About: Sean M. Davidson

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

professorial Research Fellow at UCL

Attributes	Values
rdf:type	Item
description	forskare (sv) ikertzailea (eu) 연구원 (ko) 研究者 (ja) araştırmacı (tr) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy) onderzoeker (nl) professorial Research Fellow at UCL (en)
rdfs:label	Sean M Davidson (es) Sean M Davidson (nl) Sean M Davidson (sv) Sean M Davidson (ast) Sean M. Davidson (en)
skos:prefLabel	Sean M Davidson (es) Sean M Davidson (nl) Sean M Davidson (sv) Sean M Davidson (ast) Sean M. Davidson (en)
name	Sean M Davidson (es) Sean M Davidson (nl) Sean M Davidson (sv) Sean M Davidson (ast) Sean M. Davidson (en)
LinkedIn personal profile ID	wds:Q56988847-99f94c15-4d66-cba7-aad2-d88ee2ed9cba
LinkedIn personal profile ID	sean-davidson-a8bb2047
given name	wds:Q56988847-E16BB797-CF85-499F-AEF7-7E3AE969A770
given name	Sean
instance of	wds:Q56988847-B1BBF337-9994-4661-AE34-2FAFC3E94B56
instance of	Homo sapiens
award received	wds:Q56988847-1759b570-4dcb-8261-4c64-6ead69ac7dea wds:Q56988847-adddfd6d-4f43-d1bb-38de-c344928dcdeb
award received	Fellow of the American Heart Association Special Recognition Award in Arteriosclerosis
skos:altLabel	Sean Davidson (en) Sean Davidson (sv) Sean M Davidson (en)
occupation	wds:Q56988847-09EF75D7-454E-4E59-AF14-06F4AF66F798
employer	wds:Q56988847-91B8D19E-0950-4F23-8701-B56D4B2422E6 wds:Q56988847-A745892A-A122-4060-944E-CED9379D5B8B wds:Q56988847-C9383985-0003-42BC-8DAD-19CF006B59D0 wds:Q56988847-CE29D92F-76DE-4E11-8B4F-23A8154D38D0
sex or gender	wds:Q56988847-fbfd5fd5-4182-9e25-f6a7-9a4ced9fab05
educated at	wds:Q56988847-1549D095-F38D-4F4B-BE18-A3D77BCAA61A wds:Q56988847-501DFEF0-8693-423D-890C-F4FE3F0AD512
family name	wds:Q56988847-9DF6E1C1-6671-4511-9157-0E3B6BE40BC3
official website	wds:Q56988847-55051e04-4582-42e3-8dd0-7c3baf522f81
occupation	researcher
employer	University College London Institut de Biologie de l'École Normale Supérieure
sex or gender	male
educated at	La Trobe University University of Melbourne
family name	Davidson
official website	https://iris.ucl.ac.uk/iris/browse/profile?upi=SDAVI13
ORCID iD	wds:Q56988847-EB9FF633-443B-4283-BAA8-4DF06F35FD3B
ORCID iD	https://orcid.org/0000-0001-5182-4980
ORCID iD	0000-0001-5182-4980
Gateway to Research person ID	wds:Q56988847-0038FAC0-511A-41FC-AE50-8B5649D42CEA
Gateway to Research person ID	08B4C5B2-C31E-468A-8EE5-C1B0934203F0
ResearcherID	wds:Q56988847-6127384C-E81A-48B6-8947-97D6BFB22256
Publons author ID	wds:Q56988847-3aa59098-458a-3631-74c7-23645ad636b8
ResearcherID	F-5275-2010
Publons author ID	2828338
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q56988847
is author of	Translating novel strategies for cardioprotection: the Hatter Workshop Recommendations The cardioprotective actions of leptin are lost in the Zucker obese (fa/fa) rat. Interaction of the heart and its close and distant neighbours: report of the Meeting of the ESC Working Groups Myocardial Function and Cellular Biology Parkin is recruited into aggresomes in a stress-specific manner: over-expression of parkin reduces aggresome formation but can be dissociated from parkin's effect on neuronal survival Endothelial mitochondria and heart disease De novo cardiomyocytes from within the activated adult heart after injury Microvesicles and exosomes: new players in metabolic and cardiovascular disease Exosomes: nanoparticles involved in cardioprotection? Remote ischaemic preconditioning involves signalling through the SDF-1α/CXCR4 signalling axis. Plasma exosomes protect the myocardium from ischemia-reperfusion injury. Necrostatin: a potentially novel cardioprotective agent? STAT-1 interacts with p53 to enhance DNA damage-induced apoptosis Endothelial mitochondria: contributing to vascular function and disease Metformin protects the ischemic heart by the Akt-mediated inhibition of mitochondrial permeability transition pore opening Metformin prevents myocardial reperfusion injury by activating the adenosine receptor. Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS. Trials, tribulations and speculation! Report from the 7th Biennial Hatter Cardiovascular Institute Workshop. ESC working group cellular biology of the heart: position paper: improving the preclinical assessment of novel cardioprotective therapies 9th Hatter Biannual Meeting: position document on ischaemia/reperfusion injury, conditioning and the ten commandments of cardioprotection Novel therapeutic strategies for cardioprotection. Matrix metalloproteinase inhibition protects CyPD knockout mice independently of RISK/mPTP signalling: a parallel pathway to protection. Myocardial regeneration: expanding the repertoire of thymosin β4 in the ischemic heart UK-Russia Researcher Links Workshop: extracellular vesicles - mechanisms of biogenesis and roles in disease pathogenesis, M.V. Lomonosov Moscow State University, Moscow, Russia, 1-5 March 2015 Slow calcium waves and redox changes precede mitochondrial permeability transition pore opening in the intact heart during hypoxia and reoxygenation Vascular smooth muscle cell calcification is mediated by regulated exosome secretion. STAT-1 facilitates the ATM activated checkpoint pathway following DNA damage Molecular regulation of cardiac hypertrophy Local control of nuclear calcium signaling in cardiac myocytes by perinuclear microdomains of sarcolemmal insulin-like growth factor 1 receptors Enhancing AMPK activation during ischemia protects the diabetic heart against reperfusion injury Inhibiting mitochondrial fission protects the heart against ischemia/reperfusion injury Photoaffinity labeling of nicotinic acid adenine dinucleotide phosphate (NAADP) targets in mammalian cells Loss of PINK1 increases the heart's vulnerability to ischemia-reperfusion injury.

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