cardiomyocytes Molecular characterization and association analysis with production traits of the porcine INPP5F gene Cardiac inositol 1,4,5-trisphosphate receptors Endothelin-1 signalling controls early embryonic heart rate in vitro and in vivo. Cardiac thromboxane A2 receptor activation does not directly induce cardiomyocyte hypertrophy but does cause cell death that is prevented with gentamicin and 2-APB. Cardiac expression of ryanodine receptor subtype 3; a strategic component in the intracellular Ca2+ release system of Purkinje fibers in large mammalian heart. Endothelin-B Receptors and Left Ventricular Dysfunction after Regional versus Global Ischaemia-Reperfusion in Rat Hearts. Obstruction of ventricular Ca2+ -dependent arrhythmogenicity by inositol 1,4,5-trisphosphate-triggered sarcoplasmic reticulum Ca2+ release Mitofusin 2 Is Essential for IP3-Mediated SR/Mitochondria Metabolic Feedback in Ventricular Myocytes DHHC5 Mediates β-Adrenergic Signaling in Cardiomyocytes by Targeting Gα Proteins Do endothelin receptor antagonists have an antiarrhythmic potential during acute myocardial infarction? Evidence from experimental studies. Temporal response of ectopic activity in guinea pig ventricular myocardium in response to isoproterenol and acetylcholine
is cites work of	wds:Q34103732-E91590A4-7C88-40CB-838E-1E45D71CB17B wds:Q36890713-EFD6E0C2-830C-4B3F-BD2E-52379CEF8D40 wds:Q37125035-3A5699AC-E785-4111-9A94-E019EE143909

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