About: Frank L. Heppner

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	forskare (sv) researcher (en) 연구원 (ko) 研究者 (ja) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) изилдөөчү (ky) 研究者 (zh) հետազոտող (hy)
rdfs:label	Frank Heppner (es) Frank Heppner (nl) Frank Heppner (ast) Frank L. Heppner (en)
skos:prefLabel	Frank Heppner (es) Frank Heppner (nl) Frank Heppner (ast) Frank L. Heppner (en)
name	Frank Heppner (es) Frank Heppner (nl) Frank Heppner (ast) Frank L. Heppner (en)
given name	wds:Q56955724-CFC804EA-F8D5-4795-8BBD-030991D08F40
given name	Frank
instance of	wds:Q56955724-AD0D18BB-97BA-4AE4-8981-AED62D9C736F
instance of	Homo sapiens
Google Scholar author ID	wds:Q56955724-ca1f71d1-4d7f-6a62-5c31-d33ffdf5741f
Google Scholar author ID	sJoLRl0AAAAJ
skos:altLabel	Frank Heppner (en)
occupation	wds:Q56955724-E776063E-16CC-431B-9B2F-831AF08C2BEB
employer	wds:Q56955724-0BB66DB2-52A3-4D72-83C9-C0549B3031FF
sex or gender	wds:Q56955724-2222E174-0190-4ED5-9DB7-0C2B5EC5179B
ISNI	wds:Q56955724-c780cdc8-4474-6613-8353-79c79bf8a377
VIAF ID	wds:Q56955724-f74d77ab-48a1-da0c-827d-e0a276cbaecc
GND ID	wds:Q56955724-b70f2281-4b0a-7621-6c3f-5ce0c5426321
member of	wds:Q56955724-2B09B4D7-888B-416A-8A20-48486CC28978
family name	wds:Q56955724-7F87AFFD-EB1A-44C7-A1F0-A40D5F396CCE
FactGrid item ID	wds:Q56955724-4B26CA58-2B6F-4DB9-AF6D-632275C370C6
official website	wds:Q56955724-42d5adb4-4eb8-7daf-ce17-bda4e58f32d5 wds:Q56955724-6f168fcc-473c-5a10-b454-21003b27d17c
VIAF ID	http://viaf.org/viaf/32836990
GND ID	https://d-nb.info/gnd/12081806X
FactGrid item ID	https://database.factgrid.de/entity/Q885233
occupation	researcher
employer	Charité
sex or gender	male
ISNI	0000000016175551
VIAF ID	32836990
GND ID	12081806X
member of	German Academy of Sciences Leopoldina
family name	Heppner
FactGrid item ID	Q885233
official website	https://www.charite.de/en/service/en_person_detail/person/address_detail/heppner/ https://www.dzne.de/en/research/research-areas/fundamental-research/research-groups/heppner/research-areasfocus/
Leopoldina member ID (new)	wds:Q56955724-D9F9B45D-272B-47AD-9865-70E8B9152B80
ORCID iD	wds:Q56955724-54A95659-48BB-4137-B7C7-66565B99308D
Dimensions author ID	wds:Q56955724-3195F9D1-4190-449F-926C-6354B3201E04
ORCID iD	https://orcid.org/0000-0001-9816-8917
Leopoldina member ID (new)	frank-heppner
ORCID iD	0000-0001-9816-8917
Dimensions author ID	01153074764.27
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q56955724
is author of	Paracrine inhibition of prion propagation by anti-PrP single-chain Fv miniantibodies Microglia actions in Alzheimer’s disease. Inhibition of IL-12/IL-23 signaling reduces Alzheimer's disease-like pathology and cognitive decline. Microglial ablation and lipopolysaccharide preconditioning affects pilocarpine-induced seizures in mice. Inhibiting receptor tyrosine kinase AXL with small molecule inhibitor BMS-777607 reduces glioblastoma growth, migration, and invasion in vitro and in vivo Reprogrammed quiescent B cells provide an effective cellular therapy against chronic experimental autoimmune encephalomyelitis Dendritic cells permit immune invasion of the CNS in an animal model of multiple sclerosis. An unconventional role for miRNA: let-7 activates Toll-like receptor 7 and causes neurodegeneration. Functional impairment of microglia coincides with Beta-amyloid deposition in mice with Alzheimer-like pathology The 4q12 amplicon in malignant peripheral nerve sheath tumors: consequences on gene expression and implications for sunitinib treatment Detection of kappa and delta opioid receptors in skin--outside the nervous system. A Cre-inducible diphtheria toxin receptor mediates cell lineage ablation after toxin administration. Immune system and peripheral nerves in propagation of prions to CNS. Resident microglia rather than peripheral macrophages promote vascularization in brain tumors and are source of alternative pro-angiogenic factors. Experimental autoimmune encephalomyelitis repressed by microglial paralysis. Early and rapid engraftment of bone marrow-derived microglia in scrapie. Intravascular Inflammation Triggers Intracerebral Activated Microglia and Contributes to Secondary Brain Injury After Experimental Subarachnoid Hemorrhage (eSAH). A 2015 update on predictive molecular pathology and its role in targeted cancer therapy: a review focussing on clinical relevance Immune attack: the role of inflammation in Alzheimer disease. Membrane-type 1 metalloproteinase is upregulated in microglia/brain macrophages in neurodegenerative and neuroinflammatory diseases. Modeling multiple sclerosis in laboratory animals. Impact of peripheral myeloid cells on amyloid-β pathology in Alzheimer's disease-like mice Luminescent conjugated oligothiophenes for sensitive fluorescent assignment of protein inclusion bodies Prosurvival effect of DHCR24/Seladin-1 in acute and chronic responses to oxidative stress MGMT Promoter Methylation and BRAF V600E Mutations Are Helpful Markers to Discriminate Pleomorphic Xanthoastrocytoma from Giant Cell Glioblastoma Impaired Pten expression in human malignant peripheral nerve sheath tumours An azide functionalized oligothiophene ligand--a versatile tool for multimodal detection of disease associated protein aggregates. Nuclear translocation uncovers the amyloid peptide Aβ42 as a regulator of gene transcription. Nuclear actin aggregation is a hallmark of anti-synthetase syndrome-induced dysimmune myopathy. Cerebral and peripheral amyloid phagocytes--an old liaison with a new twist. Astrocyte Depletion Impairs Redox Homeostasis and Triggers Neuronal Loss in the Adult CNS. High-fat diet-induced brain region-specific phenotypic spectrum of CNS resident microglia Glioma-derived versican promotes tumor expansion via glioma-associated microglial/macrophages Toll-like receptor 2 signaling Microglia inflict delayed brain injury after subarachnoid hemorrhage. Current concepts and controversies in prion immunopathology Personalized risk prediction of postoperative cognitive impairment - rationale for the EU-funded BioCog project.

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